نتایج جستجو برای: dasatinib

تعداد نتایج: 1954  

Journal: :Haematologica 2015
Nicolas Boissel Aline Renneville Thibaut Leguay Pascale Cornillet Lefebvre Christian Recher Thibaud Lecerf Eric Delabesse Céline Berthon Odile Blanchet Thomas Prebet Cécile Pautas Patrice Chevallier Stéphane Leprêtre Stéphane Girault Caroline Bonmati Romain Guièze Chantal Himberlin Edouard Randriamalala Claude Preudhomme Eric Jourdan Hervé Dombret Norbert Ifrah

Core-binding factor acute myeloid leukemia is a favorable acute myeloid leukemia subset cytogenetically defined by t(8;21) or inv(16)/t(16;16) rearrangements, disrupting RUNX1 (previously CBFA/AML1) or CBFB transcription factor functions. The receptor tyrosine kinase KIT is expressed in the vast majority of these acute myeloid leukemias and frequent activating KIT gene mutations have been assoc...

2015
E.V. Morozova Y.Y. Vlasova M.V. Pryanishnikova K.V. Lepik B.V. Afanasyev

The introduction of tyrosine kinase inhibitors (TKIs) in the treatment of chronic myeloid leukemia (CML) has significantly increased survival rate and quality of life for patients with CML. Despite the high efficacy of imatinib, not all patients benefit from this treatment. Resistance to imatinib can develop from a number of mechanisms. One of the main reasons for treatment failure is a mutatio...

Journal: :Blood 2008
Kimmo Porkka Perttu Koskenvesa Tuija Lundán Johanna Rimpiläinen Satu Mustjoki Richard Smykla Robert Wild Roger Luo Montserrat Arnan Benoit Brethon Lydia Eccersley Henrik Hjorth-Hansen Martin Höglund Hana Klamova Håvar Knutsen Suhag Parikh Emmanuel Raffoux Franz Gruber Finella Brito-Babapulle Hervé Dombret Rafael F Duarte Erkki Elonen Ron Paquette C Michel Zwaan Francis Y F Lee

Although imatinib, a BCR-ABL tyrosine kinase inhibitor, is used to treat acute Philadelphia chromosome-positive (Ph(+)) leukemia, it does not prevent central nervous system (CNS) relapses resulting from poor drug penetration through the blood-brain barrier. Imatinib and dasa-tinib (a dual-specific SRC/BCR-ABL kinase inhibitor) were compared in a preclinical mouse model of intracranial Ph(+) leu...

Journal: :American journal of respiratory and critical care medicine 2007
Anne Bergeron Delphine Réa Vincent Levy Clément Picard Véronique Meignin Jérome Tamburini Heriberto Bruzzoni-Giovanelli Fabien Calvo Abdellatif Tazi Philippe Rousselot

Tyrosine kinase inhibitors have revolutionized the treatment of chronic myeloid leukemia and are increasingly used for other indications. Fluid retention, however, including pleural effusions, are a significant side effect of imatinib, the first-line treatment for chronic myeloid leukemia. We investigated pleural and pulmonary complications in patients treated with dasatinib, a novel multitarge...

2011
Roxana E. Iacob Jianming Zhang Nathanael S. Gray John R. Engen

Abl kinase inhibitors targeting the ATP binding pocket are currently employed as potent anti-leukemogenic agents but drug resistance has become a significant clinical limitation. Recently, a compound that binds to the myristate pocket of Abl (GNF-5) was shown to act cooperatively with nilotinib, an ATP-competitive inhibitor to target the recalcitrant "T315I" gatekeeper mutant of Bcr-Abl. To unc...

2015
Bin Chen Xin Xu Jie Luo Heyong Wang Songwen Zhou Shi-Yong Sun

Src and the mammalian target of rapamycin (mTOR) signaling are commonly activated in non-small cell lung cancer (NSCLC) and hence potential targets for chemotherapy. Although the combined use of Src inhibitor Dasatinib with other chemotherapeutic agents has shown superior efficacy for cancer treatment, the mechanisms that lead to enhanced sensitivity of Dasatinib are not completely understood. ...

Journal: :Blood 2007
Tri K Nguyen Mohamed Rahmani Hisashi Harada Paul Dent Steven Grant

Interactions between MEK1/2 inhibitors and the dual Abl/Src kinase inhibitor dasatinib (BMS-354825) were examined in chronic myeloid leukemia (CML) cell lines and primary specimens. Cotreatment of K562 or LAMA cells with subtoxic or marginally toxic concentrations of PD184352 (or U0126) and dasatinib synergistically potentiated mitochondrial damage, caspase activation, and apoptosis. Similar in...

Journal: :Blood 2006
Mhairi Copland Ashley Hamilton Lucy J Elrick Janet W Baird Elaine K Allan Niove Jordanides Martin Barow Joanne C Mountford Tessa L Holyoake

Dasatinib (BMS-354825), a novel dual SRC/BCR-ABL kinase inhibitor, exhibits greater potency than imatinib mesylate (IM) and inhibits the majority of kinase mutations in IM-resistant chronic myeloid leukemia (CML). We have previously demonstrated that IM reversibly blocks proliferation but does not induce apoptosis of primitive CML cells. Here, we have attempted to overcome this resistance with ...

2014
Adam Obr Pavla Röselová Dana Grebeňová Kateřina Kuželová

Attachment of stem leukemic cells to the bone marrow extracellular matrix increases their resistance to chemotherapy and contributes to the disease persistence. In chronic myelogenous leukemia (CML), the activity of the fusion BCR-ABL kinase affects adhesion signaling. Using real-time monitoring of microimpedance, we studied in detail the kinetics of interaction of human CML cells (JURL-MK1, MO...

Journal: :The Journal of pharmacology and experimental therapeutics 2009
Ying Chen Sagar Agarwal Naveed M Shaik Cliff Chen Zheng Yang William F Elmquist

The novel tyrosine kinase inhibitor dasatinib (Sprycel; BMS-354825) is approved for use in imatinib (Gleevec; STI 571)-resistant or -intolerant chronic myelogenous leukemia and may be useful for other tumors in the central nervous system (CNS). The objective of this study was to investigate the role of P-glycoprotein (P-gp) and breast cancer resistance protein (BCRP) in modulating the CNS penet...

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