نتایج جستجو برای: lats

تعداد نتایج: 350  

Journal: :Cell 2011
Michelangelo Cordenonsi Francesca Zanconato Luca Azzolin Mattia Forcato Antonio Rosato Chiara Frasson Masafumi Inui Marco Montagner Anna R. Parenti Alessandro Poletti Maria Grazia Daidone Sirio Dupont Giuseppe Basso Silvio Bicciato Stefano Piccolo

Cancer stem cells (CSCs) are proposed to drive tumor initiation and progression. Yet, our understanding of the cellular and molecular mechanisms that underlie CSC properties is limited. Here we show that the activity of TAZ, a transducer of the Hippo pathway, is required to sustain self-renewal and tumor-initiation capacities in breast CSCs. TAZ protein levels and activity are elevated in prosp...

2017
Sung Jun Bae Lisheng Ni Adam Osinski Diana R Tomchick Chad A Brautigam Xuelian Luo

The Hippo pathway controls tissue growth and homeostasis through a central MST-LATS kinase cascade. The scaffold protein SAV1 promotes the activation of this kinase cascade, but the molecular mechanisms remain unknown. Here, we discover SAV1-mediated inhibition of the PP2A complex STRIPAKSLMAP as a key mechanism of MST1/2 activation. SLMAP binding to autophosphorylated MST2 linker recruits STRI...

Journal: :Genes & development 2015
Noa Furth Noa Bossel Ben-Moshe Yair Pozniak Ziv Porat Tamar Geiger Eytan Domany Yael Aylon Moshe Oren

p53 is a pivotal tumor suppressor and a major barrier against cancer. We now report that silencing of the Hippo pathway tumor suppressors LATS1 and LATS2 in nontransformed mammary epithelial cells reduces p53 phosphorylation and increases its association with the p52 NF-κB subunit. Moreover, it partly shifts p53's conformation and transcriptional output toward a state resembling cancer-associat...

Journal: :Blood 2015
Johanna Gebhart Florian Posch Silvia Koder Thomas Perkmann Peter Quehenberger Claudia Zoghlami Cihan Ay Ingrid Pabinger

Data on the clinical course of lupus anticoagulant (LA)-positive individuals with or without thrombotic manifestations or pregnancy complications are limited. To investigate mortality rates and factors that might influence mortality, we conducted a prospective observational study of LA-positive individuals. In total, 151 patients (82% female) were followed for a median of 8.2 years; 30 of the p...

2018
Jimyung Seo Joon Kim

The Hippo signaling pathway controls nuclear accumulation and stability of the transcriptional coregulator YAP and its paralog TAZ. The activity of Hippo-YAP signaling is influenced not only by biochemical signals, but also by cell shape and mechanical tension transmitted through cell-cell junctions and cell-matrix adhesions. Data accumulated thus far indicates that the actin cytoskeleton is a ...

2017
Jung-Soon Mo

The Hippo signaling pathway plays an essential role in adult-tissue homeostasis and organ-size control. In Drosophila and vertebrates, it consists of a highly conserved kinase cascade, which involves MST and Lats that negatively regulate the activity of the downstream transcription coactivators, YAP and TAZ. By interacting with TEADs and other transcription factors, they mediate both proliferat...

2017
Hung Thanh Nguyen Jan-Michael Kugler Stephen M Cohen

The YAP and TAZ transcriptional coactivators promote oncogenic transformation. Elevated YAP/TAZ activity has been documented in human tumors. YAP and TAZ are negatively regulated by the Hippo tumor suppressor pathway. The activity and stability of several Hippo pathway components, including YAP/TAZ, is regulated by ubiquitin mediated protein turnover and several ubiquitin ligase complexes have ...

Journal: :Genes & development 2013
Fa-Xing Yu Yifan Zhang Hyun Woo Park Jenna L Jewell Qian Chen Yaoting Deng Duojia Pan Susan S Taylor Zhi-Chun Lai Kun-Liang Guan

The Hippo tumor suppressor pathway plays an important role in tissue homeostasis that ensures development of functional organs at proper size. The YAP transcription coactivator is a major effector of the Hippo pathway and is phosphorylated and inactivated by the Hippo pathway kinases Lats1/2. It has recently been shown that YAP activity is regulated by G-protein-coupled receptor signaling. Here...

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