نتایج جستجو برای: myocardial free wall rupture

تعداد نتایج: 849289  

Journal: :Circulation journal : official journal of the Japanese Circulation Society 2004
Nahoko Ikeda Takanori Yasu Norifumi Kubo Taishi Hirahara Yoshitaka Sugawara Nobuhiko Kobayashi Shigemasa Hashimoto Yoshio Tsuruya Mikihisa Fujii Muneyasu Saito

BACKGROUND Cardiac rupture after acute myocardial infarction (AMI) is unpredictable and almost always fatal, so the present study reviewed all the clinical characteristics of patients with cardiac rupture to determine if its occurrence can be predicted. METHODS AND RESULTS The clinical characteristics of 1,296 consecutive AMI patients from January 1992 to February 2002 were retrospectively ev...

2012
Masayuki Goto Eiji Oda Hirooki Matsushita Ken Takarada Makoto Tomita Atsushi Saito Koichi Fuse Satoru Fujita Yoshio Ikeda Hitoshi Kitazawa Minoru Takahashi Masahito Sato Masaaki Okabe Yoshifusa Aizawa

BACKGROUND Apart from the severity of myocardial infarction and coronary artery disease, several predictors of in-hospital death (In-HD) are suggested in patients with acute myocardial infarction (AMI). METHODS We investigated predictors of In-HD and ventricular rupture (VR) including ventricular septal rupture (VSR) and free wall rupture (FWR) with stepwise multivariable logistic regressions...

Journal: :Interactive CardioVascular and Thoracic Surgery 2012

2015
Abdulkadir Ercan Orcun Gurbuz Gencehan Kumtepe Hakan Ozkan Ilker Hasan Karal Yusuf Velioglu Serdar Ener

Introduction. Dissection of the myocardium is a rare form of cardiac rupture, caused by a hemorrhagic dissection among the spiral myocardial fibers, its diagnosis is rarely established before the operation or death, and extremely few cases have been reported in the literature and none of these cases seem to have a history of previous cardiac surgery which makes our report unique. Case Presentat...

Journal: :Physiological reviews 2005
Andrew C Newby

Intimal thickening, the accumulation of cells and extracellular matrix within the inner vessel wall, is a physiological response to mechanical injury, increased wall stress, or chemical insult (e.g., atherosclerosis). If excessive, it can lead to the obstruction of blood flow and tissue ischemia. Together with expansive or constrictive remodeling, the extent of intimal expansion determines fina...

Journal: :Journal of the American College of Cardiology 1994

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