نتایج جستجو برای: h9c2 cardiac myoblast

تعداد نتایج: 279429  

Journal: :Circulation 2008
Philippe Menasché Ottavio Alfieri Stefan Janssens William McKenna Hermann Reichenspurner Ludovic Trinquart Jean-Thomas Vilquin Jean-Pierre Marolleau Barbara Seymour Jérôme Larghero Stephen Lake Gilles Chatellier Scott Solomon Michel Desnos Albert A Hagège

BACKGROUND Phase I clinical studies have demonstrated the feasibility of implanting autologous skeletal myoblasts in postinfarction scars. However, they have failed to determine whether this procedure was functionally effective and arrhythmogenic. METHODS AND RESULTS This multicenter, randomized, placebo-controlled, double-blind study included patients with left ventricular (LV) dysfunction (...

2010
Xiang Liu Yijiang Chen Yanhu Wu Tuanzhu Ha Chuanfu Li

OBJECTIVE The mechanisms by which lipopolysaccharide (LPS) pretreatment induces cardioprotection following ischaemia/reperfusion (I/R) have not been fully elucidated. We hypothesized that activation of phosphoinositide 3-kinase (PI3K)/Akt and high mobility group box 1 (HMGBx1) signaling plays an important role in LPS-induced cardioprotection. METHODS In in vivo experiments, age- and weight- m...

Journal: :Acta biochimica et biophysica Sinica 2010
Dongli Song Xiaoyu Liu Rujiao Liu Ling Yang Ji Zuo Wen Liu

Connexin 43 (Cx43), known to be the main protein building blocks of gap junctions and hemichannels in mammalian heart, plays an important role in cardiocytes proliferation. Gap junctional intercellular communication has been suggested to be necessary for cellular proliferation and differentiation. However, the effect of Cx43 hemichannel on cardiocytes proliferation and the mechanism remain uncl...

2017
Kyung Hye Lee So-Ra Lee Haneul Cho Jong Shin Woo Jung Hee Kang Yun-Mi Jeong Xian Wu Cheng Woo-Shik Kim Weon Kim

Soluble guanylate cyclase (sGC) has been suggested as a therapeutic target for cardiac ischemia-reperfusion (IR) injury. Until now, the molecular mechanism of BAY 60-2770, a sGC activator, in cardiac IR injury has not been assessed. To identify the cardioprotective effects of BAY 60-2770 in IR-injured rat hearts, IR injury was established by occlusion of LAD for 40 min and reperfusion for 7 day...

2014
A. Popolo M. Pecoraro A. Pinto

Persistent β1-adrenergic stimulation is thought to induce cardiac hypertrophy, alteration in calcium regulation and Cx43 over-expression. Adenosine is a negative feedback inhibitor of adrenergic stimulation in the heart, protecting it from toxic effects of overstimulation. The aim of our study evaluate the effects of adenosine administration on Cx43 and pCx43 expression and [Ca2+]i in Isoproter...

Journal: :Circulation research 2005
M Roselle Abraham Charles A Henrikson Leslie Tung Marvin G Chang Miguel Aon Tian Xue Ronald A Li Brian O' Rourke Eduardo Marbán

Skeletal myoblasts are an attractive cell type for transplantation because they are autologous and resistant to ischemia. However, clinical trials of myoblast transplantation in heart failure have been plagued by ventricular tachyarrhythmias and sudden cardiac death. The pathogenesis of these arrhythmias is poorly understood, but may be related to the fact that skeletal muscle cells, unlike hea...

Journal: :American journal of physiology. Cell physiology 2005
Lucia Formigli Fabio Francini Alessia Tani Roberta Squecco Daniele Nosi Lucia Polidori Silvia Nistri Laura Chiappini Valentina Cesati Alessandra Pacini Avio M Perna Giovanni E Orlandini Sandra Zecchi Orlandini Daniele Bani

The success of cellular cardiomyoplasty, a novel therapy for the repair of postischemic myocardium, depends on the anatomical integration of the engrafted cells with the resident cardiomyocytes. Our aim was to investigate the interaction between undifferentiated mouse skeletal myoblasts (C2C12 cells) and adult rat ventricular cardiomyocytes in an in vitro coculture model. Connexin43 (Cx43) expr...

2017
Samarjit Das Mark Kohr Brittany Dunkerly‐Eyring Dong I. Lee Djahida Bedja Oliver A. Kent Anthony K. L. Leung Jorge Henao‐Mejia Richard A. Flavell Charles Steenbergen

BACKGROUND MicroRNA (miRNA) is a type of noncoding RNA that can repress the expression of target genes through posttranscriptional regulation. In addition to numerous physiologic roles for miRNAs, they play an important role in pathophysiologic processes affecting cardiovascular health. Previously, we reported that nuclear encoded microRNA (miR-181c) is present in heart mitochondria, and import...

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