نتایج جستجو برای: cerebral edema

تعداد نتایج: 221342  

Journal: :Pediatric Critical Care Medicine 2017

Journal: :Journal of Neuroanaesthesiology and Critical Care 2020

Journal: :Neurologia medico-chirurgica 1968

Journal: :Journal of The Korean Medical Association 2023

Background: Mannitol and hypertonic saline are the most frequently used hyperosmolar agents to treat cerebral edema resulting from acute brain injury. However, there several issues with using therapies. Here, we focus on potential adverse effects of therapies practical tips overcome these in neurointensive care unit.Current Concepts: Among used, mannitol may decrease intravascular volume pose a...

Journal: :Hypertension 1986
P W de Leeuw F L Waltman W H Birkenhäger

A 40-year-old man was admitted to the hospital with pulmonary edema without signs of left ventricular failure. Noncardiogenic pulmonary edema was diagnosed, and a subsequent workup identified a pheochromocytoma as the cause of this condition. The clinical picture could be mimicked by infusion of exogenous norepinephrine. It is concluded that surges of catecholamines from a pheochromocytoma may ...

Journal: :Journal of neurocritical care (Print) 2022

Background: Cerebral hyperperfusion syndrome (CHS) is a failure of autoregulation after revascularization procedure. It has rarely been reported in patients with no pre-existing cerebral hypoperfusion. Case Report: We present rare case patient who underwent stent graft implantation to treat postirradiated carotid blowout syndrome. The developed hypertension, focal neurological deficit, and seiz...

Journal: :مجله علوم اعصاب شفای خاتم 0
fatemeh maghool neuroscience research center, school of medicine, kerman university of medical science, kerman, iran. mohammad khaksari physiology research center, school of medicine, kerman university of medical science, kerman, iran. gholamreza asadi karam physiology research center, school of medicine, kerman university of medical science, kerman, iran.

brain edema plays an important role in secondary tissue damage following traumatic brain injury (tbi) but the underlying mechanisms are not entirely elucidated. the g protein coupled receptor fm-4 and its ligands, neuromedin s (nms) and neuromedin u (nmu), are expressed in diverse brain areas, and have a variety of roles in nociception, inflammation, and stress but their probable changes after ...

Journal: :Stroke 1979
N M Branston D T Hope L Symon

brain ischemia: protection from irreversible damage with a rapid acting barbiturate (Methohexital). Stroke 3: 726-732, 1972 11. Clasen R, Pandolfi S, Casey D: Furosemide and pentobarbital in cryogenic cerebral injury and edema. Neurology (Minneap) 24: 642-648, 1974 12. Smith A, Marque J: Anesthetics and cerebral edema. Anesthesiology 45: 64-72, 1976 13. Anderson D, Crawford R: Corticosteroids i...

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