نتایج جستجو برای: citrullinemia

تعداد نتایج: 234  

Journal: :Advances in peritoneal dialysis. Conference on Peritoneal Dialysis 2009
Lazaro Gotloib Roberto Fudin

The management of a handful of non renal clinical conditions includes peritoneal dialysis (PD) as a therapeutic tool. Peritoneal lavage after abdominal surgery is still performed in infectious peritonitis and cases of necrotizing hemorrhagic pancreatitis. Use of PD in active psoriasis resulted in approximately 40 papers reporting mostly isolated cases and showing both therapeutic success and fa...

2013
Anna Catherine Gunz Karen Choong Murray Potter Elka Miller

The urea-cycle functions to facilitate ammonia excretion, a disruption of which results in the accumulation of toxic metabolites. The neurological outcome of neonatal-onset urea-cycle defects (UCDs) is poor, and there are no good predictors of prognosis beyond ammonia levels at presentation. The role of neuroimaging in the prognosis of neonatal-onset UCDs is unclear. We describe the magnetic re...

Journal: :Pediatric transplantation 2002
Liise K Kayler Robert M Merion Samuel Lee Randall S Sung Jeffrey D Punch Steven M Rudich Jeremiah G Turcotte Darrell A Campbell Ronald Holmes John C Magee

BACKGROUND Liver transplantation for inherited metabolic disorders aims to save the patient's life when the disorder is expected to progress to organ failure, and to cure the underlying metabolic defect. METHODS We retrospectively analyzed 146 pediatric liver transplants (28 metabolic; 118 non-metabolic) performed between 1986 and 2000. RESULTS Twenty-eight transplants were performed in 24 ...

Journal: :The Journal of pediatrics 2001
B Lee J Goss

Long-term correction of urea cycle disorders is achieved by correction of the enzymatic defect in hepatocytes. Currently, orthotopic liver transplantation is the primary means of achieving this correction. In the United States most liver transplantations for urea cycle disorders have been restricted to patients with ornithine transcarbamylase deficiency and argininosuccinic aciduria. However, p...

2017
Masako Miyashita Mami Ishikuro Masahiro Kikuya Chizuru Yamanaka Satoshi Mizuno Masato Nagai Yuki Sato Taku Obara Hirohito Metoki Atsuo Kikuchi Naoki Nakaya Atsushi Hozawa Ichiro Tsuji Nobuo Yaegashi Masayuki Yamamoto Shigeo Kure Shinichi Kuriyama

Citrin deficiency is a congenital metabolic disorder of autosomal recessive inheritance that is caused by mutations in the SLC25A13 gene. The prevalence of homozygotes of these mutations is 1/17,000, with a corresponding prevalence of 0.015 for heterozygotes 5 in Japan. It is difficult to detect citrin deficiency before the onset of adult onset type II citrullinemia (CTLN2) during the asymptoma...

Journal: :The EMBO journal 2001
L Palmieri B Pardo F M Lasorsa A del Arco K Kobayashi M Iijima M J Runswick J E Walker T Saheki J Satrústegui F Palmieri

The mitochondrial aspartate/glutamate carrier catalyzes an important step in both the urea cycle and the aspartate/malate NADH shuttle. Citrin and aralar1 are homologous proteins belonging to the mitochondrial carrier family with EF-hand Ca(2+)-binding motifs in their N-terminal domains. Both proteins and their C-terminal domains were overexpressed in Escherichia coli, reconstituted into liposo...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1967
T A Tedesco W J Mellman

Citrullinemia-is one of several human diseases caused by a defect in the KrebsHenseleit urea cycle. In this disorder a deficiency of liver argininosuccinate synthetase has been reported.' 1\Iorrow and his colleagues have recently described an infant with this condition2 and a fibroblast-like cell line has been derived from a skin biopsy of this child. In studying minimal media requirements for ...

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