نتایج جستجو برای: lamins

تعداد نتایج: 1470  

Journal: :Biochemical Society transactions 2005
K J Meaburn N Levy D Toniolo J M Bridger

Gene-poor human chromosomes are reproducibly found at the nuclear periphery in proliferating cells. There are a number of inner nuclear envelope proteins that may have roles in chromosome location and anchorage, e.g. emerin and A-type lamins. In the last decade, a number of diseases associated with tissue degeneration and premature aging have been linked with mutations in lamin A or emerin. The...

2014
Eivind Lund Anja R. Oldenburg Philippe Collas

Nuclear lamins contact the genome at the nuclear periphery through large domains and are involved in chromatin organization. Among broad peak calling algorithms available to date, none are suited for mapping lamin-genome interactions genome wide. We disclose a novel algorithm, enriched domain detector (EDD), for analysis of broad enrichment domains from chromatin immunoprecipitation (ChIP)-seq ...

Journal: :Current Biology 2016
Wei Xie Alexandre Chojnowski Thomas Boudier John S.Y. Lim Sohail Ahmed Zheng Ser Colin Stewart Brian Burke

The nuclear lamina is a universal feature of metazoan nuclear envelopes (NEs) [1]. In mammalian cells, it appears as a 10-30 nm filamentous layer at the nuclear face of the inner nuclear membrane (INM) and is composed primarily of A- and B-type lamins, members of the intermediate filament family [2]. While providing structural integrity to the NE, the lamina also represents an important signali...

2017
Raymond Kwan Graham F. Brady Maria Brzozowski Sujith V. Weerasinghe Hope Martin Min-Jung Park Makayla J. Brunt Ram K. Menon Xin Tong Lei Yin Colin L. Stewart M. Bishr Omary

BACKGROUND & AIMS Lamins are nuclear intermediate filament proteins that comprise the major components of the nuclear lamina. Mutations in LMNA, which encodes lamins A/C, cause laminopathies, including lipodystrophy, cardiomyopathy, and premature aging syndromes. However, the role of lamins in the liver is unknown, and it is unclear whether laminopathy-associated liver disease is caused by prim...

Journal: :The Journal of Cell Biology 1996
L Rao D Perez E White

Expression of the adenovirus E1A oncogene stimulates both cell proliferation and p53-dependent apoptosis in rodent cells. p53 implements apoptosis in all or in part through transcriptional activation of bax, the product of which promotes cell death. The adenovirus E1B 19K product is homologous in sequence and in function to Bcl-2, both of which bind to and inhibit the activity of Bax and thereb...

Journal: :Current Opinion in Cell Biology 2004

Journal: :Histochemistry and Cell Biology 2013

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