نتایج جستجو برای: splenocyte

تعداد نتایج: 796  

Journal: :Bulletin of Russian State Medical University 2022

Mitochondrial dysfunctions, which underlie many systemic diseases in animals and humans, may arise from accumulation of mutations the mitochondrial genome. PolG-alpha enzyme encoded by Polg gene is crucial for replication repair The aim this study was to assess possible role dysfunctions using vitro vivo animal models. experiments involved transgenic mice with inducible expression mutant varian...

2014
Jing-Ning Huang Ming-Chin Tsai Shun-Lung Fang Margaret Dah-Tsyr Chang Yu-Rou Wu Jaw-Ji Tsai Lin-Shien Fu Heng-Kuei Lin Yi-Jun Chen Tsai-Wei Li

BACKGROUND We evaluated the effects of T helper cell differentiation in a mite-allergic animal model treated with inhaled heparins of different molecular weight. METHOD BALB/c mice were divided into four groups: 1. Control, 2. Mite intratracheal (mIT), 3. Inhaled heparin (hIN), 4. Inhaled low-molecular-weight heparin (lmwhIN). Groups 2, 3, and 4 were sensitized twice with Der p allergen subcu...

Journal: :Blood 2001
M Cetkovic-Cvrlje B A Roers B Waurzyniak X P Liu F M Uckun

To prevent the development of acute graft-versus-host disease (GVHD) in lethally irradiated C57BL/6 (H-2b) recipient mice transplanted with bone marrow-splenocyte grafts from major histocompatibility complex (MHC) disparate BALB/c mice (H-2d), recipient mice were treated with the rationally designed JAK3 inhibitor WHI-P131 [4-(4'-hydroxyphenyl)-amino-6,7-dimethoxyquinazoline] (20 mg/kg, 3 times...

Journal: :Journal of immunology 2010
Aurore Dubois Nathalie Deruytter Brigitte Adams Akira Kanda Sandrine Delbauve Sebastien Fleury David Torres Angélique François Michel Pétein Michel Goldman David Dombrowicz Véronique Flamand

Th2-biased immune responses characterizing neonates may influence the later onset of allergic disease. The contribution of regulatory T cell populations in the prevention of Th2-driven pathologies in early life is poorly documented. We investigated the potential of CD8(+) T cells stimulated at birth with alloantigens to modulate the development of allergic airway inflammation. Newborn mice were...

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