نتایج جستجو برای: aβ1

تعداد نتایج: 893  

Journal: :Neurology 2013
Stephanie J B Vos Ineke A van Rossum Frans Verhey Dirk L Knol Hilkka Soininen Lars-Olof Wahlund Harald Hampel Magda Tsolaki Lennart Minthon Giovanni B Frisoni Lutz Froelich Flavio Nobili Wiesje van der Flier Kaj Blennow Robin Wolz Philip Scheltens Pieter Jelle Visser

OBJECTIVE To compare the predictive accuracy of β-amyloid (Aβ)1-42 and total tau in CSF, hippocampal volume (HCV), and APOE genotype for Alzheimer disease (AD)-type dementia in subjects with amnestic mild cognitive impairment (aMCI) and nonamnestic mild cognitive impairment (naMCI). METHODS We selected 399 subjects with aMCI and 226 subjects with naMCI from a multicenter memory clinic-based c...

Journal: :Archives of neurology 2012
Abhay Moghekar Joshua Goh Ming Li Marilyn Albert Richard J O'Brien

OBJECTIVE To determine whether cerebrospinal fluid (CSF) biomarkers for Alzheimer disease fluctuate significantly over time in a cohort of older, mildly symptomatic individuals. DESIGN Biomarker validation in a clinical cohort. SETTING University hospital inpatient unit. PARTICIPANTS Ten patients admitted for CSF drainage for diagnostic purposes. MAIN OUTCOME MEASURES The CSF levels of ...

2016
Xi Wang Xiao-Gang Zhang Ting-Ting Zhou Na Li Chun-Yan Jang Zhi-Cheng Xiao Quan-Hong Ma Shao Li

Aberrant increases in neuronal network excitability may contribute to the cognitive deficits in Alzheimer's disease (AD). However, the mechanisms underlying hyperexcitability are not fully understood. Such overexcitation of neuronal networks has been detected in the brains of APP/PS1 mice. In the present study, using current-clamp recording techniques, we observed that 12 days in vitro (DIV) pr...

Journal: :Journal of Alzheimer's disease : JAD 2015
Ying Sun Anders Bresell Mattias Rantalainen Kina Höglund Thibaud Lebouvier Hugh Salter

Alzheimer's disease (AD) is the most common form of dementia, with no disease-modifying treatment yet available. Early detection of patients at risk of developing AD is of central importance. Blood-based genetic signatures can serve as early detection and as population-based screening tools. In this study, we aimed to identify genetic markers and gene signatures associated with cerebrospinal fl...

Journal: :Scientific reports 2016
Giuseppe Di Fede Marcella Catania Emanuela Maderna Michela Morbin Fabio Moda Laura Colombo Alessandro Rossi Alfredo Cagnotto Tommaso Virgilio Luisa Palamara Margherita Ruggerone Giorgio Giaccone Ilaria Campagnani Massimo Costanza Rosetta Pedotti Matteo Salvalaglio Mario Salmona Fabrizio Tagliavini

We developed a novel therapeutic strategy for Alzheimer's disease (AD) exploiting the properties of a natural variant of Amyloid-β (Aβ) carrying the A2V substitution, which protects heterozygous carriers from AD by its ability to interact with wild-type Aβ, hindering conformational changes and assembly thereof. As prototypic compound we designed a six-mer mutated peptide (Aβ1-6A2V), linked to t...

2018
Gianvito Grasso Martina Rebella Stefano Muscat Umberto Morbiducci Jack Tuszynski Andrea Danani Marco A Deriu

Alzheimer's disease is the most fatal neurodegenerative disorder characterized by the aggregation and deposition of Amyloid β (Aβ) oligomers in the brain of patients. Two principal variants of Aβ exist in humans: Aβ1-40 and Aβ1-42. The former is the most abundant in the plaques, while the latter is the most toxic species and forms fibrils more rapidly. Interestingly, fibrils of Aβ1-40 peptides ...

Journal: :JAMA neurology 2015
Jon B Toledo Maria Bjerke Xiao Da Susan M Landau Norman L Foster William Jagust Clifford Jack Michael Weiner Christos Davatzikos Leslie M Shaw John Q Trojanowski

IMPORTANCE Cerebrospinal fluid (CSF) and positron emission tomographic (PET) amyloid biomarkers have been proposed for the detection of Alzheimer disease (AD) pathology in living patients and for the tracking of longitudinal changes, but the relation between biomarkers needs further study. OBJECTIVE To determine the association between CSF and PET amyloid biomarkers (cross-sectional and longi...

Journal: :Journal of Alzheimer's disease : JAD 2012
Walter Maetzler Markus Langkamp Stefanie Lerche Jana Godau Kathrin Brockmann Alexandra Gaenslen Heiko Huber Isabel Wurster Raphael Niebler Gerhard W Eschweiler Daniela Berg

Reduced levels of naturally occurring autoantibodies against amyloid-β (Aβ) have been described in Alzheimer's disease (AD). Lifetime depression doubles the risk of AD, thus these autoantibodies may also be reduced in this group. We measured serum IgG autoantibody titers against Aβ1-42, S100b and α-synuclein in 214 individuals with depression and 419 controls. Titers against Aβ1-42 were lower i...

2017
Li-Juan Min Yodai Kobayashi Masaki Mogi Kana Tsukuda Akio Yamada Koji Yamauchi Fumiaki Abe Jun Iwanami Jin-Zhong Xiao Masatsugu Horiuchi

There is a growing interest in identifying natural food ingredients that may serve to prevent dementia such as that due to Alzheimer disease (AD). Peptides derived from food proteins have been demonstrated to have various physiological activities such as a hypotensive action. Recent findings have indicated possible associations of hypertension with AD progression, and suggest that angiotensin c...

ژورنال: :کومش 0
علی یعقوبی ali yaghoubi dept. of physical education, faculty of physical education and sport sciences, university of birjand, birjand, iranگروه تربیت بدنی، دانشکده ی تربیت بدنی و علوم ورزشی، دانشگاه بیرجند، بیرجند، ایران مرضیه ثاقب جو marziyeh saghebjoo dept. of physical education, faculty of physical education and sport sciences, university of birjand, birjand, iranگروه تربیت بدنی، دانشکده ی تربیت بدنی و علوم ورزشی، دانشگاه بیرجند، بیرجند، ایران ضیاء فلاح محمدی zia fallah-mohammadi dept. of exercise physiology, faculty of physical education and sport sciences, university of mazandaran, babolsar, iranگروه فیزیولوژی ورزش، دانشکده ی تربیت بدنی و علوم ورزشی، دانشگاه مازندران، بابلسر، ایران مهدی هدایتی mehdi hedayati research institute for endocrine sciences, cellular and molecular research center, shahid beheshti university of medical sciences, tehran, iranپژوهشکده ی علوم غدد درون ریز و متابولیسم، مرکز تحقیقات سلولی و مولکولی، دانشگاه علوم پزشکی شهید بهشتی، تهران، ایران اکبر حاجی زاده مقدم akbar hajizadeh moghaddam dept. of biology, faculty of basic sciences, university mazandaran, babolsar, iranگروه زیست شناسی، دانشکده ی علوم پایه، دانشگاه مازندران، بابلسر، ایران

سابقه و هدف: هدف از تحقیق حاضر بررسی اثر 8 هفته تمرین هوازی بر سطوح آمیلوئید بتا 42 (aβ 1-42)، نپریلیزین و γ-سکرتاز هیپوکامپ موش های نر مدل آلزایمری با تزریق هوموسیستئین بود. مواد و روش ها: بدین منظور 50 سر موش صحرایی نر (سن 12 هفته) در 5 گروه آلزایمر کنترل، آلزایمر تمرین، سالم کنترل، سالم تمرین و شم قرار داده شدند. برای القای آلزایمر، از تزریق هوموسیستئین با دوز 6/0 مولار به درون بطن مغز موش ه...

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