نتایج جستجو برای: delayed expression

تعداد نتایج: 964022  

Journal: :Journal of animal science 2014
J M Thomas S L Lock S E Poock M R Ellersieck M F Smith D J Patterson

This experiment was designed to test the hypothesis that delayed insemination of nonestrous cows would increase pregnancy rates when using sex-sorted semen in conjunction with fixed-time artificial insemination (FTAI). Estrus was synchronized for 656 suckled beef cows with the 7-d CO-Synch + controlled internal drug release (CIDR) protocol (100 μg GnRH + CIDR [1.38 g progesterone] on d 0, 25 mg...

Journal: :Plant physiology 2006
Ruslana Radchuk Volodymyr Radchuk Winfriede Weschke Ljudmilla Borisjuk Hans Weber

The classic role of SUCROSE NONFERMENTING-1 (Snf1)-like kinases in eukaryotes is to adapt metabolism to environmental conditions such as nutrition, energy, and stress. During pea (Pisum sativum) seed maturation, developmental programs of growing embryos are adjusted to changing physiological and metabolic conditions. To understand regulation of the switch from cell proliferation to differentiat...

2013
Nouceyba Abdelkrim Adel Tellili Mohamed Naceur Abdelkrim

The additive fault tolerant control (FTC) for delayed system is studied in this work. To design the additive control, two steps are necessary; the first one is the estimation of the sensor fault amplitude using a Luenberger observer with delay, and the second one consists to generate the additive fault tolerant control law and to add it to the nominal control of delayed system. The additive con...

2014
Hyo Jeong Kim Young-Chul Choi Hyung Jun Park Young-Mock Lee Heung Dong Kim Joon Soo Lee Hoon-Chul Kang

Congenital muscular dystrophy type 1A (MDC1A) is an autosomal recessive disorder characterized by hypotonia, elevated serum creatine kinase level, delayed motor milestones, white matter changes observed by brain magnetic resonance imaging, and normal intelligence. A mutation in the laminin α2 (LAMA2) gene, located at 6q22-23, is a genetic cause of MDC1A. Patients have merosin (laminin α2)-defic...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 1999
M W Halterman C C Miller H J Federoff

Hypoxia-induced delayed neuronal death is known to require de novo gene expression; however, the molecular mediators that are involved remain undefined. The transcription factor hypoxia-inducible factor-1alpha (HIF-1alpha), in addition to promoting the expression of adaptive genes under conditions of hypoxia, has been implicated as being a necessary component in p53-mediated cell death in tumor...

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