نتایج جستجو برای: exhausted cd4 t cell

تعداد نتایج: 2183294  

2010
Kaori Sakuishi Lionel Apetoh Jenna M. Sullivan Bruce R. Blazar Vijay K. Kuchroo Ana C. Anderson

The immune response plays an important role in staving off cancer; however, mechanisms of immunosuppression hinder productive anti-tumor immunity. T cell dysfunction or exhaustion in tumor-bearing hosts is one such mechanism. PD-1 has been identified as a marker of exhausted T cells in chronic disease states, and blockade of PD-1-PD-1L interactions has been shown to partially restore T cell fun...

Journal: :Journal of immunology 2008
Nienke Vrisekoop Rogier van Gent Anne Bregje de Boer Sigrid A Otto Jan C C Borleffs Radjin Steingrover Jan M Prins Taco W Kuijpers Tom F W Wolfs Sibyl P M Geelen Irma Vulto Peter Lansdorp Kiki Tesselaar José A M Borghans Frank Miedema

It remains uncertain whether full T cell reconstitution can be established in HIV-infected children and adults with long-term sustained virological control by highly active antiretroviral therapy (HAART). In this study, we comprehensively analyzed various phenotypical markers of CD4 T cell recovery. In addition to measuring T cell activation and proliferation markers, CD4 T cell generation and ...

Journal: :Journal of Crohn's and Colitis 2023

Abstract Background Chronic colonic inflammation in inflammatory bowel disease (IBD) patients increases the risk of colitis-associated cancer (CAC). Cancer development CAC is different from that observed sporadic colorectal (CRC). Most studies have focused on role pro-inflammatory immune cell subsets CAC, yet it becoming increasingly clear immunosuppression may also drive progression. We hypoth...

Journal: :Cancer Biology and Medicine 2020

Journal: :reports of biochemistry and molecular biology 0
nader zarinfar tel: +988614173502; fax: +988614173526 ali ganji department of immunology, molecular and medicine research center, school of medicine, arak university of medical sciences, arak, iran. ghasem mosayebi department of immunology, molecular and medicine research center, school of medicine, arak university of medical sciences, arak, iran. ehsanollah ghaznavi-rad molecular and medicine research center, arak university of medical sciences, arak, iran. khadije khosravi department of immunology, molecular and medicine research center, school of medicine, arak university of medical sciences, arak, iran.

background:   brucellosis is one of the most common chronic diseases, with widespread distribution. in spite of cell-mediated immunity (cmi) modulated mainly via activated t-helper type 1 (th1) cells, brucellosis can advance to chronic disease in about 10-30% of cases. regulatory t cells (treg cells) are involved the immune response to brucellosis; however, their role, particularly in the chang...

Journal: :iranian journal of allergy, asthma and immunology 0
hui zhong department of pediatrics, shanghai first people's hospital, shanghai jiaotong university, shanghai, china xiao jian zhou department of pediatrics, shanghai first people's hospital, shanghai jiaotong university, shanghai, china jian gou hong department of pediatrics, shanghai first people's hospital, shanghai jiaotong university, shanghai, china

cytokine production  in response to allergens may influence the development of atopy- predisposing immune responses, initializing the  early programming of  allergy and  asthma. vitamin d  intake  may be  protective  due  to  its  immunoregulatory properties,  that  may contribute  to  influence the expression of  the atopic phenotype  initiated in early life. the objective of our study was to ...

Journal: :Journal of immunology 2009
Eugene V Ravkov Matthew A Williams

The parameters controlling the generation of robust CD4(+) T cell recall responses remain poorly defined. In this study, we compare recall responses by CD4(+) and CD8(+) memory T cells following rechallenge. Homologous rechallenge of mice immune to either lymphocytic choriomeningitis virus or Listeria monocytogenes results in robust CD8(+) T cell recall responses but poor boosting of CD4(+) T c...

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