نتایج جستجو برای: frameshift mutations

تعداد نتایج: 174767  

Journal: :Tumori 2010
Yoo Ri Kim Nak Gyun Chung Mi Ran Kang Nam Jin Yoo Sug Hyung Lee

AIMS AND BACKGROUND Microsatellite instability (MSI) in sporadic gastric cancer (GC) and colorectal cancer (CRC) causes frameshift mutations in gene sequences that contribute to cancer pathogenesis. Many mutations have already been identified in these two cancer types, but some are still undiscovered. METHODS We analyzed seven genes (cell cycle control and DNA damage signaling/repair-related ...

Journal: :Molecular pathology : MP 2001
A J Alvi J S Rader M Broggini F Latif E R Maher

AIMS To investigate the possible role of mutations in the transforming growth factor beta receptor type II gene (TGFBRII) in ovarian cancer and its relation to microsatellite instability (MSI), 43 sporadic ovarian tumours were analysed for mutations over the entire coding region of the TGFBRII gene. METHODS Mutational analysis was performed using the polymerase chain reaction (PCR), single st...

Journal: :American journal of human genetics 2014
Ellen Cottenie Andrzej Kochanski Albena Jordanova Boglarka Bansagi Magdalena Zimon Alejandro Horga Zane Jaunmuktane Paola Saveri Vedrana Milic Rasic Jonathan Baets Marina Bartsakoulia Rafal Ploski Pawel Teterycz Milos Nikolic Ros Quinlivan Matilde Laura Mary G Sweeney Franco Taroni Michael P Lunn Isabella Moroni Michael Gonzalez Michael G Hanna Conceicao Bettencourt Elodie Chabrol Andre Franke Katja von Au Markus Schilhabel Dagmara Kabzińska Irena Hausmanowa-Petrusewicz Sebastian Brandner Siew Choo Lim Haiwei Song Byung-Ok Choi Rita Horvath Ki-Wha Chung Stephan Zuchner Davide Pareyson Matthew Harms Mary M Reilly Henry Houlden

Using a combination of exome sequencing and linkage analysis, we investigated an English family with two affected siblings in their 40s with recessive Charcot-Marie Tooth disease type 2 (CMT2). Compound heterozygous mutations in the immunoglobulin-helicase-μ-binding protein 2 (IGHMBP2) gene were identified. Further sequencing revealed a total of 11 CMT2 families with recessively inherited IGHMB...

Journal: :Investigative ophthalmology & visual science 2002
Shirly G Panicker Aramati B M Reddy Anil K Mandal Niyaz Ahmed Hampapathalu A Nagarajaram Seyed E Hasnain Dorairajan Balasubramanian

PURPOSE To determine the possible molecular genetic defect underlying primary congenital glaucoma (PCG) in India and to identify the pathogenic mutations causing this childhood blindness. METHODS Twenty-two members of five clinically well-characterized consanguineous families were studied. The primary candidate gene CYP1B1 was amplified from genomic DNA, sequenced, and analyzed in control sub...

2009
Marta Gîrdea Laurent Noé Gregory Kucherov

Frameshift mutations in protein-coding DNA sequences produce a drastic change in the resulting protein sequence, which prevents classic protein alignment methods from revealing the proteins’ common origin. Moreover, when a large number of substitutions are additionally involved in the divergence, the homology detection becomes difficult even at the DNA level. To cope with this situation, we pro...

2010
Magnus von Knebel-Doeberitz

Deficient DNA mismatch repair (MMR) boosts the accumulation of frameshift mutations in genes encompassing coding microsatellites (cMS). This results in the translation of proteins with mutation-induced frameshift peptides (neoantigens) rendering microsatellite-unstable (MSI) cancers highly immunogenic. MSI cancers express a defined set of neoantigens resulting from functionally relevant driver ...

2017
Simon Lalonde Oliver A Stone Samuel Lessard Adam Lavertu Jessica Desjardins Mélissa Beaudoin Manuel Rivas Didier Y R Stainier Guillaume Lettre

The introduction of frameshift indels by genome editing has emerged as a powerful technique to study the functions of uncharacterized genes in cell lines and model organisms. Such mutations should lead to mRNA degradation owing to nonsense-mediated mRNA decay or the production of severely truncated proteins. Here, we show that frameshift indels engineered by genome editing can also lead to skip...

Journal: :Journal of Korean Medical Science 2001
K. W. Lee J. M. Lee K. S. Jung

Mutations in the pyrazinamidase (PZase) gene (pncA) are considered the major mechanism of pyrazinamide (PZA) resistance in Mycobacterium tuberculosis. The aim of this study was designed to determine pncA mutations among ten PZA resistant and two PZA susceptible M. tuberculosis strains from Turkey and also to compare the PZase activity of them with the genotype. All isolates were identified by B...

Journal: :Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 1999
K Yamamoto J P Cox T Friedrich P T Christie M Bald P N Houtman M J Lapsley L Patzer M Tsimaratos W G Van'T Hoff K Yamaoka T J Jentsch R V Thakker

Dent's disease is an X-linked renal tubular disorder characterized by low molecular weight proteinuria, hypercalciuria, nephrocalcinosis, nephrolithiasis, and renal failure. The disease is caused by mutations in a renal chloride channel gene, CLCN5, which encodes a 746 amino acid protein (CLC-5), with 12 to 13 transmembrane domains. In this study, an additional six unrelated patients with Dent'...

2008
Chiara Redaelli Rosalind A Coleman Laura Moro Catherine Dacou-Voutetakis Solaf Mohamed Elsayed Daniele Prati Agostino Colli Donatella Mela Roberto Colombo Daniela Tavian

BACKGROUND Chanarin-Dorfman syndrome (CDS) is a rare autosomal recessive disorder characterized by nonbullous congenital ichthyosiform erythroderma (NCIE) and an intracellular accumulation of triacylglycerol (TG) droplets in most tissues. The clinical phenotype involves multiple organs and systems, including liver, eyes, ears, skeletal muscle and central nervous system (CNS). Mutations in ABHD5...

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