نتایج جستجو برای: ercc5

تعداد نتایج: 216  

Journal: :The EMBO journal 2006
Ryan D Bomgarden Patrick J Lupardus Deena V Soni Muh-Ching Yee James M Ford Karlene A Cimprich

An essential component of the ATR (ataxia telangiectasia-mutated and Rad3-related)-activating structure is single-stranded DNA. It has been suggested that nucleotide excision repair (NER) can lead to activation of ATR by generating such a signal, and in yeast, DNA damage processing through the NER pathway is necessary for checkpoint activation during G1. We show here that ultraviolet (UV) radia...

2014
Xiao-Feng He Li-Rong Liu Wu Wei Yi Liu Jiao Su Su-Lan Wang Xu-Liang Shen Xian-Bin Yang

BACKGROUND The XPG (xeroderma pigmentosum type G) Asp1104His and XPF (xeroderma pigmentosum type F) Arg415Gln polymorphisms had been implicated in cancer susceptibility. The previous published data on the association between XPG Asp1104His and XPF Arg415Gln polymorphisms and cancer risk remained controversial. METHODOLOGY/PRINCIPAL FINDINGS To derive a more precise estimation of the associati...

Journal: :Neuroscience letters 2008
Yue Zheng Xiao-Rui Cheng Wen-Xia Zhou Yong-Xiang Zhang

Alzheimer's disease (AD) is a progressive, neurodegenerative disease, which primarily affects the elderly. Clinical signs of AD are characterized by the neuron loss and cognitive impairment. At gene and protein levels, the senescence-accelerated mouse/prone 8 (SAMP8) is a suitable animal model to investigate the fundamental mechanisms of age-related learning and memory deficits. Huang-Lian-Jie-...

Journal: :Carcinogenesis 2011
Tasha R Smith Wen Liu-Mares Beth O Van Emburgh Edward A Levine Glenn O Allen Jeff W Hill Isildinha M Reis Laura A Kresty Mark D Pegram Mark S Miller Jennifer J Hu

Defective DNA repair may contribute to early age and late stage at time of diagnosis and mutations in critical tumor suppressor genes, such as TP53 in breast cancer. Using DNA samples from 436 breast cancer cases (374 Caucasians and 62 African-Americans), we tested these associations with 18 non-synonymous single-nucleotide polymorphisms (nsSNPs) in four DNA repair pathways: (i) base excision r...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2012
Abul Kalam Azad Isabelle Bairati Elodie Samson Dangxiao Cheng Maryam Mirshams Xin Qiu Sevtap Savas John Waldron Changshu Wang David Goldstein Wei Xu Francois Meyer Geoffrey Liu

PURPOSE From the published literature, we identified 23 germ line sequence variants in 17 genes from hypothesis-generating studies that were associated with prognosis of head and neck cancer, including sequence variants of DNA repair (ERCC1, ERCC4, ERCC5, MSH2, XPA, ERCC2, XRCC1, XRCC3), DNA methylation (DNMT3B), cell cycle and proliferation (CCND1, TP53), xenobiotic metabolism (GSTM1, GSTT1, C...

Journal: :Orphanet journal of rare diseases 2016
Nadège Calmels Géraldine Greff Cathy Obringer Nadine Kempf Claire Gasnier Julien Tarabeux Marguerite Miguet Geneviève Baujat Didier Bessis Patricia Bretones Anne Cavau Béatrice Digeon Martine Doco-Fenzy Bérénice Doray François Feillet Jesus Gardeazabal Blanca Gener Sophie Julia Isabel Llano-Rivas Artur Mazur Caroline Michot Florence Renaldo-Robin Massimiliano Rossi Pascal Sabouraud Boris Keren Christel Depienne Jean Muller Jean-Louis Mandel Vincent Laugel

BACKGROUND Deficient nucleotide excision repair (NER) activity causes a variety of autosomal recessive diseases including xeroderma pigmentosum (XP) a disorder which pre-disposes to skin cancer, and the severe multisystem condition known as Cockayne syndrome (CS). In view of the clinical overlap between NER-related disorders, as well as the existence of multiple phenotypes and the numerous gene...

2013
Mei Liu Ronghua Wu Fuye Yang Tao Wang Pingping Zhang Jianren Gu Dafang Wan Shengli Yang

A novel human gene, FN1BP1 (fibronectin 1 binding protein 1), was identified using the human placenta cDNA library. Northern blotting showed a transcript of ∼2.8 kb in human placenta, liver, and skeletal muscle tissues. This mRNA transcript length was similar to the full FN1BP1 sequence obtained previously. We established a conditionally induced stable cell line of Hep3B-Tet-on-FN1BP1 to invest...

2016
Theresa A. Koleck Catherine M. Bender Susan M. Sereika Adam M. Brufsky Barry C. Lembersky Priscilla F. McAuliffe Shannon L. Puhalla Priya Rastogi Yvette P. Conley

PURPOSE The purpose of this exploratory candidate gene association study was to examine relationships between polymorphisms in oxidative stress and DNA repair genes and pre-adjuvant therapy cognitive function (CF) in postmenopausal women diagnosed with early stage-breast cancer. METHODS Using a neuropsychological test battery, CF was assessed in 138 women diagnosed with breast cancer prior to...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 1999
L Cheng Y Guan L Li R J Legerski J Einspahr J Bangert D S Alberts Q Wei

DNA repair is central to the integrity of the human genome. Reduced DNA repair capacity has been linked to genetic susceptibility to cancer. An adequate expression level of DNA repair genes is essential for normal DNA repair activities. Although there is tissue specificity in the expression, searching for a surrogate tissue is needed for molecular epidemiological studies. In this study, the rel...

Journal: :Carcinogenesis 2007
Stefan Michiels Patrick Danoy Philippe Dessen Alex Bera Thomas Boulet Christine Bouchardy Mark Lathrop Alain Sarasin Simone Benhamou

DNA repair is essential for the maintenance of genetic stability. We undertook sequencing to determine common genetic variants in 70 genes involved in three major repair pathways (base excision repair, nucleotide excision repair and mismatch repair) and in DNA synthesis, and investigated their relationship to lung and head and neck (H-N) cancers. Of the 70 genes examined, 62 were successfully s...

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