نتایج جستجو برای: il7r

تعداد نتایج: 206  

2017
Xudan Lei Shijiao Cai Yang Chen Jianlin Cui Yajie Wang Zongjin Li Yuhao Li

Interleukin 7 receptor (IL-7R) has been associated with the pathogenesis of multiple sclerosis (MS), though the mechanisms are not clear. Because myelin expression is highly conserved between zebrafish and mammals, zebrafish have become an ideal model for studying demyelination. We used a transgenic (Tg; mbp:nfsB-egfp) zebrafish line in which oligodendrocytes expressed green fluorescent protein...

Journal: :The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques 2013
Yongqiang Dai Jin Li Xiaonan Zhong Yuge Wang Wei Qiu Zhengqi Lu Aimin Wu Jian Bao Fuhua Peng Xueqiang Hu

BACKGROUND Neuromyelitis optica (NMO) and multiple sclerosis (MS) are chronic neuro-inflammatory diseases believed to arise from complex interactions between environmental and genetic factors. Recently, single nucleotide polymorphisms (SNPs) in interleukin (IL)-2 and -7 receptor alpha genes have been identified as novel susceptibility alleles for MS in genome-wide association studies. However, ...

2017
Ling Li Libin Guo Qingshui Wang Xiaolong Liu Yongyi Zeng Qing Wen Shudong Zhang Hang Fai Kwok Yao Lin Jingfeng Liu

The death-associated protein kinase 1 (DAPK1) can act as an oncogene or a tumor suppressor gene depending on the cellular context as well as external stimuli. Our study aims to investigate the prognostic significance of DAPK1 in liver cancer in both mRNA and protein levels. The mRNA expression of DAPK1 was extracted from the Gene Expression Omnibus database in three independent liver cancer dat...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2011
Hongfang Wang James Zou Bo Zhao Eric Johannsen Todd Ashworth Hoifung Wong Warren S Pear Jonathan Schug Stephen C Blacklow Kelly L Arnett Bradley E Bernstein Elliott Kieff Jon C Aster

Notch1 regulates gene expression by associating with the DNA-binding factor RBPJ and is oncogenic in murine and human T-cell progenitors. Using ChIP-Seq, we find that in human and murine T-lymphoblastic leukemia (TLL) genomes Notch1 binds preferentially to promoters, to RBPJ binding sites, and near imputed ZNF143, ETS, and RUNX sites. ChIP-Seq confirmed that ZNF143 binds to ∼40% of Notch1 sites...

Journal: :Journal of neurochemistry 2004
Ting-Fang Wang Chia-Nung Ding Guey-Shin Wang Shih-Chi Luo Yi-Ling Lin Youlin Ruan Robert Hevner John L R Rubenstein Yi-Ping Hsueh

Tbr-1, a neuron-specific T-box transcription factor, plays a critical role in brain development. Here, we performed a computational search using the non-palindromic T-box binding sequence, namely the non-palindromic T-element, to determine the putative downstream target genes of Tbr-1. More than 20 identified genes containing the non-palindromic T-element in the 5' regulatory region were found ...

2012
SHAI IZRAELI THOMAS MICHIELS STEFAN N. CONSTANTINESCU

Mutational activation of cytokine receptors has been described in solid cancers but has not been generally known to drive acute leukemia. We have recently described a subgroup of acute lymphoblastic leukemia characterized by an aberrant expression of the receptor to the cytokine thymic stromal lymphopoietin (TSLP). This receptor is a heterodimer of CRLF2 and IL7RA. In addition to its aberrant e...

Journal: :Hematological Oncology 2023

Introduction: Central Nervous System (CNS) involvement in Diffuse Large B Cell Lymphoma (DLBCL) is a rare complication associated with bad prognosis. The CNS Prognostic Index identifies patients high risk of relapse; however, its specificity limited and not accurate. Biomarkers that can predict relapse are still missing. For reason, we aimed to characterize the transcriptomic, genomic epigenomi...

Journal: :The New England journal of medicine 2014
Kathryn G Roberts Yongjin Li Debbie Payne-Turner Richard C Harvey Yung-Li Yang Deqing Pei Kelly McCastlain Li Ding Charles Lu Guangchun Song Jing Ma Jared Becksfort Michael Rusch Shann-Ching Chen John Easton Jinjun Cheng Kristy Boggs Natalia Santiago-Morales Ilaria Iacobucci Robert S Fulton Ji Wen Marcus Valentine Cheng Cheng Steven W Paugh Meenakshi Devidas I-Ming Chen Shalini Reshmi Amy Smith Erin Hedlund Pankaj Gupta Panduka Nagahawatte Gang Wu Xiang Chen Donald Yergeau Bhavin Vadodaria Heather Mulder Naomi J Winick Eric C Larsen William L Carroll Nyla A Heerema Andrew J Carroll Guy Grayson Sarah K Tasian Andrew S Moore Frank Keller Melissa Frei-Jones James A Whitlock Elizabeth A Raetz Deborah L White Timothy P Hughes Jaime M Guidry Auvil Malcolm A Smith Guido Marcucci Clara D Bloomfield Krzysztof Mrózek Jessica Kohlschmidt Wendy Stock Steven M Kornblau Marina Konopleva Elisabeth Paietta Ching-Hon Pui Sima Jeha Mary V Relling William E Evans Daniela S Gerhard Julie M Gastier-Foster Elaine Mardis Richard K Wilson Mignon L Loh James R Downing Stephen P Hunger Cheryl L Willman Jinghui Zhang Charles G Mullighan

BACKGROUND Philadelphia chromosome-like acute lymphoblastic leukemia (Ph-like ALL) is characterized by a gene-expression profile similar to that of BCR-ABL1-positive ALL, alterations of lymphoid transcription factor genes, and a poor outcome. The frequency and spectrum of genetic alterations in Ph-like ALL and its responsiveness to tyrosine kinase inhibition are undefined, especially in adolesc...

2016
Stefan Nagel Claudia Pommerenke Corinna Meyer Maren Kaufmann Hans G. Drexler Roderick A.F. MacLeod

Recently, we identified deregulated expression of the B-cell specific transcription factor MEF2C in T-cell acute lymphoid leukemia (T-ALL). Here, we performed sequence analysis of a regulatory upstream section of MEF2C in T-ALL cell lines which, however, proved devoid of mutations. Unexpectedly, we found strong conservation between the regulatory upstream region of MEF2C (located at chromosomal...

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