نتایج جستجو برای: type 1 tyrosinemia

تعداد نتایج: 3647237  

Journal: :Neurochemistry International 2012
Gabriela K. Ferreira Milena Carvalho-Silva Cinara L. Gonçalves Júlia S. Vieira Giselli Scaini Fernando V. Ghedim Pedro F. Deroza Alexandra I. Zugno Talita C.B. Pereira Giovanna M.T. Oliveira Luiza W. Kist Maurício R. Bogo Patrícia F. Schuck Gustavo C. Ferreira Emilio L. Streck

Tyrosinemia is a rare genetic disease caused by mutations on genes that codify enzymes responsible for tyrosine metabolism. Considering that tyrosinemics patients usually present symptoms associated with central nervous system alterations that ranges from slight decreases in intelligence to severe mental retardation, we decided to investigate whether acute and chronic administration of L-tyrosi...

Journal: :Molecules 2008
Kai-jin Wang Ning Li Hu Wang

Two pairs of diastereoisomeric acetylenic norlignan compounds with PhCH(OR(1))CH(OR(2))CH(2)C triple bond CPh skeleta: (1R, 2R)-1-O-methylnyasicoside (1) and (1S, 2R)-1-O-methylnyasicoside (2), and (1R, 2R)-crassifogenin D (3) and (1S, 2R)- crassifogenin D (4), were isolated from the ethanolic extract of rhizomes of Curculigo crassifolia. Compounds 3 and 4 are new and their structures were eluc...

2011
Reto Eggenschwiler Komal Loya Malte Sgodda Francoise André Tobias Cantz

Direct reprogramming of somatic cells into pluripotent cells by retrovirus-mediated expression of OCT4, SOX2, KLF4, and C-MYC is a promising approach to derive disease-specific induced pluripotent stem cells (iPSCs). In this study, we focused on three murine models for metabolic liver disorders: the copper storage disorder Wilson's disease (toxic-milk mice), tyrosinemia type 1 (fumarylacetoacet...

Journal: :Pediatric transplantation 2002
Liise K Kayler Robert M Merion Samuel Lee Randall S Sung Jeffrey D Punch Steven M Rudich Jeremiah G Turcotte Darrell A Campbell Ronald Holmes John C Magee

BACKGROUND Liver transplantation for inherited metabolic disorders aims to save the patient's life when the disorder is expected to progress to organ failure, and to cure the underlying metabolic defect. METHODS We retrospectively analyzed 146 pediatric liver transplants (28 metabolic; 118 non-metabolic) performed between 1986 and 2000. RESULTS Twenty-eight transplants were performed in 24 ...

Journal: :The Journal of nutrition 2007
Kimitoshi Nakamura Yasuhiko Tanaka Hiroshi Mitsubuchi Fumio Endo

Hereditary tyrosinemia I (HT I) is a genetic disorder of tyrosine metabolism characterized by progressive liver damage from infancy and by a high risk for hepatocellular carcinoma. HT I is due to mutations in the fumarylacetoacetate hydrolase (Fah) gene, which encodes the last enzyme in the tyrosine catabolic pathway. Disturbances in tyrosine metabolism lead to increased levels of succinylaceto...

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