نتایج جستجو برای: pkd1

تعداد نتایج: 895  

Journal: :Circulation research 2015
Philipp Du Bois Cristina Pablo Tortola Doerte Lodka Melanie Kny Franziska Schmidt Kunhua Song Sibylle Schmidt Rhonda Bassel-Duby Eric N Olson Jens Fielitz

RATIONALE Skeletal muscle wasting with accompanying cachexia is a life threatening complication in congestive heart failure. The molecular mechanisms are imperfectly understood, although an activated renin-angiotensin aldosterone system has been implicated. Angiotensin (Ang) II induces skeletal muscle atrophy in part by increased muscle-enriched E3 ubiquitin ligase muscle RING-finger-1 (MuRF1) ...

2014
Atousa Hafizi Saeid Reza Khatami Hamid Galehdari Gholamreza Shariati Ali Hossein Saberi Mohammad Hamid

INTRODUCTION Autosomal dominant polycystic kidney disease (ADPKD) is one of the most common genetic kidney disorders with the incidence of 1 in 1,000 births. ADPKD is genetically heterogeneous with two genes identified: PKD1 (16p13.3, 46 exons) and PKD2 (4q21, 15 exons). Eighty five percent of the patients with ADPKD have at least one mutation in the PKD1 gene. Genetic studies have demonstrated...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2007
Sabrine Hassane Nanna Claij Irma S Lantinga-van Leeuwen J Conny Van Munsteren Natascha Van Lent Roeland Hanemaaijer Martijn H Breuning Dorien J M Peters Marco C DeRuiter

OBJECTIVE Autosomal Dominant Polycystic Kidney Disease (ADPKD) is a multi-system disorder characterized by progressive cyst formation in the kidneys. Serious complications of ADPKD are intracranial and aortic aneurysms. The condition is mainly caused by mutations in the PKD1 or PKD2 gene. We have carefully analyzed vascular remodeling in hypomorphic Pkd1(nl/nL) mouse model with dissecting aneur...

Journal: :Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 1996
C Löhning M Pohlschmidt M A Glücksmann-Kuis G Duyk P Bork M O Schneider S T Reeders A M Frischauf

The complete sequence of the polycystic kidney disease gene (PKD1) and its transcript have been described. The predicted protein is not a member of a previously described gene family, but contains several structural motifs that are present in proteins of known function. Most of these domains are present in the extracellular parts of proteins involved in interactions with other proteins and carb...

2017
Samantha J. England Paul C. Campbell Santanu Banerjee Annika J. Swanson Katharine E. Lewis

Polycystic kidney disease (PKD) proteins are trans-membrane proteins that have crucial roles in many aspects of vertebrate development and physiology, including the development of many organs as well as left-right patterning and taste. They can be divided into structurally-distinct PKD1-like and PKD2-like proteins and usually one PKD1-like protein forms a heteromeric polycystin complex with a P...

ژورنال: :مجله علمی - پژوهشی دانشگاه علوم پزشکی سبزوار 2015
محسن اخوان سپهی حسین ثقفی سید مجید موسوی موحد محمدرضا حایری محمد قره بگلو

زمینه و هدف: بیماری کلیهی پلیکیستیک (polycystic kidney disease=pkd) جزء بیماریهای شایع ارثی  است که در آن قشر و مدولای کلیه با تعداد زیادی کیست اشغال می شود و درنهایت در سنین میانسالی و پس ازآن منجر به esrd می­گردد. نحوه­ی انتقال آن به صورت اتوزومال غالب یا اتوزومال مغلوب است. نوع اتوزومال غالب بیماری شایعترین بیماری ارثی کلیه انسان می باشد که بیشتر در افراد بالغ بروز می کند. در این بیماری ارثی...

2014
Zhousheng Xiao Li Cao Yingjuan Liang Jinsong Huang Amber Rath Stern Mark Dallas Mark Johnson Leigh Darryl Quarles

Polycystin-1 (Pkd1) interacts with polycystin-2 (Pkd2) to form an interdependent signaling complex. Selective deletion of Pkd1 in the osteoblast lineage reciprocally regulates osteoblastogenesis and adipogenesis. The role of Pkd2 in skeletal development has not been defined. To this end, we conditionally inactivated Pkd2 in mature osteoblasts by crossing Osteocalcin (Oc)-Cre;Pkd2+/null mice wit...

2015
Attila Varga Pál Gyulavári Zoltán Greff Krisztina Futosi Tamás Németh Laura Simon-Szabó Krisztina Kerekes Csaba Szántai-Kis Diána Brauswetter Márton Kokas Gábor Borbély Anna Erdei Attila Mócsai György Kéri Tibor Vántus

Emerging evidence suggests that the vascular endothelial growth factor receptor 2 (VEGFR2) and protein kinase D1 (PKD1) signaling axis plays a critical role in normal and pathological angiogenesis and inflammation related processes. Despite all efforts, the currently available therapeutic interventions are limited. Prior studies have also proved that a multiple target inhibitor can be more effi...

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