نتایج جستجو برای: infantile pompe disease

تعداد نتایج: 1498901  

Journal: :Lijecnicki vjesnik 2015
Ivan Pećin Diana Muačević-Katanec Iveta Šimić Ksenija Fumić Kristina Potočki Nediljko Šućur Željko Reiner

These guidelines provide a short summary of recommendations on Pompe disease, how to diagnose this disease, management of adult patients with this disease, follow-up of the patients and recommendations on therapy and genetic testing. Early diagnosis and management of patients with Pompe disease requires a multidisciplinary approach of several different experts. These guidelines were produced by...

Journal: :Medical archives 2013
Myftar Barbullushi Alma Idrizi Eriola Bolleku Anila Laku Arben Pilaca

Pompe disease is an acid maltase deficiency being part of glycogen storage diseases that affects all age groups. In both childhood and adult forms, the classic clinical picture is that of a progressive myopathy. Respiratory muscle involvement is common, may occur early in the course of the disease, and is the most frequent cause of mortality from acid maltase deficiency. Its association with rh...

2013
C Pérez Fernández L Bosanska U Plöckinger A Pöllinger

Introduction Glycogen storage disease type II (Pompe disease) is a rare, progressive muscle disorder with a wide range of phenotypic presentations. It is caused by an inherited deficiency of acid a-glucosidase (GAA), which leads to lysosomal glycogen accumulation in various tissues, most notably cardiac, skeletal, and smooth muscle. The gradual pathologic storage of GAA in muscle cells causes i...

Journal: :Human molecular genetics 2011
Hsiang-Po Huang Pin-Hsun Chen Wuh-Liang Hwu Ching-Yu Chuang Yin-Hsiu Chien Lee Stone Chung-Liang Chien Li-Tzu Li Shu-Chuan Chiang Hsin-Fu Chen Hong-Nerng Ho Chung-Hsuan Chen Hung-Chih Kuo

Pompe disease is caused by autosomal recessive mutations in the acid alpha-glucosidase (GAA) gene, which encodes GAA. Although enzyme replacement therapy has recently improved patient survival greatly, the results in skeletal muscles and for advanced disease are still not satisfactory. Here, we report the derivation of Pompe disease-induced pluripotent stem cells (PomD-iPSCs) from two patients ...

Journal: :Orphan Drugs: Research and Reviews 2016

Journal: :Human molecular genetics 2008
Nina Raben Victoria Hill Lauren Shea Shoichi Takikita Rebecca Baum Noboru Mizushima Evelyn Ralston Paul Plotz

The role of autophagy, a catabolic lysosome-dependent pathway, has recently been recognized in a variety of disorders, including Pompe disease, the genetic deficiency of the glycogen-degrading lysosomal enzyme acid-alpha glucosidase. Accumulation of lysosomal glycogen, presumably transported from the cytoplasm by the autophagic pathway, occurs in multiple tissues, but pathology is most severe i...

2013
Deniz Güngör Michelle Kruijshaar Iris Plug Ralph D’Agostino Marloes Hagemans Arnold Reuser Ans van der Ploeg

Background Since 2006, enzyme replacement therapy (ERT) has been available as a treatment for patients with Pompe disease. ERT has shown efficacy concerning muscle strength and pulmonary function in adult patients. However, no data on the effect of ERT on the survival of adult patients are currently available. Our objective was to assess the effect of ERT on survival in adult patients with Pomp...

Journal: :Neuromuscular Disorders 2011
Linda E.M. van den Berg Juna M. de Vries Robert M. Verdijk Ans T. van der Ploeg Arnold J.J. Reuser Pieter A. van Doorn

We present a case of adult Pompe disease (acid maltase deficiency) with an uncommon clinical presentation characterized by severe fatigue and myalgia prior to the onset of limb girdle weakness. Remarkably, the muscle biopsy demonstrated selective involvement of type 1 muscle fibers. The cause and clinical effects of fiber type specific involvement are currently unknown, but the phenomenon might...

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