نتایج جستجو برای: mitochondrial myopathy

تعداد نتایج: 143464  

2016
Amy E. Vincent Yi Shiau Ng Kathryn White Tracey Davey Carmen Mannella Gavin Falkous Catherine Feeney Andrew M. Schaefer Robert McFarland Grainne S. Gorman Robert W. Taylor Doug M. Turnbull Martin Picard

Mitochondrial functions are intrinsically linked to their morphology and membrane ultrastructure. Characterizing abnormal mitochondrial structural features may thus provide insight into the underlying pathogenesis of inherited and acquired mitochondrial diseases. Following a systematic literature review on ultrastructural defects in mitochondrial myopathy, we investigated skeletal muscle biopsi...

Journal: :Sexually transmitted infections 2003
K P Prime S G Edwards M R Pakianathan J L Holton F Scaravilli R F Miller

A 66 year old HIV infected male heavy smoker presented with arthralgia, myalgia, and weight loss which was originally ascribed to nucleoside induced mitochondrial toxicity. Despite withdrawal of antiretroviral therapy a proximal myopathy developed. Further investigation excluded malignancy. Polymyositis was diagnosed on muscle biopsy. The patient recovered completely with oral prednisolone. Thi...

Journal: :International Journal of Otorhinolaryngology and Head and Neck Surgery 2017

Journal: :Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 1983

Journal: :JCSM clinical reports 2021

Background ‘Mitochondrial Myopathy’ (MM) refers to genetically-confirmed Primary Mitochondrial Disease (PMD) that predominantly impairs skeletal muscle function. Validated outcome measures encompassing core MM domains of weakness, fatigue, imbalance, impaired dexterity, and exercise intolerance do not exist. The goal this study was validate clinically-meaningful, quantitative specific MM. Metho...

2012
Daniel R Crooks Thanemozhi G Natarajan Churning Chen Hongzhan Huang Manik C Ghosh Wing-Hang Tong Ronald G Haller Cathy Wu Tracey A Rouault

ISCU Myopathy, a disease characterized by life-long exercise intolerance and impaired mitochondrial oxidative metabolism, is caused by deficiency of the Fe-S cluster scaffold protein ISCU. We performed gene expression analysis on muscle biopsies from ISCU Myopathy patients to elucidate which molecular processes were transcriptionally remodeled in response to impaired Fe-S cluster assembly. We f...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید