نتایج جستجو برای: cag repeats length

تعداد نتایج: 331727  

2017
William Sproviero Aleksey Shatunov Daniel Stahl Maryam Shoai Wouter van Rheenen Ashley R. Jones Safa Al-Sarraj Peter M. Andersen Nancy M. Bonini Francesca L. Conforti Philip Van Damme Hussein Daoud Maria Del Mar Amador Isabella Fogh Monica Forzan Ben Gaastra Cinzia Gellera Aaron D. Gitler John Hardy Pietro Fratta Vincenzo La Bella Isabelle Le Ber Tim Van Langenhove Serena Lattante Yi-Chung Lee Andrea Malaspina Vincent Meininger Stéphanie Millecamps Richard Orrell Rosa Rademakers Wim Robberecht Guy Rouleau Owen A. Ross Francois Salachas Katie Sidle Bradley N. Smith Bing-Wen Soong Gianni Sorarù Giovanni Stevanin Edor Kabashi Claire Troakes Christine van Broeckhoven Jan H. Veldink Leonard H. van den Berg Christopher E. Shaw John F. Powell Ammar Al-Chalabi

We investigated a CAG trinucleotide repeat expansion in the ATXN2 gene in amyotrophic lateral sclerosis (ALS). Two new case-control studies, a British dataset of 1474 ALS cases and 567 controls, and a Dutch dataset of 1328 ALS cases and 691 controls were analyzed. In addition, to increase power, we systematically searched PubMed for case-control studies published after 1 August 2010 that invest...

Journal: :Neurology 2013
Miriam Moscovitch-Lopatin Rachel E Goodman Shirley Eberly James J Ritch H Diana Rosas Samantha Matson Wayne Matson David Oakes Anne B Young Ira Shoulson Steven M Hersch

OBJECTIVE We measured the levels of mutant huntingtin (mtHtt) and total huntingtin (tHtt) in blood leukocytes from Prospective Huntington At-Risk Observational Study (PHAROS) subjects at 50% risk of carrying the Huntington disease mutation using a homogeneous time-resolved fluorescence (HTRF) assay to assess its potential as a biomarker. METHODS Peripheral blood mononuclear cells from consent...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2005
Andrew J Li Dimitry L Lerner Maria-Emily R Gapuzan Beth Y Karlan

PURPOSE The androgen receptor (AR) harbors a polymorphic CAG repeat sequence in exon 1, coding for a polyglutamine tract whose length inversely correlates with AR transactivation function. AIB1, an AR coactivator, expresses a similar polymorphic glutamine sequence within the carboxyl-terminal coding region. We hypothesized that genotypic variations in the androgen-signaling pathway promote aggr...

Journal: :Human reproduction 2005
I E Aknin-Seifer R L Touraine H Lejeune C Jimenez J Chouteau J P Siffroi K McElreavey T Bienvenu C Patrat R Levy

BACKGROUND Recent data emphasized the implication of polymerase gamma (POLG) CAG repeats in infertility, making it a very attractive gene for study. A comparison of POLG CAG repeats in infertile and fertile men showed a clear association between the absence of the usual 10-CAG allele and male infertility, excluding azoospermia. It has also been suggested that the POLG gene polymorphism should b...

Journal: :Human molecular genetics 2008
Vincent Dion Yunfu Lin Leroy Hubert Robert A Waterland John H Wilson

Expanded CAG repeat tracts are the cause of at least a dozen neurodegenerative disorders. In humans, long CAG repeats tend to expand during transmissions from parent to offspring, leading to an earlier age of disease onset and more severe symptoms in subsequent generations. Here, we show that the maintenance DNA methyltransferase Dnmt1, which preserves the patterns of CpG methylation, plays a k...

Journal: :Journal of tropical pediatrics 2008
Léon Mutesa Geneviève Pierquin Karin Segers Jean François Vanbellinghen Laetitia Gahimbare Vincent Bours

Spinocerebellar ataxia type 2 (SCA2) is an autosomal dominant neurodegenerative disease that results from the expansion of an unstable trinucleotide CAG repeat encoding for a polyglutamine tract. In normal individuals, alleles contain between 14 and 31 CAG repeats, whereas the pathological alleles have more than 35 CAG repeats. The clinical phenotype of SCA2 includes a progressive cerebellar at...

Journal: :Journal of medical genetics 1993
Y P Goldberg S E Andrew J Theilmann B Kremer F Squitieri H Telenius J D Brown M R Hayden

Huntington's disease (HD) is associated with expansion of a CAG repeat in a new gene. We have recently defined a premutation in a paternal allele of 30 to 38 CAG repeats in the HD gene which is greater than that seen in the general population (< 30 repeats) but below the range seen in patients with HD (> 38). These intermediate alleles are unstable during transmission through the germline and i...

Journal: :Journal of medical genetics 1995
V Chan Y L Yu T P Chan B Yip C M Chang M T Wong Y W Chan T K Chan

Allelic frequencies of RFLPs at loci closely linked to the HD gene, D4S95, D4S91, D4S141, and D4S90, were determined in 13 Huntington's disease (HD) patients from nine Chinese families and 129 normal subjects. These were similar for non-HD and HD chromosomes and the HD gene in Chinese is associated with multiple haplotypes. Hence the HD gene probably arose independently in the background haplot...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2002
Ann W Hsing Anand P Chokkalingam Yu-Tang Gao Guan Wu Xin Wang Jie Deng Jiaorong Cheng Isabell A Sesterhenn F Kash Mostofi Tzuying Chiang Yuh-Ling Chen Frank Z Stanczyk Chawnshang Chang

In an earlier report, we showed that a shorter CAG repeat length in the androgen receptor (AR) gene is associated with an increased risk of prostate cancer in China, the population with the lowest reported prostate cancer incidence in the world. Because AR coactivators enhance transactivation of AR, in this report we evaluated the relationship of a CAG/CAA repeat length polymorphism in the AIB1...

2007
Nagehan Ersoy

Huntington’s Disease (HD) is a late-onset and progressive neurodegenerative disease of the central nervous system with autosomal dominant inheritance. The prevalence of the disease is about 1/100 000 among individuals of European descent. The clinical symptoms of HD involve motor dysfunction, behavioural disturbances and cognitive decline. The pathology of HD is restricted to the brain, and the...

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