نتایج جستجو برای: irak4

تعداد نتایج: 227  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2017
Mark M Hughes Peter Lavrencic Rebecca C Coll Thomas Ve Dylan G Ryan Niamh C Williams Deepthi Menon Ashley Mansell Philip G Board Mehdi Mobli Bostjan Kobe Luke A J O'Neill

MyD88 adaptor-like (MAL) is a critical protein in innate immunity, involved in signaling by several Toll-like receptors (TLRs), key pattern recognition receptors (PRRs). Crystal structures of MAL revealed a nontypical Toll/interleukin-1 receptor (TIR)-domain fold stabilized by two disulfide bridges. We therefore undertook a structural and functional analysis of the role of reactive cysteine res...

2012
Young-In Kim Jeoung-Eun Park Ki Han Kwon Cheol Yi Hong Ae-Kyung Yi

As a part of the negative feedback mechanism, CpG DNA induces IRAK-M expression in monocytic cells. In the present study we investigated a biochemical signaling pathway and the transcription factors responsible for CpG DNA-mediated Irak-m gene expression. CpG DNA-induced Irak-m expression did not require new protein synthesis and was regulated at the transcriptional level through an endosomal p...

2013
Tatiana Barichello Jaqueline S. Generoso Lutiana R. Simões Samuel G. Elias João Quevedo

Pneumococcal meningitis is a life-threatening disease characterized by an acute purulent infection affecting the pia mater, the arachnoid, and the subarachnoid spaces. Streptococcus pneumoniae crosses the blood-brain barrier (BBB) by both transcellular traversal and disruption of the intraepithelial tight junctions to allow intercellular traversal. During multiplication, pneumococci release the...

2016
Nan Mu Jintao Gu Tonglie Huang Cun Zhang Zhen Shu Meng Li Qiang Hao Weina Li Wangqian Zhang Jinkang Zhao Yong Zhang Luyu Huang Shuning Wang Xiaohang Jin Xiaochang Xue Wei Zhang Yingqi Zhang

The main etiopathogenesis of rheumatoid arthritis (RA) is overexpressed inflammatory cytokines and tissue injury mediated by persistent NF-κB activation. MicroRNAs widely participate in the regulation of target gene expression and play important roles in various diseases. Here, we explored the mechanisms of microRNAs in RA. We found that microRNA (miR)-10a was downregulated in the fibroblast-li...

2015
Emine Guven-Maiorov Ozlem Keskin Attila Gursoy Carter VanWaes Zhong Chen Chung-Jung Tsai Ruth Nussinov

Activated Toll-like receptors (TLRs) cluster in lipid rafts and induce pro- and anti-tumor responses. The organization of the assembly is critical to the understanding of how these key receptors control major signaling pathways in the cell. Although several models for individual interactions were proposed, the entire TIR-domain signalosome architecture has not been worked out, possibly due to i...

2015
Helene Ingels Lone Schejbel A.C. Lundstedt Lise Jensen Inga A. Laursen Lars P. Ryder Niels H.H. Heegaard Helle Konradsen Jens Jørgen Christensen Carsten Heilmann Hanne V. Marquart

BACKGROUND Recurrent invasive pneumococcal disease (rIPD) occurs mostly in children with an underlying disease, but some cases remain unexplained. Immunodeficiency has been described in children with rIPD, but the prevalence is unknown. We used a nationwide registry of all laboratory-confirmed cases of rIPD to identify cases of unexplained rIPD and examine them for immunodeficiency. METHODS C...

Journal: :Blood 2015
Makon-Sébastien Njock Henry S Cheng Lan T Dang Maliheh Nazari-Jahantigh Andrew C Lau Emilie Boudreau Mark Roufaiel Myron I Cybulsky Andreas Schober Jason E Fish

The blood contains high concentrations of circulating extracellular vesicles (EVs), and their levels and contents are altered in several disease states, including cardiovascular disease. However, the function of circulating EVs, especially the microRNAs (miRNAs) that they contain, are poorly understood. We sought to determine the effect of secreted vesicles produced by quiescent endothelial cel...

Journal: :Blood 2016
François Guilhot

is a survival-enhancing oncoprotein that is frequently overexpressed in ABC-DLBCL by virtue of gene amplification (focal copy number gains) at 18q. The investigators cleverly recapitulated this aspect of the ABC-DLBCL genetic network by combining the MYD88 allele with a newly developed inducible BCL2 allele in double-transgenic mice that developed tumors resembling human ABC-DLBCL with full pen...

2015
Chi Ma Narrissa P. Spies Ting Gong Can Xin Jones Wen-Ming Chu

CpG-ODNs activate dendritic cells (DCs) to produce interferon alpha (IFNα) and beta (IFNβ). Previous studies demonstrated that Toll-like receptor 9 (TLR9) deficient DCs exhibited a residual IFNα response to CpG-A, indicating that yet-unidentified molecules are also involved in induction of IFNα by CpG-A. Here, we report that the catalytic subunit of DNA-dependent protein kinase (DNA-PKcs) but n...

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