نتایج جستجو برای: coenzyme q10

تعداد نتایج: 19286  

Journal: :Movement disorders : official journal of the Movement Disorder Society 2015
Raymond Y Lo Karla P Figueroa Stefan M Pulst Chi-Ying Lin Susan Perlman George Wilmot Christopher Gomez Jeremy Schmahmann Henry Paulson Vikram G Shakkottai Sarah Ying Theresa Zesiewicz Khalaf Bushara Michael Geschwind Guangbin Xia S H Subramony Tetsuo Ashizawa Sheng-Han Kuo

The aim of this study was to investigate the association between drug exposure and disease severity in SCA types 1, 2, 3 and 6. The Clinical Research Consortium for Spinocerebellar Ataxias (CRC-SCA) enrolled 319 participants with SCA1, 2, 3, and 6 from 12 medical centers in the United States and repeatedly measured clinical severity by the Scale for Assessment and Rating of Ataxia (SARA), the U...

2010
Massih Bahar Shahnaz Khaghani Parvin Pasalar Maliheh Paknejad Mohammad Reza Khorramizadeh Hossein Mirmiranpour Siavash Gerayesh Nejad

BACKGROUND/AIMS Matrix Metalloproteinases 2 is a key molecule in cellular invasion and metastasis. Mitochondrial ROS has been established as a mediator of MMP activity. Coenzyme Q(10) contributes to intracellular ROS regulation. Coenzyme Q(10) beneficial effects on cancer are still in controversy but there are indications of Coenzyme Q(10) complementing effect on tamoxifen receiving breast canc...

2010
Eunju Park

The purpose of this study was to investigate the relationship between birth weight and appetite related hormones, insulin resistance, and antioxidant status in overweight children aged 9-10 years. Thirty-four healthy overweight children (18 boys, 16 girls) were evaluated with respect to anthropometric measurement, lipid profiles, leptin, ghrelin, glucose, insulin, C-peptide, lipid soluble vitam...

2016
Abhinav Sharma

Abstract—Heart failure (HF) with either preserved or reduced ejection fraction is associated with increased morbidity and mortality. Evidence-based therapies are often limited by tolerability, hypotension, electrolyte disturbances, and renal dysfunction. Coenzyme Q10 (CoQ10) may represent a safe therapeutic option for patients with HF. CoQ10 is a highly lipophilic molecule with a chemical struc...

Journal: :Journal of the American College of Nutrition 2001
F L Crane

Coenzyme Q is well defined as a crucial component of the oxidative phosphorylation process in mitochondria which converts the energy in carbohydrates and fatty acids into ATP to drive cellular machinery and synthesis. New roles for coenzyme Q in other cellular functions are only becoming recognized. The new aspects have developed from the recognition that coenzyme Q can undergo oxidation/reduct...

2016
Elham Mousavinejad Mohammad Ali Ghaffari Mohammad Reza Afsharmanesh Sahar Sadegh-Nejadi Wesam kooti

This review was conducted in order to determine the effect of Oral Coenzyme Q10 supplement on children diagnosed with Autism spectrum of disorders. Among the most common treatments used for autistic spectrum disorders, vitamin/mineral supplements are considered to be the most common treatments. An oral vitamin/mineral supplementation has benefits in improving the nutritional and metabolic statu...

Journal: :Journal of lipid research 1996
J Lagendijk J B Ubbink W J Vermaak

It has been postulated that lipid peroxidation plays a crucial role in the pathogenesis of atherosclerosis. As CoQ10H2 (reduced form of coenzyme Q10) is easily oxidized to CoQ10 (oxidized form of coenzyme Q10), it has been proposed that the CoQ10H2/CoQ10 ratio may be used as a possible marker of in vivo oxidative stress. However, sample preparation has an important effect on the redox status of...

2015
Sonia Silvestri Patrick Orlando Tatiana Armeni Lucia Padella Francesca Brugè Giovanna Seddaiu Gian Paolo Littarru Luca Tiano

Reactive oxygen species not only cause damage but also have a physiological role in the protection against pathogens and in cell signalling. Mitochondrial nutrients, such as coenzyme Q10 and α-lipoic acid, beside their acknowledged antioxidant activities, show interesting features in relation to their redox state and consequent biological activity. In this study, we tested whether oral suppleme...

2017
Roy Moncayo Helga Moncayo

Laboratory medicine approaches the evaluation of thyroid function mostly through the single determination of the blood level of thyroid stimulating hormone (TSH). Some authors have suggested an upper reference value for TSH of 2.5 mIU/L. This suggestion has not been confirmed by recent clinical studies. These studies have delivered a clinically valid reference range going from 0.3 to 3.5 mIU/L....

Journal: :The Journal of clinical investigation 2011
Saskia F Heeringa Gil Chernin Moumita Chaki Weibin Zhou Alexis J Sloan Ziming Ji Letian X Xie Leonardo Salviati Toby W Hurd Virginia Vega-Warner Paul D Killen Yehoash Raphael Shazia Ashraf Bugsu Ovunc Dominik S Schoeb Heather M McLaughlin Rannar Airik Christopher N Vlangos Rasheed Gbadegesin Bernward Hinkes Pawaree Saisawat Eva Trevisson Mara Doimo Alberto Casarin Vanessa Pertegato Gianpietro Giorgi Holger Prokisch Agnès Rötig Gudrun Nürnberg Christian Becker Su Wang Fatih Ozaltin Rezan Topaloglu Aysin Bakkaloglu Sevcan A Bakkaloglu Dominik Müller Antje Beissert Sevgi Mir Afig Berdeli Seza Varpizen Martin Zenker Verena Matejas Carlos Santos-Ocaña Placido Navas Takehiro Kusakabe Andreas Kispert Sema Akman Neveen A Soliman Stefanie Krick Peter Mundel Jochen Reiser Peter Nürnberg Catherine F Clarke Roger C Wiggins Christian Faul Friedhelm Hildebrandt

Steroid-resistant nephrotic syndrome (SRNS) is a frequent cause of end-stage renal failure. Identification of single-gene causes of SRNS has generated some insights into its pathogenesis; however, additional genes and disease mechanisms remain obscure, and SRNS continues to be treatment refractory. Here we have identified 6 different mutations in coenzyme Q10 biosynthesis monooxygenase 6 (COQ6)...

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