نتایج جستجو برای: compensatory growth

تعداد نتایج: 832714  

2015
Andreas Walzer Natalia Lepp Peter Schausberger

Compensatory or catch-up growth following growth impairment caused by transient environmental stress, due to adverse abiotic factors or food, is widespread in animals. Such growth strategies commonly balance retarded development and reduced growth. They depend on the type of stressor but are unknown for predation risk, a prime selective force shaping life history. Anti-predator behaviours by im...

2000
CHRISTOPHER A. LEPCZYK WILLIAM H. KARASOV

—We tested for the presence of compensatory growth (i.e. faster age-specific growth) following ephemeral periods of food restriction in altricial nestlings using the House Sparrow (Passer domesticus) as a model species. To simulate periods of poor food conditions, we raised nestlings in captivity, fed them a synthetic diet, and held them at constant body mass for 48 h beginning on either day 3 ...

Journal: :The American naturalist 2005
Marc Mangel Stephan B Munch

Compensatory or catch-up growth (CG) is widely observed following periods of resource deprivation. Because of this commonness, it is generally assumed that compensatory growth is adaptive, but most theory to date has explicitly ignored considerations of fitness. Following a period of deprivation, when resources become plentiful again, individuals may not respond at all and continue on a "normal...

2014
Matthew R. DiStasi Joseph L. Unthank Steven J. Miller

DiStasi MR, Unthank JL, Miller SJ. Nox2 and p47 modulate compensatory growth of primary collateral arteries. Am J Physiol Heart Circ Physiol 306: H1435–H1443, 2014. First published March 14, 2014; doi:10.1152/ajpheart.00828.2013.—The role of NADPH oxidase (Nox) in both the promotion and impairment of compensatory collateral growth remains controversial because the specific Nox and reactive oxyg...

Journal: :The Journal of clinical investigation 2004
Rohit N Kulkarni Ulupi S Jhala Jonathon N Winnay Stan Krajewski Marc Montminy C Ronald Kahn

Inadequate compensatory beta cell hyperplasia in insulin-resistant states triggers the development of overt diabetes. The mechanisms that underlie this crucial adaptive response are not fully defined. Here we show that the compensatory islet-growth response to insulin resistance in 2 models--insulin receptor (IR)/IR substrate-1 (IRS-1) double heterozygous mice and liver-specific IR KO (LIRKO) m...

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