نتایج جستجو برای: slc30a8

تعداد نتایج: 228  

2012
Katarzyna Linder Robert Wagner Erifili Hatziagelaki Caroline Ketterer Martin Heni Fausto Machicao Norbert Stefan Harald Staiger Hans-Ulrich Häring Andreas Fritsche

INTRODUCTION Single nucleotide polymorphisms (SNPs) in approximately 40 genes have been associated with an increased risk for type 2 diabetes (T2D) in genome-wide association studies. It is not known whether a similar genetic impact on the risk of prediabetes (impaired glucose tolerance [IGT] or impaired fasting glycemia [IFG]) exists. METHODS In our cohort of 1442 non-diabetic subjects of Eu...

Journal: :Diabetes 2008
Allan F. Moore Kathleen A. Jablonski Jarred B. McAteer Richa Saxena Toni I. Pollin Paul W. Franks Robert L. Hanson Alan R. Shuldiner William C. Knowler David Altshuler Jose C. Florez

OBJECTIVE Genome-wide association scans (GWASs) have identified novel diabetes-associated genes. We evaluated how these variants impact diabetes incidence, quantitative glycemic traits, and response to preventive interventions in 3,548 subjects at high risk of type 2 diabetes enrolled in the Diabetes Prevention Program (DPP), which examined the effects of lifestyle intervention, metformin, and ...

2009
Jianhua Zhao Mingyao Li Jonathan P. Bradfield Kai Wang Haitao Zhang Patrick Sleiman Cecilia E. Kim Kiran Annaiah Wendy Glaberson Joseph T. Glessner F. George Otieno Kelly A. Thomas Maria Garris Cuiping Hou Edward C. Frackelton Rosetta M. Chiavacci Robert I. Berkowitz Hakon Hakonarson Struan F.A. Grant

OBJECTIVE A number of studies have found that reduced birth weight is associated with type 2 diabetes later in life; however, the underlying mechanism for this correlation remains unresolved. Recently, association has been demonstrated between low birth weight and single nucleotide polymorphisms (SNPs) at the CDKAL1 and HHEX-IDE loci, regions that were previously implicated in the pathogenesis ...

Journal: :Diabetes 2008
Joshua P. Lewis Nicholette D. Palmer Pamela J. Hicks Michele M. Sale Carl D. Langefeld Barry I. Freedman Jasmin Divers Donald W. Bowden

OBJECTIVE Several whole-genome association studies have reported identification of type 2 diabetes susceptibility genes in various European-derived study populations. Little investigation of these loci has been reported in other ethnic groups, specifically African Americans. Striking differences exist between these populations, suggesting they may not share identical genetic risk factors. Our o...

Journal: :Diabetes 2009
Nicholas J. Timpson Cecilia M. Lindgren Michael N. Weedon Joshua Randall Willem H. Ouwehand David P. Strachan N. William Rayner Mark Walker Graham A. Hitman Alex S.F. Doney Colin N.A. Palmer Andrew D. Morris Andrew T. Hattersley Eleftheria Zeggini Timothy M. Frayling Mark I. McCarthy

OBJECTIVE This study examined how differences in the BMI distribution of type 2 diabetic case subjects affected genome-wide patterns of type 2 diabetes association and considered the implications for the etiological heterogeneity of type 2 diabetes. RESEARCH DESIGN AND METHODS We reanalyzed data from the Wellcome Trust Case Control Consortium genome-wide association scan (1,924 case subjects,...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2017
Hao Wan Chengfeng Merriman Mark A Atkinson Clive H Wasserfall Kieran M Mcgrail Yongye Liang Dax Fu Hongjie Dai

Identified as a major biomarker for type 1 diabetes (T1D) diagnosis, zinc transporter 8 autoantibody (ZnT8A) has shown promise for staging disease risk and disease diagnosis. However, existing assays for ZnT8 autoantibody (ZnT8A) are limited to detection by soluble domains of ZnT8, owing to difficulties in maintaining proper folding of a full-length ZnT8 protein outside its native membrane envi...

2016
Cailin Yu Jeremy C. Burns William H. Robinson Paul J. Utz Peggy P. Ho Lawrence Steinman Alan B. Frey

Type 1 diabetes is an autoimmune disease in which insulin-producing pancreatic islet β cells are the target of self-reactive B and T cells. T cells reactive with epitopes derived from insulin and/or IGRP are critical for the initiation and maintenance of disease, but T cells reactive with other islet antigens likely have an essential role in disease progression. We sought to identify candidate ...

2012
Émmanuelle Énée Roland Kratzer Jean-Baptiste Arnoux Emilie Barilleau Yamina Hamel Christophe Marchi Jacques Beltrand Bénédicte Michaud Lucienne Chatenoud Jean-Jacques Robert Peter van Endert

Type 1 diabetes results from the destruction of β-cells by an autoimmune T-cell response assisted by antigen-presenting B cells producing autoantibodies. CD8(+) T-cell responses against islet cell antigens, thought to play a central role in diabetes pathogenesis, can be monitored using enzyme-linked immunosorbent spot (ELISpot) assays. However, such assays have been applied to monitoring of adu...

2016
Zhen Wang Ying Zheng Yiting Tu Zhijie Dai Jian Lin Zhiguang Zhou

Background. Fulminant type 1 diabetes (FT1D) is a novel subtype of type 1 diabetes characterized by extremely rapid onset and complete deficiency of insulin due to the destruction of pancreatic β cells. However, the precise mechanisms underlying the etiology of this disease remain unclear. Methods. A total of 22 patients with FT1D and 10 healthy subjects were recruited. Serum antibodies to GAD,...

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