نتایج جستجو برای: somatic hypermutation

تعداد نتایج: 43405  

2013
Natalie Jäger Matthias Schlesner David T.W. Jones Simon Raffel Jan-Philipp Mallm Kristin M. Junge Dieter Weichenhan Tobias Bauer Naveed Ishaque Marcel Kool Paul A. Northcott Andrey Korshunov Ruben M. Drews Jan Koster Rogier Versteeg Julia Richter Michael Hummel Stephen C. Mack Michael D. Taylor Hendrik Witt Benedict Swartman Dietrich Schulte-Bockholt Marc Sultan Marie-Laure Yaspo Hans Lehrach Barbara Hutter Benedikt Brors Stephan Wolf Christoph Plass Reiner Siebert Andreas Trumpp Karsten Rippe Irina Lehmann Peter Lichter Stefan M. Pfister Roland Eils

Mutation is a fundamental process in tumorigenesis. However, the degree to which the rate of somatic mutation varies across the human genome and the mechanistic basis underlying this variation remain to be fully elucidated. Here, we performed a cross-cancer comparison of 402 whole genomes comprising a diverse set of childhood and adult tumors, including both solid and hematopoietic malignancies...

Journal: :The Journal of biological chemistry 2004
Kefei Yu Feng-Ting Huang Michael R Lieber

Activation-induced deaminase (AID) is required for both immunoglobulin class switch recombination and somatic hypermutation. AID is known to deaminate cytidines in single-stranded DNA, but the relationship of this step to the class switch or somatic hypermutation processes is not entirely clear. We have studied the activity of a recombinant form of the mouse AID protein that was purified from a...

Journal: :The Journal of Experimental Medicine 2003
Sarah K. Dickerson Eleonora Market Eva Besmer F. Nina Papavasiliou

Activation-induced deaminase (AID) is a protein indispensable for the diversification of immunoglobulin (Ig) genes by somatic hypermutation (SHM), class switch recombination (CSR), and gene conversion. To date, the precise role of AID in these processes has not been determined. Here we demonstrate that purified, tetrameric AID can deaminate cytidine residues in DNA, but not in RNA. Furthermore,...

Journal: :The Journal of Experimental Medicine 2000
Margrit Wiesendanger Burkhard Kneitz Winfried Edelmann Matthew D. Scharff

Although the primary function of the DNA mismatch repair (MMR) system is to identify and correct base mismatches that have been erroneously introduced during DNA replication, recent studies have further implicated several MMR components in somatic hypermutation of immunoglobulin (Ig) genes. We studied the immune response in mice deficient in MutS homologue (MSH)3 and MSH6, two mutually exclusiv...

Journal: :PLoS Biology 2004
Hiroshi Arakawa Huseyin Saribasak Jean-Marie Buerstedde

Depending on the species and the lymphoid organ, activation-induced cytidine deaminase (AID) expression triggers diversification of the rearranged immunoglobulin (Ig) genes by pseudo V (psiV) gene- templated gene conversion or somatic hypermutation. To investigate how AID can alternatively induce recombination or hypermutation, psiV gene deletions were introduced into the rearranged light chain...

Journal: :Journal of virology 2001
R S Harris D S Croom-Carter A B Rickinson M S Neuberger

It has been suggested that Epstein-Barr virus (EBV) might suppress antibody maturation either by facilitating bypass of the germinal center reaction or by inhibiting hypermutation directly. However, by infecting the Burkitt's lymphoma (BL) cell line Ramos, which hypermutates constitutively and can be considered a transformed analogue of a germinal center B cell, with EBV as well as by transfect...

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