نتایج جستجو برای: neurodegeneration with brain iron accumulation

تعداد نتایج: 9480785  

Journal: :Journal of ultrasound in medicine : official journal of the American Institute of Ultrasound in Medicine 2014
Milija D Mijajlovic Georgios Tsivgoulis Nadezda Sternic

Transcranial sonography is a highly sensitive noninvasive sonographic method for detection of early and specific echogenic changes in basal ganglia of patients with some neurodegenerative diseases. Transcranial sonography showed substantia nigra hyperechogenicity as a typical echo feature in idiopathic Parkinson disease and lenticular nucleus hyperechogenicity as a characteristic finding in aty...

Journal: :Journal of neural transmission. Supplementum 2006
M Gerlach K L Double M B H Youdim P Riederer

Histopathological, biochemical and in vivo brain imaging techniques, such as magnetic resonance imaging and transcranial sonography, revealed a consistent increase of substantia nigra (SN) iron in Parkinson's disease (PD). Increased iron deposits in the SN may have genetic and non-genetic causes. There are several rare movement disorders associated with neurodegeneration, and genetic abnormalit...

2015
Wei Li Holly J. Garringer Charles B. Goodwin Briana Richine Anthony Acton Natalia VanDuyn Barry B. Muhoberac Jose Irimia-Dominguez Rebecca J. Chan Munro Peacock Richard Nass Bernardino Ghetti Ruben Vidal

Ferritin, a 24-mer heteropolymer of heavy (H) and light (L) subunits, is the main cellular iron storage protein and plays a pivotal role in iron homeostasis by modulating free iron levels thus reducing radical-mediated damage. The H subunit has ferroxidase activity (converting Fe(II) to Fe(III)), while the L subunit promotes iron nucleation and increases ferritin stability. Previous studies on ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2008
Ruben Vidal Leticia Miravalle Xiaoying Gao Ana G Barbeito Martin A Baraibar Shahryar K Hekmatyar Mario Widel Navin Bansal Marie B Delisle Bernardino Ghetti

Increased iron levels and iron-mediated oxidative stress play an important role in the pathogenesis of many neurodegenerative diseases. The finding that mutations in the ferritin light polypeptide (FTL) gene cause a neurodegenerative disease known as neuroferritinopathy or hereditary ferritinopathy (HF) provided a direct connection between abnormal brain iron storage and neurodegeneration. HF i...

Journal: :Biochemical Society transactions 2008
Ilaria Pelizzoni Romina Macco Daniele Zacchetti Fabio Grohovaz Franca Codazzi

Iron and calcium are required for general cellular functions, as well as for specific neuronal-related activities. However, a pathological increase in their levels favours oxidative stress and mitochondrial damage, leading to neuronal death. Neurodegeneration can thus be determined by alterations in ionic homoeostasis and/or pro-oxidative-antioxidative equilibrium, two conditions that vary sign...

Journal: :Molecular pharmacology 2013
Erwann Rousseau Patrick P Michel Etienne C Hirsch

Previous studies on postmortem human brain tissue have shown that the iron-binding glycoprotein lactoferrin is upregulated in dopamine (DA) neurons resistant to degeneration in Parkinson disease (PD). To study how this could possibly relate to disease progression, we used midbrain cultures and experimental settings that model the progressive loss of DA neurons in this disorder. Human lactoferri...

2016
Maiko Hayashida Sadayuki Hashioka Hiroyuki Miki Michiharu Nagahama Rei Wake Tsuyoshi Miyaoka Jun Horiguchi

Aceruloplasminemia is an autosomal recessive disorder of iron metabolism caused by mutations in the ceruloplasmin gene. Its prevalence is 1 in 2,000,000 people in Japan. This is a disorder of neurodegeneration with iron accumulation in the brain revealed by MRI. The iron overload induces oxidative stress and generation of reactive oxygen species, which triggers a cascade of pathological events ...

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