نتایج جستجو برای: unknown mutations

تعداد نتایج: 374830  

Hamid Tebyanian, Hamze farhadian Mehdi Behdani Mohamad Reza Imen Shahidi,

Objective: The Rifampicin resistance and susceptibility of Mycobacterium tuberculosis are caused by mutations in the 81-base pair region of the rpoB gene encoding the b-subunit of RNA polymerase. Methods: Isoniazid resistance of M. tuberculosis is related to mutations in inha , oxyR and ahpC genes which 30 to 90 percent of Isoniazid resistance is occurred in 3015 codons of kat...

Journal: :Human molecular genetics 2010
Daniel Calva-Cerqueira Fadi S Dahdaleh George Woodfield Sathivel Chinnathambi Peter L Nagy Joy Larsen-Haidle Ronald J Weigel James R Howe

Juvenile polyposis (JP) is an autosomal dominant hamartomatous polyposis syndrome where affected individuals are predisposed to colorectal and upper gastrointestinal cancer. Forty-five percent of JP patients have mutations or deletions involving the coding regions of SMAD4 and BMPR1A, but the genetic basis of other cases is unknown. We set out to identify the JP gene in a large kindred having 1...

Journal: :Cell systems 2015
Martin L Miller Ed Reznik Nicholas P Gauthier Bülent Arman Aksoy Anil Korkut Jianjiong Gao Giovanni Ciriello Nikolaus Schultz Chris Sander

In cancer genomics, recurrence of mutations in independent tumor samples is a strong indicator of functional impact. However, rare functional mutations can escape detection by recurrence analysis owing to lack of statistical power. We enhance statistical power by extending the notion of recurrence of mutations from single genes to gene families that share homologous protein domains. Domain muta...

Journal: :Cancer discovery 2012
Nicholas J Roberts Yuchen Jiao Jun Yu Levy Kopelovich Gloria M Petersen Melissa L Bondy Steven Gallinger Ann G Schwartz Sapna Syngal Michele L Cote Jennifer Axilbund Richard Schulick Syed Z Ali James R Eshleman Victor E Velculescu Michael Goggins Bert Vogelstein Nickolas Papadopoulos Ralph H Hruban Kenneth W Kinzler Alison P Klein

UNLABELLED Pancreatic cancers are the fourth most-common cause of cancer-related deaths in the Western world, with >200,000 cases reported in 2010. Although up to 10% of these cases occur in familial patterns, the hereditary basis for predisposition in the vast majority of affected families is unknown. We used next-generation sequencing, including whole-genome and whole-exome analyses, and iden...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2001
S K Remold R E Lenski

Numerous studies have shown genotype-by-environment (GxE) interactions for traits related to organismal fitness. However, the genetic architecture of the interaction is usually unknown because these studies used genotypes that differ from one another by many unknown mutations. These mutations were also present as standing variation in populations and hence had been subject to prior selection. B...

2012
Stephanie Hicks Sharon E Plon Marek Kimmel

Background Whole genome and whole exome sequencing projects yield thousands of missense mutations with unknown functionality. Direct estimation of the sensitivity and specificity of bioinformatic algorithms predicting the impact of missense mutations on protein function requires a ‘gold standard’ or set of mutations with known functionality. In the absence of a gold standard, additional statist...

2011
Jenn-Yu Wu Chong-Jen Yu Yeun-Chung Chang Chih-Hsin Yang Jin-Yuan Shih Pan-Chyr Yang

Purpose: Clinical features of epidermal growth factor receptor (EGFR) mutations, L858R, deletions in exon 19, T790M, and insertions in exon 20, in non–small cell lung cancer (NSCLC) are well known. The clinical significance of other uncommon EGFR mutations, such as their association with the effectiveness of EGFR tyrosine kinase inhibitors (TKI), is not well understood. This study aimed to impr...

Journal: :Molecular biology and evolution 2017
Fabien Duveau William Toubiana Patricia J Wittkopp

Variation in gene expression is widespread within and between species, but fitness consequences of this variation are generally unknown. Here, we use mutations in the Saccharomyces cerevisiae TDH3 promoter to assess how changes in TDH3 expression affect cell growth. From these data, we predict the fitness consequences of de novo mutations and natural polymorphisms in the TDH3 promoter. Nearly a...

Journal: :Science 1999
E K Davies A D Peters P D Keightley

Deleterious mutations with very small phenotypic effects could be important for several evolutionary phenomena, but the extent of their contribution has been unknown. Fitness effects of induced mutations in lines of Caenorhabditis elegans were measured using a system for which the number of deleterious point mutations in the DNA can be estimated. In fitness assays, only about 4 percent of the d...

2002
HIDEYUKI AOSHIMA TADASHI YOSHIDA SHlNICHI KAWAI SHINYA KOBAYASHI YUTAKA MIZUSHIMA

Mutations of the thyrotropin receptor (TSH-R) gene have been reported in some cases of hyperthyroidism. We report a case of a family that had a high incidence of hyperthyroidism (6/13) which strongly suggested hereditary factors. We then analyzed whether the family had mutations of the TSH-R gene, No significant mutations in exon 10 of the TSH-R gene were found in the patient by restriction fra...

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