Journal:
:مجله علوم اعصاب شفای خاتم
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stefanie bock institute of translational neurology, department of neurology, university of muenster, muenster, germany
tobias ruck institute of translational neurology, department of neurology, university of muenster, muenster, germany
jonas franz anostic institute, center for nanotechnology, university of muenster, muenster, germany
christoph riethmüller anostic institute, center for nanotechnology, university of muenster, muenster, germany
stefan bittner department of neurology, university medical center of the johannes gutenberg-university mainz, mainz, germany
sven g meuth institute of translational neurology, department of neurology, university of muenster, muenster, germany
kcnk2 was previously shown to regulate immune-cell trafficking into the central nervous system (cns). kcnk2-/- mice demonstrated a more severe disease course in experimental autoimmune encephalomyelitis, an animal model of multiple sclerosis, due to an increased immune-cell migration into the cns. an upregulation of the cellular adhesion molecules icam1 and vcam1 on brain endothelial cells in k...
