نتایج جستجو برای: irf7

تعداد نتایج: 556  

Journal: :Journal of virology 2012
Clélia Dental Jonathan Florentin Besma Aouar Francoise Gondois-Rey David Durantel Thomas F Baumert Jacques A Nunes Daniel Olive Ivan Hirsch Ruzena Stranska

Plasmacytoid dendritic cells (pDCs) respond to viral infection by production of alpha interferon (IFN-α), proinflammatory cytokines, and cell differentiation. The elimination of hepatitis C virus (HCV) in more than 50% of chronically infected patients by treatment with IFN-α suggests that pDCs can play an important role in the control of HCV infection. pDCs exposed to HCV-infected hepatoma cell...

Journal: :Journal of immunology 2007
Qingde Zhou Salomon Amar

Porphyromonas gingivalis (P. gingivalis) can trigger an inflammatory condition leading to the destruction of periodontal tissues. However P. gingivalis LPS and its fimbriae (FimA) play different roles compared with the live bacteria in the context of intracellular molecule induction and cytokine secretion. To elucidate whether this difference results from different signaling pathways in host im...

Journal: :PLoS ONE 2009
Hiroyasu Konno Takuya Yamamoto Kohsuke Yamazaki Jin Gohda Taishin Akiyama Kentaro Semba Hideo Goto Atsushi Kato Toshiaki Yujiri Takahiko Imai Yasushi Kawaguchi Bing Su Osamu Takeuchi Shizuo Akira Yasuko Tsunetsugu-Yokota Jun-ichiro Inoue

BACKGROUND In response to viral infection, the innate immune system recognizes viral nucleic acids and then induces production of proinflammatory cytokines and type I interferons (IFNs). Toll-like receptor 7 (TLR7) and TLR9 detect viral RNA and DNA, respectively, in endosomal compartments, leading to the activation of nuclear factor kappaB (NF-kappaB) and IFN regulatory factors (IRFs) in plasma...

2014
Takuo Mizukami Haruka Momose Madoka Kuramitsu Kazuya Takizawa Kumiko Araki Keiko Furuhata Ken J. Ishii Isao Hamaguchi Kazunari Yamaguchi

Vaccines are beneficial and universal tools to prevent infectious disease. Thus, safety of vaccines is strictly evaluated in the preclinical phase of trials and every vaccine batch must be tested by the National Control Laboratories according to the guidelines published by each country. Despite many vaccine production platforms and methods, animal testing for safety evaluation is unchanged thus...

Journal: :Physiological genomics 2014
Jaume Padilla Nathan T Jenkins Pamela K Thorne Jeffrey S Martin R Scott Rector J Wade Davis M Harold Laughlin

We used next-generation RNA sequencing (RNA-Seq) technology on the whole transcriptome to identify genes whose expression is consistently affected by obesity across multiple arteries. Specifically, we examined transcriptional profiles of the iliac artery as well as the feed artery, first, second, and third branch order arterioles in the soleus, gastrocnemius, and diaphragm muscles from obese Ot...

2014
Kimberly L. W. Schultz Patty S. Vernon Diane E. Griffin

Differentiation of Neurons Restricts Arbovirus Replication and Increases 1 Expression of the Alpha Isoform of IRF-7 2 3 Kimberly L. W. Schultz, Patty S. Vernon, and Diane E. Griffin 4 5 W. Harry Feinstone Department of Molecular Microbiology and Immunology, Johns 6 Hopkins Bloomberg School of Public Health, Baltimore, MD 21205 7 8 Running title: Antiviral priming of differentiated neurons 9 10 ...

Journal: :Cell 2007
Daniel Panne Tom Maniatis Stephen C Harrison

Transcriptional activation of the interferon-beta (IFN-beta) gene requires assembly of an enhanceosome containing ATF-2/c-Jun, IRF-3/IRF-7, and NFkappaB. These factors bind cooperatively to the IFN-beta enhancer and recruit coactivators and chromatin-remodeling proteins to the IFN-beta promoter. We describe here a crystal structure of the DNA-binding domains of IRF-3, IRF-7, and NFkappaB, bound...

Journal: :Cell 2008
Effie Apostolou Dimitris Thanos

Transcriptional activation of the IFN-beta gene by virus infection requires the cooperative assembly of an enhanceosome. We report that the stochastic and monoallelic expression of the IFN-beta gene depends on interchromosomal associations with three identified distinct genetic loci that could mediate binding of the limiting transcription factor NF-kappaB to the IFN-beta enhancer, thus triggeri...

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