نتایج جستجو برای: myelin oligodendrocyte glycoprotein

تعداد نتایج: 105655  

Journal: :the iranian journal of pharmaceutical research 0
akbar karimi department of biology, science and research branch, islamic azad university, tehran, iran. farhad ahmadi department of toxicology and pharmacology, faculty of pharmacy, shahid beheshti university of medical sciences, tehran, iran. kazem parivar department of biology, science and research branch, islamic azad university, tehran, iran. mohammad nabiuni tarbiat moallem university, tehran, iran. saied haghighi department of biology, science and research branch, islamic azad university, tehran, iran. sohrab imani department of biology, science and research branch, islamic azad university, tehran, iran.

multiple sclerosis (ms) is a progressive and autoimmune neurodegenerative disease of the central nervous system (cns). this disease is recognized through symptoms like inflammation, demyelination and the destruction of neurological actions. experimental allergic encephalomyelitis (eae) is a widely accepted animal model for ms. eae is created in animals by injecting the tissue of myelin basic pr...

Journal: :Journal of Immunology 2023

Abstract Multiple Sclerosis is a demyelinating autoimmune disease brought on by self-reactive, myelin-specific T effector cells. Immunosuppressants are the most utilized tool to combat progression; however, they bring unwanted side effects. We have previously published novel gene immunotherapy that uses an adeno-associated virus (AAV) containing full sequence of myelin oligodendrocyte glycoprot...

Journal: :Archives of neurology 2010
Eric C Klawiter Laura Piccio Jeri-Anne Lyons Robert Mikesell Kevin C O'Connor Anne H Cross

OBJECTIVE To evaluate antibodies to myelin oligodendrocyte glycoprotein (MOG) in the serum and cerebrospinal fluid (CSF) of patients with multiple sclerosis (MS) and control individuals. DESIGN Prospective case-control series. SETTING Academic referral center. PATIENTS Twenty-six controls with noninflammatory neurologic disease and 35 patients with MS donated serum and CSF for recombinant...

2015
Hideki Nakajima Masakatsu Motomura Keiko Tanaka Azusa Fujikawa Ruka Nakata Yasuhiro Maeda Tomoaki Shima Akihiro Mukaino Shunsuke Yoshimura Teiichiro Miyazaki Hirokazu Shiraishi Atsushi Kawakami Akira Tsujino

OBJECTIVES To investigate the differences of clinical features, cerebrospinal fluid (CSF), MRI findings and response to steroid therapies between patients with optic neuritis (ON) who have myelin oligodendrocyte glycoprotein (MOG) antibodies and those who have seronegative ON. SETTING We recruited participants in the department of neurology and ophthalmology in our hospital in Japan. METHOD...

Journal: :Clinical and translational discovery 2023

Myelin oligodendrocyte glycoprotein (MOG) antibody-associated disease (MOGAD) is an immune-mediated inflammatory central nervous system (CNS) that has been gradually recognized in recent years and currently considered as a distinct entity distinguished from multiple sclerosis neuromyelitis optica spectrum disorder. Lately, the new diagnostic criteria for MOGAD proposed by International Panel we...

Journal: :Clinical and Experimental Neuroimmunology 2021

Myelin oligodendrocyte glycoprotein (MOG)-associated disease is an autoimmune of the central nervous system, associated with presence immunoglobulin G serum antibodies against MOG. Recent data have allowed characterization clinical spectrum MOG-associated disease, which now considered a new entity, distinct from multiple sclerosis and neuromyelitis optica disorders. Optic neuritis most common p...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2005
M Douglas Benson Mario I Romero Mark E Lush Q Richard Lu Mark Henkemeyer Luis F Parada

The inability of CNS axons to regenerate after traumatic spinal cord injury is due, in part, to the inhibitory effects of myelin. The three major previously identified constituents of this activity (Nogo, myelin-associated glycoprotein, and oligodendrocyte myelin glycoprotein) were isolated based on their potent inhibition of axon outgrowth in vitro. All three myelin components transduce their ...

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