نتایج جستجو برای: muscular function

تعداد نتایج: 1248677  

Journal: :مجله علوم اعصاب شفای خاتم 0
omid badri department of physiology, school of medicine, tabriz university of medical sceinces, tabriz, iran. parviz shahabi neuroscience research center, tabriz university of medical sciences, tabriz, iran. mehdi farhoudi neuroscience research center, tabriz university of medical sciences, tabriz, iran. mohsen sharif-zak department of biochemistry, school of medicine, tabriz university of medical sceinces, tabriz, iran. armin azadi department of management of informatics. school of management, tabriz university of medical sciences, tabriz, iran.

repairing the damaged nerves caused by physical injury (trauma), is one of the major challenges facing modern medical sciences. evening primrose oil (epo) extraction of oenothera biennis seed that enriches with omega-6,γ-linoleic acid and linoleic acid that they have essential role in myelination and neural membrane structure. the aim of this study was to evaluate the functional recovery of sci...

Journal: :iranian journal of pathology 2010
elahe keyhani jalal gharesouran kimia kahrizi yousef shafeghati hossein najmabadi

background and objective: becker muscular dystrophy (bmd) is a subtype of dystrophinopathies and designated as “mild form of dystrophinopathy”. the frequency rate of the disease is 1:18000 to 1:30000 in different populations and the symptoms are presented at about 8-9 years of age. the diagnostic panel composed of serum ceratin kinase (sck) measurement, electromyography (emg), and as a major co...

Journal: :iranian journal of pharmaceutical research 0
a arzi m rezaei

l-tyrosine, b6 and folic acid are involved in biosynthesis of dopa and consequently dopamine. the aim of this study was to investigate the antiparkinsonian effect of these agents in perphenazine-induced catatonia in rats. murprogo method or scored muscular rigidity, which is induced by a phenothiazine, was used to evaluate the antiparkinsonian effect of these agents. a significant decrease in m...

Journal: :BMC Medicine 2007
Richard T Hinkle Frank R Lefever Elizabeth T Dolan Deborah L Reichart Jefferey A Dietrich Kathryn E Gropp Robert I Thacker Jeffrey P Demuth Paula J Stevens Xiaoyan A Qu Alex R Varbanov Feng Wang Robert J Isfort

BACKGROUND Duchenne muscular dystrophy results from mutation of the dystrophin gene, causing skeletal and cardiac muscle loss of function. The mdx mouse model of Duchenne muscular dystrophy is widely utilized to evaluate the potential of therapeutic regimens to modulate the loss of skeletal muscle function associated with dystrophin mutation. Importantly, progressive loss of diaphragm function ...

2015
Karen Moraes Denise M. Paisani Nathália C. T. Pacheco Luciana D. Chiavegato

BACKGROUND A living donor transplant improves the survival and quality of life of a transplant patient. However, the impact of transplantation on postoperative lung function and respiratory muscular strength in kidney donors remains unknown. OBJECTIVE To evaluate pulmonary function, respiratory muscle strength, quality of life and the incidence of postoperative pulmonary complications (PPCs) ...

Journal: :Development 2013
Natalie Gibb Danielle L Lavery Stefan Hoppler

Wnt signalling is a key regulator of vertebrate heart development, yet it is unclear which specific Wnt signalling components are required to regulate which aspect of cardiogenesis. Previously, we identified Wnt6 as an endogenous Wnt ligand required for controlling heart muscle differentiation via canonical Wnt/β-catenin signalling. Here we show for the first time a requirement for an endogenou...

Journal: :Integrative cancer therapies 2005
Lisa K Sprod Scott N Drum Ann T Bentz Susan D Carter Carole M Schneider

Breast cancer treatment often results in impaired shoulder function, in particular, decrements in muscular endurance and range of motion, which may lead to decreased quality of life. The purpose of this investigation was to determine the effects of walking pole use on shoulder function in female breast cancer survivors. Participants had previously been treated with 1 or a combination of the fol...

Journal: :Arquivos De Neuro-psiquiatria 2023

Background: Spinal muscular atrophy (SMA) is a disorder caused by homozygous loss of function the SMN1 gene. This gene produces survival motor neuron (SMN) protein, which important in homeostasis. The SMN2 has homology with SMN1, but only expresses 10% functional full-length SMN protein. treatment available Brazilian public health system Nusinersen, an antisense oligonucleotide that increases p...

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