نتایج جستجو برای: hdl3 and apoa

تعداد نتایج: 16827413  

Journal: :The Journal of biological chemistry 1989
Z H Beg J A Stonik J M Hoeg S J Demosky T Fairwell H B Brewer

In vitro phosphorylation of purified human plasma apolipoprotein A-I (apoA-I) by a recently characterized Ca2+/calmodulin-dependent kinase (Beg, Z. H., Stonik, J. A., and Brewer, H. B., Jr. (1987) J. Biol. Chem. 262, 13228-13240) was time-, Ca2+-, and calmodulin-dependent. Maximal phosphorylation of human apoA-I revealed a stoichiometry of approximately 1 mol of PO4/mol of apoA-I. Phosphorylati...

Journal: :Circulation Research 2006

2012
Dong Won Yi Dong Wook Jeong Sang Yeoup Lee Seok Man Son Yang Ho Kang

BACKGROUND Apolipoprotein A-II (apoA-II) is the second-most abundant apolipoprotein in human high-density lipoprotein and its role in cardio metabolic risk is not entirely clear. It has been suggested to have poor anti-atherogenic or even pro-atherogenic properties, but there are few studies on the possible role of apoA-II in Asian populations. The aim of this study is to evaluate the role of a...

2014
Sophie Stukas Jerome Robert Michael Lee Iva Kulic Michael Carr Katherine Tourigny Jianjia Fan Dhananjay Namjoshi Kalistyne Lemke Nicole DeValle Jeniffer Chan Tammy Wilson Anna Wilkinson Rafi Chapanian Jayachandran N. Kizhakkedathu John R. Cirrito Michael N. Oda Cheryl L. Wellington

BACKGROUND Brain lipoprotein metabolism is dependent on lipoprotein particles that resemble plasma high-density lipoproteins but that contain apolipoprotein (apo) E rather than apoA-I as their primary protein component. Astrocytes and microglia secrete apoE but not apoA-I; however, apoA-I is detectable in both cerebrospinal fluid and brain tissue lysates. The route by which plasma apoA-I enters...

Journal: :Arteriosclerosis, Thrombosis, and Vascular Biology 2005

Journal: :The Biochemical journal 2012
Shobini Jayaraman Giorgio Cavigiolio Olga Gursky

HDL (high-density lipoproteins) remove cell cholesterol and protect from atherosclerosis. The major HDL protein is apoA-I (apolipoprotein A-I). Most plasma apoA-I circulates in lipoproteins, yet ~5% forms monomeric lipid-poor/free species. This metabolically active species is a primary cholesterol acceptor and is central to HDL biogenesis. Structural properties of lipid-poor apoA-I are unclear ...

Journal: :Translational research : the journal of laboratory and clinical medicine 2013
Hamid Moradi Hamid M Said Nosratola D Vaziri

Chronic kidney disease is associated with premature death from cardiovascular disease, which is, in part, driven by high density lipoprotein deficiency and dysfunction. One of the main causes of high density lipoprotein deficiency in chronic kidney disease is diminished plasma apolipoprotein (Apo)A-I level. Plasma ApoA-I is reduced in dialysis patients and hepatic ApoA-I messenger RNA (mRNA) is...

Journal: :Journal of lipid research 1988
P de Knijff M Rosseneu U Beisiegel W de Keersgieter R R Frants L M Havekes

Recently, we determined the apolipoprotein E (apoE) phenotype distribution in 2,000 randomly selected 35-year-old male individuals by slab gel isoelectric focusing of delipidated plasma samples, followed by immunoblotting using anti-apoE antiserum. These blots have been successfully re-used for immunovisualization of apoA-IV isoelectric focusing patterns. In a population sample of 1,393 individ...

Journal: :Journal of lipid research 2006
Stefan F C Vaessen Frank G Schaap Jan-Albert Kuivenhoven Albert K Groen Barbara A Hutten S Matthijs Boekholdt Hiroaki Hattori Manjinder S Sandhu Sheila A Bingham Robert Luben Jutta A Palmen Nicholas J Wareham Steve E Humphries John J P Kastelein Philippa J Talmud Kay-Tee Khaw

In mouse models, apolipoprotein A-V (apoA-V) exhibits triglyceride (TG)-lowering effects. We investigated the apoA-V/TG relationship and the association of apoA-V with coronary artery disease (CAD) risk by determining serum apoA-V levels and genotypes in a nested case-control (n = 1,034/2,031) study. Both univariate and multivariate apoA-V levels showed no association with future CAD (P = 0.4 a...

Journal: :Journal of lipid research 2013
Panagiotis Fotakis Ioanna Tiniakou Andreas K Kateifides Christina Gkolfinopoulou Angeliki Chroni Efstratios Stratikos Vassilis I Zannis Dimitris Kardassis

We studied the significance of four hydrophobic residues within the 225-230 region of apoA-I on its structure and functions and their contribution to the biogenesis of HDL. Adenovirus-mediated gene transfer of an apoA-I[F225A/V227A/F229A/L230A] mutant in apoA-I⁻/⁻ mice decreased plasma cholesterol, HDL cholesterol, and apoA-I levels. When expressed in apoA-I⁻/⁻ × apoE⁻/⁻ mice, approximately 40%...

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