نتایج جستجو برای: scar formation

تعداد نتایج: 538754  

Journal: :Journal of cellular physiology 2005
Jessica Drapeau Viviane El-Helou Robert Clement Samar Bel-Hadj Hugues Gosselin Louis-Eric Trudeau Louis Villeneuve Angelino Calderone

Nerve fiber innervation of the scar following myocardial damage may have occurred either via the growth of pre-existing fibers and/or the mobilization of neural stem cells. The present study examined whether neural stem cells were recruited to the infarct region of the rat heart following coronary artery ligation. The neural stem cell marker nestin was detected in the infarct region of 1-week p...

Journal: :Spine 2006
Kei Miyamoto Koichi Masuda Nozomu Inoue Masahiko Okuma Carol Muehleman Howard S An

STUDY DESIGN An in vivo study on the effects of an agent for scar prevention in a dog laminectomy model. OBJECTIVE To examine the anti-adhesion properties of a thrombin-based hemostatic gelatin (FLOSEAL; Baxter International Inc., Deerfield, IL). SUMMARY OF BACKGROUND DATA Postlaminectomy dural adhesion sometimes causes symptomatic problems. Although a new type of hemostatic agent, a thromb...

2009
C Troidl H Möllmann H Nef F Masseli S Voss S Szardien M Willmer A Rolf J Rixe K Troidl S Kostin C Hamm A Elsässer

An important goal in cardiology is to minimize myocardial necrosis and to support a discrete but resilient scar formation after myocardial infarction (MI). Macrophages are a type of cells that influence cardiac remodelling during MI. Therefore, the goal of the present study was to investigate their transcriptional profile and to identify the type of activation during scar tissue formation. Liga...

Journal: :Journal of neurosurgery 2003
Staci Goussev Jung-Yu C Hsu Yong Lin Tjoson Tjoa Nino Maida Zena Werb Linda J Noble-Haeusslein

OBJECT Matrix metalloproteinases (MMPs), particularly MMP-9/gelatinase B, promote early inflammation and barrier disruption after spinal cord injury (SCI). Early blockade of MMPs after injury provides neuroprotection and improves motor outcome. There is recent evidence, however, that MMP-9 and MMP-2/gelatinase A participate in later wound healing in the injured cord. The authors therefore exami...

Journal: :Актуальні питання педіатрії, акушерства та гінекології 2020

2013
Ben Short

M igrating cells move forward by harnessing the force of actin polymerization to form a protru-sive lamellipodium at the leading edge. A protein called Lamellipodin (Lpd) promotes lamellipodium formation, but how it does so is unclear. Law et al. reveal that Lpd generates membrane protrusions in association with a key regulator of actin branching called the Scar/WAVE complex and that this inter...

2016
Hannes Petersen Fatemeh Tavakoli Sebastian Kruber Adrian Münscher Alexandra Gliese Nils‐Owe Hansen Stephanie Uschold Dennis Eggert Wesley D. Robertson Tobias Gosau Susanne Sehner Marcel Kwiatkowski Hartmut Schlüter Udo Schumacher Rainald Knecht R.J. Dwayne Miller

BACKGROUND AND OBJECTIVE As a result of wound healing the original tissue is replaced by dysfunctional scar tissue. Reduced tissue damage during surgical procedures beneficially affects the size of the resulting scar and overall healing time. Thus the choice of a particular surgical instrument can have a significant influence on the postoperative wound healing. To overcome these problems of wou...

2013
Ah-Lai Law Anne Vehlow Maria Kotini Lauren Dodgson Daniel Soong Eric Theveneau Cristian Bodo Eleanor Taylor Christel Navarro Upamali Perera Magdalene Michael Graham A. Dunn Daimark Bennett Roberto Mayor Matthias Krause

Cell migration is essential for development, but its deregulation causes metastasis. The Scar/WAVE complex is absolutely required for lamellipodia and is a key effector in cell migration, but its regulation in vivo is enigmatic. Lamellipodin (Lpd) controls lamellipodium formation through an unknown mechanism. Here, we report that Lpd directly binds active Rac, which regulates a direct interacti...

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