نتایج جستجو برای: mouse tnf

تعداد نتایج: 329385  

Journal: :Carcinogenesis 2013
Martin Chopra Simone S Riedel Marlene Biehl Stefanie Krieger Viktoria von Krosigk Carina A Bäuerlein Christian Brede Ana-Laura Jordan Garrote Sabrina Kraus Viktoria Schäfer Miriam Ritz Katharina Mattenheimer Axelle Degla Anja Mottok Hermann Einsele Harald Wajant Andreas Beilhack

The cytokine tumor necrosis factor (TNF) has pleiotropic functions both in normal physiology and disease. TNF signals by the virtue of two cell surface receptors, TNF receptor 1 (TNFR1) and TNF receptor 2 (TNFR2). Exogenous TNF promotes experimental metastasis in some models, yet the underlying mechanisms are poorly understood. To study the contribution of host TNFR1 and TNFR2 on tumor cell pro...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2006
Robert E Iosif Christine T Ekdahl Henrik Ahlenius Cornelis J H Pronk Sara Bonde Zaal Kokaia Sten-Eirik W Jacobsen Olle Lindvall

Tumor necrosis factor-alpha (TNF-alpha) is a proinflammatory cytokine, acting through the TNF-R1 and TNF-R2 receptors. The two receptors have been proposed to mediate distinct TNF-alpha effects in the CNS, TNF-R1 contributing to neuronal damage and TNF-R2 being neuroprotective. Whether TNF-alpha and its receptors play any role for neurogenesis in the adult brain is unclear. Here we used mouse m...

2013
Mayur Sarangdhar Akash Kushwaha Jeanine Dahlquist Anil Jegga Bruce Aronow

Anti-TNF drugs are frequently associated with serious Adverse Events (AEs), which necessitates an improved understanding of individual factors that determine efficacy and safety of anti-TNF agents. We mined the US FDA's Adverse Event Reporting System (AERS) for anti-TNF-associated AEs to identify and stratify patient subgroups and drug combinations that exhibit specifically correlated complicat...

Journal: :The Journal of Experimental Medicine 1994
W J Hrushesky T Langevin Y J Kim P A Wood

Recombinant human tumor necrosis factor-alpha (TNF-alpha) has demonstrable antitumor activity in transplantable murine tumor models and patients with cancer but is highly toxic to both animals and human beings. The narrow therapeutic index of TNF-alpha has limited its anticancer utility. Toxicity associated with many standard anticancer drugs is highly dependent upon the circadian timing of the...

Journal: :EMBO reports 2003
Anne Devin Yong Lin Zheng-gang Liu

The death-domain kinase RIP (receptor-interacting protein) is an important effector of tumour necrosis factor (TNF) signalling and is essential for TNF-induced nuclear factor-kappaB activation. However, the function of RIP in the TNF-induced activation of mitogen-activated protein kinases (MAPKs) has not been fully investigated. In this report, using Rip null (Rip(-/-)) mouse fibroblast cells, ...

Journal: :The Journal of biological chemistry 2002
Shuichiro Ito Kenji Wakabayashi Osamu Ubukata Shinko Hayashi Fumihiko Okada Tadashi Hata

Bone remodeling involves the resorption of bone by osteoclasts and the synthesis of bone matrix by osteoblasts. Receptor activator of NF-kappa B ligand (RANKL, also known as ODF and OPGL), a member of the tumor necrosis factor (TNF) family, triggers osteoclastogenesis by forming a complex with its receptor, RANK. We have determined the crystal structure of the extracellular domain of mouse RANK...

Journal: :Infection and immunity 2005
Theo Sterns Nils Pollak Bernd Echtenacher Daniela N Männel

Susceptibility to bacterial infections after a primary immune stimulation differs drastically depending on the presensitization of the innate immune system. To determine the conditions that either induce protection or enhanced susceptibility to infection with Salmonella enterica serovar Typhimurium, we pretreated mice either with tumor necrosis factor (TNF), whole killed bacteria, or sublethal ...

Journal: :Infection and immunity 1997
K Takashima K Tateda T Matsumoto Y Iizawa M Nakao K Yamaguchi

The production and role of tumor necrosis factor alpha (TNF-alpha) in pneumococcal pneumonia were investigated in a mouse pneumonia model. When approximately 10(6) CFU of Streptococcus pneumoniae TUM19 were used to inoculate CBA/J mice intranasally, TNF-alpha levels in the lungs and serum began to increase from 1 and 3 days after infection, respectively, concomitantly with the increase in bacte...

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