نتایج جستجو برای: y microdeletion

تعداد نتایج: 495553  

2014
Katherina Walz Juan I Young

2Iq23.1 microdeletion syndrome is a recently described rare disease that includes intellectual disability, motor delay, autistic-like behaviors, and craniofacial abnormalities. Dosage insufficiency of the methyl-CpG-binding domain protein 5 (MBD5) gene was suggested as the genetic cause, since all the described patients carry a partial or total heterozygous deletion of MBD5. We reported the gen...

2011
Emmanuel ADETIBA Frank A. IBIKUNLE

In this research work, we built and ensembled different EGFR microdeletion mutations’ based Artificial Neural Networks(ANNs) for improved diagnosis of Non-Small Cell Lung Cancer(NSCLC). We developed two novel algorithms, namely; Genomic Nucleotide Encoding & Normalization (GNEN) algorithm to encode and normalize the EGFR nucleotides and SimMicrodel algorithm to programmatically simulate microde...

2016
Jonathan D. J. Labonne Tyler D. Graves Yiping Shen Julie R. Jones Il-Keun Kong Lawrence C. Layman Hyung-Goo Kim

BACKGROUND Among the 21 annotated genes at Xq22.2, PLP1 is the only known gene involved in Xq22.2 microdeletion and microduplication syndromes with intellectual disability. Using an atypical microdeletion, which does not encompass PLP1, we implicate a novel gene GLRA4 involved in intellectual disability, behavioral problems and craniofacial anomalies. CASE PRESENTATION We report a female pati...

Journal: :American journal of hypertension 2007
Akihiko Morita Tomohiro Nakayama Masayoshi Soma Tomohiko Mizutani

BACKGROUND Calcitonin-related peptide alpha (CALCA) is a neuropeptide that is a very potent vasodilator. It has been reported that CALCA knockout mice have a significantly elevated systolic blood pressure (BP). The aims of this study were to discover novel polymorphisms or mutations in the 5' flanking region of the human CALCA gene in Japanese subjects and to assess the association between this...

2017
Julia K. Herzig Lars Bullinger Alpaslan Tasdogan Philipp Zimmermann Martin Schlegel Veronica Teleanu Daniela Weber Frank G. Rücker Peter Paschka Anna Dolnik Edith Schneider Florian Kuchenbauer Florian H. Heidel Christian Buske Hartmut Döhner Konstanze Döhner Verena I. Gaidzik

We have previously identified a recurrent deletion at chromosomal band 3p14.1-p13 in patients with acute myeloid leukemia (AML). Among eight protein-coding genes, this microdeletion affects the protein phosphatase 4 regulatory subunit 2 (PPP4R2), which plays an important role in DNA damage response (DDR). Investigation of mRNA expression during murine myelopoiesis determined that Ppp4r2 is high...

2018
D. Dell’Edera C. Dilucca A. Allegretti F. Simone M. G. Lupo C. Liccese R. Davanzo

BACKGROUND The recurrent ∼ 600 kb 16p11.2 microdeletion is among the most commonly known genetic etiologies of autism spectrum disorder, overweightness, and related neurodevelopmental disorders. CASE PRESENTATION Our patient is a 2-year-old white girl from the first pregnancy of a non-consanguineous healthy young white couple (father 33-years old and mother 29-years old). Our patient and her ...

Journal: :Human molecular genetics 2009
Leanne M Dibbens Saul Mullen Ingo Helbig Heather C Mefford Marta A Bayly Susannah Bellows Costin Leu Holger Trucks Tanja Obermeier Michael Wittig Andre Franke Hande Caglayan Zuhal Yapici Thomas Sander Evan E Eichler Ingrid E Scheffer John C Mulley Samuel F Berkovic

Microdeletion at chromosomal position 15q13.3 has been described in intellectual disability, autism spectrum disorders, schizophrenia and recently in idiopathic generalized epilepsy (IGE). Using independent IGE cohorts, we first aimed to confirm the association of 15q13.3 deletions and IGE. We then set out to determine the relative occurrence of sporadic and familial cases and to examine the li...

Journal: :European journal of medical genetics 2011
Christèle Dubourg Damien Sanlaville Martine Doco-Fenzy Cédric Le Caignec Chantal Missirian Sylvie Jaillard Caroline Schluth-Bolard Emilie Landais Odile Boute Nicole Philip Annick Toutain Albert David Patrick Edery Anne Moncla Dominique Martin-Coignard Catherine Vincent-Delorme Isabelle Mortemousque Bénédicte Duban-Bedu Sèverine Drunat Mylène Beri Jean Mosser Sylvie Odent Véronique David Joris Andrieux

Chromosome 17q21.31 microdeletion was one of the first genomic disorders identified by chromosome microarrays. We report here the clinical and molecular characterization of a new series of 14 French patients with this microdeletion syndrome. The most frequent clinical features were hypotonia, developmental delay and facial dysmorphism, but scaphocephaly, prenatal ischemic infarction and percept...

Journal: :modares journal of medical sciences: pathobiology 2007
sara pouranvari mehrdad noruzinia aliakbar zinalou saeedreza ghafari masoud houshmand

objective: 22q11.2 chromosomal region is a hot spot for many cytogenetic rearrangements especially microdeletions which are responsible for digeorge and velocardiofacial syndromes. the most characteristic sign in these patients is congenital cardiac conotruncal anomalies. the gold standard diagnostic test for these microdeletions is fish (fluorescent in situ hybridization). however this diagnos...

Journal: :Molecular syndromology 2012
M H Willemsen A T Vulto-van Silfhout W M Nillesen W M Wissink-Lindhout H van Bokhoven N Philip E M Berry-Kravis U Kini C M A van Ravenswaaij-Arts B Delle Chiaie A M M Innes G Houge T Kosonen K Cremer M Fannemel A Stray-Pedersen W Reardon J Ignatius K Lachlan C Mircher P T J M Helderman van den Enden M Mastebroek P E Cohn-Hokke H G Yntema S Drunat T Kleefstra

Kleefstra syndrome is characterized by the core phenotype of developmental delay/intellectual disability, (childhood) hypotonia and distinct facial features. The syndrome can be either caused by a microdeletion in chromosomal region 9q34.3 or by a mutation in the euchromatin histone methyltransferase 1 (EHMT1) gene. Since the early 1990s, 85 patients have been described, of which the majority h...

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