نتایج جستجو برای: smarca4 gene

تعداد نتایج: 1141500  

Journal: :Blood 2014
Jenny Zhang Dereje Jima Andrea B Moffitt Qingquan Liu Magdalena Czader Eric D Hsi Yuri Fedoriw Cherie H Dunphy Kristy L Richards Javed I Gill Zhen Sun Cassandra Love Paula Scotland Eric Lock Shawn Levy David S Hsu David Dunson Sandeep S Dave

In this study, we define the genetic landscape of mantle cell lymphoma (MCL) through exome sequencing of 56 cases of MCL. We identified recurrent mutations in ATM, CCND1, MLL2, and TP53. We further identified a number of novel genes recurrently mutated in patients with MCL including RB1, WHSC1, POT1, and SMARCA4. We noted that MCLs have a distinct mutational profile compared with lymphomas from...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2015
Melanie Bischof Matthias Weider Melanie Küspert Klaus-Armin Nave Michael Wegner

Myelinating Schwann cells in the vertebrate peripheral nervous system rely on Brg1 (Smarca4) for terminal differentiation. Brg1 serves as central ATP-hydrolyzing subunit of the chromatin remodelling BAF complexes and is recruited during myelination as part of these complexes by the transcription factor Sox10 in Schwann cells. Here, we analyzed the role of Brg1 during development of myelinating ...

Journal: :Nature communications 2014
Robert Huether Li Dong Xiang Chen Gang Wu Matthew Parker Lei Wei Jing Ma Michael N Edmonson Erin K Hedlund Michael C Rusch Sheila A Shurtleff Heather L Mulder Kristy Boggs Bhavin Vadordaria Jinjun Cheng Donald Yergeau Guangchun Song Jared Becksfort Gordon Lemmon Catherine Weber Zhongling Cai Jinjun Dang Michael Walsh Amanda L Gedman Zachary Faber John Easton Tanja Gruber Richard W Kriwacki Janet F Partridge Li Ding Richard K Wilson Elaine R Mardis Charles G Mullighan Richard J Gilbertson Suzanne J Baker Gerard Zambetti David W Ellison Jinghui Zhang James R Downing

Studies of paediatric cancers have shown a high frequency of mutation across epigenetic regulators. Here we sequence 633 genes, encoding the majority of known epigenetic regulatory proteins, in over 1,000 paediatric tumours to define the landscape of somatic mutations in epigenetic regulators in paediatric cancer. Our results demonstrate a marked variation in the frequency of gene mutations acr...

2014
Shahram Torkamandi Meysam Moghbeli Moein Farshchian Abolfazl Rad Mohammad Reza Abbaszadegan

OBJECTIVES Epigenetic regulation of gene expression can be carried out through chromatin remodeling enzymes such as SWI/SNF. Brg1 also known as SMARCA4 is a catalytic subunit of SWI/SNF, which is necessary for MMPs expression. Matrix metalloproteinases (MMPs) are known as important player enzymes during tumor progression and metastasis. Aberrant epigenetic modification of chromatin should be pr...

2014
Yisong Wang Anish Thomas Christopher Lau Arun Rajan Yuelin Zhu J. Keith Killian Iacopo Petrini Trung Pham Betsy Morrow Xiaogang Zhong Paul S. Meltzer Giuseppe Giaccone

Genetic alterations and etiology of thymic epithelial tumors (TETs) are largely unknown, hampering the development of effective targeted therapies for patients with TETs. Here TETs of advanced-stage patients enrolled in a clinical trial of molecularly-guided targeted therapies were employed for targeted sequencing of 197 cancer-associated genes. Comparative sequence analysis of 78 TET/blood pai...

2016
Anliang Dong Yan Lu Bingjian Lu

Ovarian carcinoma is the most lethal gynecological malignancy worldwide. Recent advance in genomic/epigenomic researches will impact on our prevention, detection and intervention on ovarian carcinoma. Detection of germline mutations in BRCA1/BRCA2, mismatch repair genes, and other genes in the homologous recombination/DNA repair pathway propelled the genetic surveillance of most hereditary ovar...

2014
Siddharth Manvati Kailash Chandra Mangalhara P. Kalaiarasan Niloo Srivastava Bhupender Kumar R. N. K. Bamezai

Moderately increased DNA damage due to the exogenous miR-101 (4 fold) over-expression in MCF7 cells was substantiated by an increase in the number of γ-H2AX foci, correlating with a simple-to-do Halo-assay. miR-101 induced mild/moderate DNA damage favoured senescence rather than apoptosis. An experimental support emanated from the induced mild/moderate DNA damage with 1 µM/5 µM etoposide in MCF...

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