نتایج جستجو برای: gstm1

تعداد نتایج: 1660  

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2009
Sandi L Navarro Jyh-Lurn Chang Sabrina Peterson Chu Chen Irena B King Yvonne Schwarz Shuying S Li Lin Li John D Potter Johanna W Lampe

Glutathione S-transferases (GST) detoxify a wide range of carcinogens. Isothiocyanates (ITC), from cruciferous vegetables, are substrates for and inducers of GST. GST variants may alter ITC clearance such that response to crucifers varies by genotype. In a randomized cross-over trial, we tested the hypothesis that changes in serum GSTA1/2 concentration in response to cruciferous vegetable feedi...

Journal: :Anticancer research 2011
Tomomitsu Tahara Tomoyuki Shibata Masakatsu Nakamura Hiromi Yamashita Daisuke Yoshioka Masaaki Okubo Joh Yonemura Yoshio Kamiya Takamitsu Ishizuka Yoshihito Nakagawa Mitsuo Nagasaka Masami Iwata Ichiro Hirata Tomiyasu Arisawa

BACKGROUND Genetic factors related to DNA repair or xenobiotic pathways might confer different degrees of susceptibility to Helicobacter pylori (H. pylori)-related gastric carcinogenesis. The association between XRCC1 Arg399Gln, and Arg194Trp, GSTP1 Ile104Val, and GSTT1, GSTM1 null polymorphisms with gastric intestinal metaplasia, severity of histological gastritis, and peptic ulcer diseases we...

Journal: :Carcinogenesis 2002
Kroum Alexandrov Ingolf Cascorbi Margarita Rojas Guy Bouvier Erik Kriek Helmut Bartsch

Benzo[a]pyrene diol epoxide (BPDE)-DNA adducts are involved in the induction of p53 mutations and probably in the causation of human lung cancer associated with cigarette smoking. The ratio between CYP1A1 and GST enzyme activities is a critical determinant of the target dose of carcinogenic BPDE and other DNA-reactive PAH metabolites. In this review, we summarize the published data on modulatio...

Journal: :The American journal of clinical nutrition 2005
Amy V Gasper Ahmed Al-Janobi Julie A Smith James R Bacon Paul Fortun Clare Atherton Moira A Taylor Christopher J Hawkey David A Barrett Richard F Mithen

BACKGROUND Broccoli consumption is associated with a reduction in the risk of cancer, particularly in persons with a functional glutathione S-transferase M1 allele, as opposed rotrose whose GSTM1 gene has been deleted. Sulforaphane, the major isothiocyanate derived from 4-methylsulfinylbutyl glucosinolate, is thought to be the main agent conferring protection. OBJECTIVE We compared sulforapha...

Journal: :Carcinogenesis 2003
Rayjean J Hung Paolo Boffetta Jürgen Brockmöller Dorota Butkiewicz Ingolf Cascorbi Margie L Clapper Seymour Garte Aage Haugen Ari Hirvonen Sisko Anttila Ivan Kalina Loïc Le Marchand Stephanie J London Agneta Rannug Marjorie Romkes Jan Salagovic Bernadette Schoket Laura Gaspari Emanuela Taioli

Polymorphisms for genes encoding the metabolic enzymes cytochrome P450 1A1 (CYP1A1) and glutathione S-transferase M1 (GSTM1) might contribute to the variability in individual susceptibility to lung cancer. The role of CYP1A1 and GSTM1 in lung carcinogenesis might be more important at low levels of exposure to carcinogens. Non-smokers represent a population at low exposure, however, they are oft...

Journal: :Chest 2005
Yung-Ling Lee Tzuen-Ren Hsiue Yeu-Chin Lee Ying-Chu Lin Yueliang Leon Guo

STUDY OBJECTIVES Genetic polymorphisms in the glutathione S-transferase P1 gene (GSTP1) and the glutathione S-transferase M1 gene (GSTM1) have been implicated as risk factors for asthma. However, their roles in asthma pathogenesis and the interaction between these two genes have not been extensively investigated. This study, therefore, examined the relationship among GSTP1 and GSTM1 genotypes a...

2016
Haixia Liu Wei Shi Lianli Zhao Dianlu Dai Jinghua Gao Xiangjun Kong

BACKGROUND Gastric and colorectal cancers remain the major causes of cancer-related death. Although chemotherapy improves the prognosis of the patients with gastrointestinal cancers, some patients do not benefit from therapy and are exposed to the adverse effects. The polymorphisms in genes including GSTM1 and GSTT1 have been explored to predict therapeutic efficacy; however, the results were i...

Journal: :American journal of epidemiology 2002
Lawrence S Engel Emanuela Taioli Ruth Pfeiffer Montserrat Garcia-Closas Pamela M Marcus Qing Lan Paolo Boffetta Paolo Vineis Herman Autrup Douglas A Bell Robert A Branch Jürgen Brockmöller Ann K Daly Susan R Heckbert Ivan Kalina Daehee Kang Takahiko Katoh Amalia Lafuente Henry J Lin Marjorie Romkes Jack A Taylor Nathaniel Rothman

Smoking is a known risk factor for bladder cancer. The product of the GSTM1 gene, glutathione S-transferase M1 (GSTM1), is involved in the detoxification of polycyclic aromatic hydrocarbons found in tobacco smoke; a homozygous deletion of this gene in approximately 50% of Caucasians and Asians results in a lack of GSTM1 enzyme activity. Most studies examining the relation between bladder cancer...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2006
Edward S Peters Michael D McClean Carmen J Marsit Brian Luckett Karl T Kelsey

Investigations of the ability of polymorphisms in the GSTM1, GSTT1, and GSTP1 genes to alter susceptibility to head and neck squamous cell carcinoma (HNSCC) have examined gene-environment interaction in their detoxification of tobacco-associated carcinogens. Little work has been done to ask if these variant genes also modify the interaction of tobacco and alcohol in the development of HNSCC. To...

Journal: :Journal of the National Cancer Institute 1999
M García-Closas K T Kelsey S E Hankinson D Spiegelman K Springer W C Willett F E Speizer D J Hunter

BACKGROUND The enzymes encoded by the glutathione S-transferase mu 1 (GSTM1) and theta 1 (GSTT1) genes are involved in the metabolism (mainly inactivation, but activation is possible) of a wide range of carcinogens that are ubiquitous in the environment; the enzyme encoded by the GSTT1 gene may also be active in endogenous mutagenic processes. Homozygous deletions of the GSTM1 and GSTT1 genes a...

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