Short-lasting unilateral neuralgiform headaches with conjunctival injection and tearing (SUNCT) syndrome is one of the trigeminal autonomic cephalalgias. Headache particularly occurs in the orbital-supraorbital region and lasts for approximately 49 s (1,2). The varicella-zoster virus (VZV) is a member of the herpes virus family, and it can remain latent in the trigeminal nerve ganglion and expa...

Varicella zoster virus-associated neuroretinitis is rare. We report a patient who presented with blurred vision of the left eye and extraocular movement pain. A fundoscopic examination revealed disc edema, hyperemia, and macular edema. The impression was neuroretinitis. Intravenous methylprednisolone pulse therapy was administered. However, visual recovery was incomplete with optical coherence ...

The Varicella Zoster virus, analpha virus can present in two distinct forms. The primary infection, chicken pox or varicella, is a contagious but usually benign illness that occurs in epidemics among the susceptible children. After the initial infection, the virus remains dormant until there is a reactivation, leading to the secondary form of disease called Herpes Zoster. Herpes Zoster (HZ) is ...

Herpes zoster, or shingles, is caused by reactivation of varicella zoster virus, which causes chickenpox. There are an estimated 1 million cases in the Unites States annually, with an individual lifetime risk of 30%. Patients with conditions that decrease cell-mediated immunity are 20 to 100 times more likely to develop herpes zoster. Patients may present with malaise, headache, low-grade fever...

Herpes zoster, also known as shingles, occurs upon reactivation of a primary infection with varicella zoster virus (VZV). Risk factors for reactivation include stress, older age, and immunosuppression, all of which are associated with a decrease in host immunity. Common complications of herpes zoster include scarring and post-herpetic neuralgia (PHN). Cutaneous lesions such as granuloma annular...

Reactivation of latent varicella zoster virus as herpes zoster is thought to result from waning of specific cell-mediated immunity, but little is known about its determinants in individuals with no underlying immunosuppression. We systematically reviewed studies of zoster epidemiology in adults and analysed data from a large morbidity study to identify factors that might be modulated to reduce ...

Herpes zoster is caused by reactivation of varicella zoster virus (VZV) from the dorsal sensory ganglia or the cranial nerve ganglia after a previous primary infection with chickenpox. It is known that pediatric zoster rarely occurs and usually not so severe when it develops, except in immunocompromised children 1) . In particular, herpes zoster in infancy is very rare but can be developed foll...

Ganglia obtained at autopsy were examined by in situ hybridization from one patient with zoster (also called herpes zoster or shingles), two varicella-zoster virus (VZV)-seropositive patients with clinical evidence of zoster, one VZV-seronegative child, and one fetus. Ganglia positive for VZV had a hybridization signal in both neuronal and nonneuronal satellite cells. Ganglia obtained from the ...

An indirect peroxidase technique was developed for determination of IgA antibodies to varicella zoster virus (VZV). The antigen consisted of acetone-fixed trypsinised VZV-infected cells. Rabbit antihuman IgA peroxidase conjugate was used to detect human IgA antibodies bound to viral antigen. In parallel IgG antibodies to VZV were determined by an immunoperoxidase antibody to membrane antigen (I...

Static latency is the hallmark of all herpes viruses. The varicella zoster virus, for instance, causes varicella (chickenpox), and after a latent phase of between 5 and 40 years, it can give rise to herpes zoster (shingles). This latency and the subsequent reactivation has intrigued and puzzled virologists. Although several factors have been suggested, it is unknown what triggers reactivation. ...