نتایج جستجو برای: ژن asah

تعداد نتایج: 16032  

2017
Liang Xu Yuanjian Fang Xudan Shi Xianyi Chen Jun Yu Zeyu Sun Jianmin Zhang Jing Xu

Background. The ideal management of SAH patients with negative initial DSA findings remains unresolved. Objective. (i) To present risk factors, clinical courses, and outcomes in different types of SAH patients with negative DSA findings; (ii) to explore the differences of basal vein between aSAH patients and NASAH patients; and (iii) to evaluate the value of repeated DSA for these patients. Met...

2018

Aneurysmal subarachnoid hemorrhage (SAH) affects 5-10 per 100,000 individuals per year1 and is associated with high rates of morbidity and mortality [1-4]. Main contributor to poor outcomes after SAH are the early cerebral injury (ECI) caused by the immediate increase in intracranial pressure, decreased cerebral perfusion pressure and global ischemia [1,5-9] and delayed cerebral ischemia (DCI) ...

2012
Sung Hun Kim Pil-Wook Chung Yu Sam Won Young Joon Kwon Hyun Chul Shin Chun Sik Choi

OBJECTIVE Shunt-dependent chronic hydrocephalus (SDCH) is known to be a major complication associated with aneurysmal subarachnoid hemorrhage (aSAH). Old age is known to be one of numerous factors related to the development of SDCH. This study investigated whether postoperative cisternal drainage affects the incidence of SDCH and clinical outcome in elderly patients with aSAH. METHODS Fifty-n...

2013
Kevin R. Carr Scott L. Zuckerman J Mocco

Cerebral vasospasm (CVS) is a potentially lethal complication of aneurysmal subarachnoid hemorrhage (aSAH). Recently, the symptomatic presentation of CVS has been termed delayed cerebral ischemia (DCI), occurring as early as 3-4 days after the sentinel bleed. For the past 5-6 decades, scientific research has promulgated the theory that cerebral vasospasm plays a primary role in the pathology of...

2013
Ioannis Siasios Eftychia Z. Kapsalaki Kostas N. Fountas

Aneurysmal subarachnoid hemorrhage- (aSAH-) associated vasospasm constitutes a clinicopathological entity, in which reversible vasculopathy, impaired autoregulatory function, and hypovolemia take place, and lead to the reduction of cerebral perfusion and finally ischemia. Cerebral vasospasm begins most often on the third day after the ictal event and reaches the maximum on the 5th-7th postictal...

2017
Rachael K. Raw Richard M. Wilkie Mark Mon-Williams Stuart A. Ross Kenan Deniz Tony Goddard Tufail Patankar

Current methods of assessing the outcomes of Intracranial Aneurysm Treatment (IAT) for Aneurysmal Subarachnoid Haemorrhage (aSAH) are relatively insensitive, and thus unlikely to detect subtle deficits. Failures to identify cognitive and motor outcomes of IAT might prevent delivery of optimal post-operative care. There are also concerns over risks associated with using IAT as a preventative mea...

2014
Giulia Cossu Mahmoud Messerer Mauro Oddo Roy Thomas Daniel

Delayed cerebral vasospasm has classically been considered the most important and treatable cause of mortality and morbidity in patients with aneurysmal subarachnoid hemorrhage (aSAH). Secondary ischemia (or delayed ischemic neurological deficit, DIND) has been shown to be the leading determinant of poor clinical outcome in patients with aSAH surviving the early phase and cerebral vasospasm has...

2017
Denny Milakara Cristian Grozea Markus Dahlem Sebastian Major Maren K.L. Winkler Janos Lückl Michael Scheel Vasilis Kola Karl Schoknecht Svetlana Lublinsky Alon Friedman Peter Martus Jed A. Hartings Johannes Woitzik Jens P. Dreier

In many cerebral grey matter structures including the neocortex, spreading depolarization (SD) is the principal mechanism of the near-complete breakdown of the transcellular ion gradients with abrupt water influx into neurons. Accordingly, SDs are abundantly recorded in patients with traumatic brain injury, spontaneous intracerebral hemorrhage, aneurysmal subarachnoid hemorrhage (aSAH) and mali...

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