نتایج جستجو برای: pompe

تعداد نتایج: 1229  

Journal: :Current gene therapy 2009
Dwight D Koeberl Priya S Kishnani

Significant advances in therapy for lysosomal storage disorders have occurred with an accelerating pace over the past decade. Although enzyme replacement therapy has improved the outcome of lysosomal storage disorders, antibody responses have occurred and sometimes prevented efficacy, especially in cross-reacting immune material negative patients with Pompe disease. Preclinical gene therapy exp...

2009
Michael Beck

Pompe disease is a lysosomal storage disorder characterized by muscle weakness and cardiomyopathy. It shows a broad variability regarding the clinical severity as well as the age of onset. In the past, two different recombinant enzyme preparations have been developed for the treatment of Pompe patients: alpha-glucosidase, produced in rabbit milk, and alpha-glucosidase, produced in Chinese hamst...

Pompe disease (PD), also known as “glycogen storage disease type II (OMIM # 232300)” is a rare autosomal recessive disorder characterized by progressive glycogen accumulation in cellular lysosomes. It ultimately leads to cellular damage. Infantile-onset Pompe disease (IOPD) is the most severe type of this disease and is characterized by severe hypertrophic cardiomyopathy and generalized hypoton...

Journal: :BMC Musculoskeletal Disorders 2013

Journal: :Human Molecular Genetics 2011

Journal: :Journal of visualized experiments : JoVE 2017
Jan Lukas Anne-Marie Knospe Susanne Seemann Valentina Citro Maria V Cubellis Arndt Rolfs

The use of personalized medicine to treat rare monogenic diseases like lysosomal storage disorders (LSDs) is challenged by complex clinical trial designs, high costs, and low patient numbers. Hundreds of mutant alleles are implicated in most of the LSDs. The diseases are typically classified into 2 to 3 different clinical types according to severity. Moreover, molecular characterization of the ...

Journal: :Journal of neuromuscular diseases 2015
Simone Sampaolo Olimpia Farina Federica Cipullo Filomena Napolitano Teresa Esposito Luca Lombardi Giuseppe Di Iorio

members of the third generation of a late-onset Pompe disease family which counts 36 individuals. Clinical, laboratory, and GAA enzymatic and genetic studies disclosed widespread myalgias and low back pain as well as mild weakness of the pelvic girdle muscles in 5 individuals (3 females, 2 males; aged 24–30 years), 3 of whom had a slight increase in CPK. Symptom onset was during the second deca...

Journal: :Molecular genetics and metabolism 2010
Karen M Ashe Kristin M Taylor Qiuming Chu Elizabeth Meyers Allen Ellis Varvara Jingozyan Katherine Klinger Patrick F Finn Christopher G F Cooper Wei-Lien Chuang John Marshall John M McPherson Robert J Mattaliano Seng H Cheng Ronald K Scheule Rodney J Moreland

Pompe disease, also known as glycogen storage disease (GSD) type II, is caused by deficiency of lysosomal acid alpha-glucosidase (GAA). The resulting glycogen accumulation causes a spectrum of disease severity ranging from a rapidly progressive course that is typically fatal by 1-2years of age to a more slowly progressive course that causes significant morbidity and early mortality in children ...

2009
Aneal Khan Barbara Ramage Ion Robu Laura Benard

Side-alternating vibration training (SAVT) was used for 15 weeks in a patient with Late-onset Pompe disease who had never used enzyme replacement or chaperone therapy. Prior to the use of SAVT, the patient had experienced declining muscle performance and her 6-minute walk distance decreased from 210 to 155 metres in 6 months. After SAVT, her 6-minute walk distance increased 70% from 166 to 282 ...

Journal: :Journal of neuromuscular diseases 2015
Edward Silk Richard K Jones Christian Hendriksz Reena Sharma Ana Jovanovic Gisela Wilcox Richard J Baker

This study is a non-interventional correlation study. Two groups (n = 40), consisting of 20 LOPD patients and 20 healthy age-matched (HAM), individuals will have their gait patterns assessed by instrumented gait analysis (Qualisys OQUS). The aim of the study is to identify the differences in gait between LOPD patients and HAM individuals. The LOPD group will be recruited from the Mark Holland m...

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