نتایج جستجو برای: congenital hyperinsulinism

تعداد نتایج: 124753  

2017
Indraneel Banerjee Diva De Leon Mark J. Dunne

We have recently published on the limited effectiveness of sirolimus as a treatment option for hypoglycaemia as a consequence of hyperinsulinism. Our data oppose the view that mTOR inhibitors provide new opportunities for the treatment of patients with hyperinsulinism. We are not convinced by the argument that any benefit for some patients outweighs the potential and later long-term problems th...

Journal: :Diabetes 2008
Maha Abdulhadi-Atwan Jeremy Bushmann Sharona Tornovsky-Babaey Avital Perry Abdulsalam Abu-Libdeh Benjamin Glaser Show-Ling Shyng David H. Zangen

OBJECTIVE Congenital hyperinsulinism, usually associated with severe neonatal hypoglycemia, may progress to diabetes, typically during the 4th decade of life in nonpancreatectomized patients. We aimed to genotype the ATP-sensitive K(+) channel in a 10.5-year-old girl presenting with overt diabetes following hyperinsulinism in infancy. RESEARCH DESIGN AND METHODS A female aged 10.5 years prese...

Journal: :Pediatrics 2005
Irina Giurgea Tim Ulinski Guy Touati Christine Sempoux Fanny Mochel Francis Brunelle Jean-Marie Saudubray Claire Fekete Pascale de Lonlay

Clinical history and inappropriate insulin secretion during hypoglycemic episodes permit the diagnosis of hyperinsulinism. We report 2 cases of factitious hyperinsulinism leading to partial pancreatectomy. Case 1 was an 8-year-old girl who presented with severe hypoglycemia and elevated insulin and C-peptide levels. Catheterization of pancreatic veins was performed to localize the excess insuli...

Journal: :The Journal of clinical endocrinology and metabolism 2001
C MacMullen J Fang B Y Hsu A Kelly P de Lonlay-Debeney J M Saudubray A Ganguly T J Smith C A Stanley

The hyperinsulinism/hyperammonemia (HI/HA) syndrome is a form of congenital hyperinsulinism in which affected children have recurrent symptomatic hypoglycemia together with asymptomatic, persistent elevations of plasma ammonium levels. We have shown that the disorder is caused by dominant mutations of the mitochondrial enzyme, glutamate dehydrogenase (GDH), that impair sensitivity to the allost...

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