نتایج جستجو برای: ژن peroxiredoxin

تعداد نتایج: 17682  

Journal: :Circulation 2013
Dong Hoon Kang Doo Jae Lee Jiran Kim Joo Young Lee Hyun-Woo Kim Kihwan Kwon W Robert Taylor Hanjoong Jo Sang Won Kang

BACKGROUND Typical 2-Cys peroxiredoxin (Prx) is inactivated by overoxidation of the peroxidatic cysteine residue under oxidative stress. However, the significance in the context of vascular disease is unknown. METHODS AND RESULTS Immunohistochemical analyses revealed that 2-Cys Prxs, particularly Prx type II, are heavily overoxidized in balloon-injured rodent carotid vessels and in human athe...

Journal: :Blood 2014
Markus Schneider Marcin Szaumkessel Julia Richter Ole Ammerpohl Martin-Leo Hansmann Ralf Küppers Reiner Siebert Maciej Giefing

In a recent issue of Blood, Agrawal-Singh et al reported the identification of PRDX2 (peroxiredoxin 2) as a novel tumor suppressor gene in acute myeloid leukemia (AML). The authors observed recurrent histone H3 deacetylation and DNA hypermethylation at the PRDX2 promoter region in primary leukemia samples. Epigenetic alterations correlated with low expression of PRDX2 protein and were associate...

Journal: :Endocrines 2022

Background: Peroxiredoxin 4 (PRDX4), a secreted antioxidant enzyme, can protect against hepatocellular carcinoma and lung adenocarcinoma, but its role in papillary thyroid (PTC) is still unclear. In this study, we investigated the association of PRDX4 expression with prognosis patients advanced PTC. Methods: We conducted retrospective case-control study at Kanazawa Medical University Hospital. ...

Journal: :Acta Crystallographica Section F Structural Biology and Crystallization Communications 2013

Journal: :Journal of Biological Chemistry 2007

Journal: :Biocell 2022

Peroxiredoxin 1 (PRDX1) participates in tumor cell proliferation, apoptosis, migration, invasion, and the epithelial-to-mesenchymal transition (EMT). This study aimed to investigate effect of PRDX1 on EMT airway epithelial cells stimulated with lipopolysaccharide (LPS) transforming growth factor-beta (TGF-β1). overexpression significantly increased proliferation migration human bronchial (BEAS-...

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