نتایج جستجو برای: ژن calhm1

تعداد نتایج: 15840  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2012
Zhongming Ma Adam P Siebert King-Ho Cheung Robert J Lee Brian Johnson Akiva S Cohen Valérie Vingtdeux Philippe Marambaud J Kevin Foskett

Extracellular Ca(2+) (Ca(2+)(o)) plays important roles in physiology. Changes of Ca(2+)(o) concentration ([Ca(2+)](o)) have been observed to modulate neuronal excitability in various physiological and pathophysiological settings, but the mechanisms by which neurons detect [Ca(2+)](o) are not fully understood. Calcium homeostasis modulator 1 (CALHM1) expression was shown to induce cation current...

Journal: :The Journal of biological chemistry 2013
Adam P Siebert Zhongming Ma Jeremy D Grevet Angelo Demuro Ian Parker J Kevin Foskett

CALHM1 (calcium homeostasis modulator 1) forms a plasma membrane ion channel that mediates neuronal excitability in response to changes in extracellular Ca(2+) concentration. Six human CALHM homologs exist with no homology to other proteins, although CALHM1 is conserved across >20 species. Here we demonstrate that CALHM1 shares functional and quaternary and secondary structural similarities wit...

Journal: :Cell 2008
Ute Dreses-Werringloer Jean-Charles Lambert Valérie Vingtdeux Haitian Zhao Horia Vais Adam Siebert Ankit Jain Jeremy Koppel Anne Rovelet-Lecrux Didier Hannequin Florence Pasquier Daniela Galimberti Elio Scarpini David Mann Corinne Lendon Dominique Campion Philippe Amouyel Peter Davies J. Kevin Foskett Fabien Campagne Philippe Marambaud

Alzheimer's disease (AD) is a genetically heterogeneous disorder characterized by early hippocampal atrophy and cerebral amyloid-beta (Abeta) peptide deposition. Using TissueInfo to screen for genes preferentially expressed in the hippocampus and located in AD linkage regions, we identified a gene on 10q24.33 that we call CALHM1. We show that CALHM1 encodes a multipass transmembrane glycoprotei...

2013
Elizabeth M. Adler

This month's installment of Generally Physiological concerns identification of a channel crucial for the perception of sweet, bitter, and umami tastes; a role for caffeine in honeybee asso­ ciative olfactory memory; and meth­ ods for imaging calcium throughout entire astrocytes. A taste for CALHM1 Sweet, bitter, and umami (the savory taste elicited by glutamate) substan­ c es bind to G protein–...

Journal: :Seizure 2014
Xiang Li Yongcai Wang Jia Gu Qingming Meng Yong Gao Hongyu Zhao Zhongmin Yin

PURPOSE Mesial temporal lobe epilepsy (MTLE) is one of the most common forms of epilepsies in adults. The calcium homeostasis modulator 1 gene (CALHM1) has been considered one of the candidate genes that play a role in epileptogenesis due to its function in calcium homeostasis and amyloid β (Aβ) regulation. Recently, the association of a single nucleotide polymorphism (rs11191692) of CALHM1 has...

Journal: :Chemical senses 2015
Göran Hellekant Jared Schmolling Philippe Marambaud Teresa A Rose-Hellekant

Stimulation of Type II taste receptor cells (TRCs) with T1R taste receptors causes sweet or umami taste, whereas T2Rs elicit bitter taste. Type II TRCs contain the calcium channel, calcium homeostasis modulator protein 1 (CALHM1), which releases adenosine triphosphate (ATP) transmitter to taste fibers. We have previously demonstrated with chorda tympani nerve recordings and two-bottle preferenc...

Journal: :Biophysical Journal 2011

Journal: :Journal of Alzheimer's disease : JAD 2010
John S K Kauwe Carlos Cruchaga Sarah Bertelsen Kevin Mayo Wayne Latu Petra Nowotny Anthony L Hinrichs Anne M Fagan David M Holtzman Alison M Goate

Recent large-scale genetic studies of late-onset Alzheimer's disease have identified risk variants in CALHM1, GAB2, and SORL1. The mechanisms by which these genes might modulate risk are not definitively known. CALHM1 and SORL1 may alter amyloid-β (Aβ) levels and GAB2 may influence phosphorylation of the tau protein. In this study we have analyzed disease associated genetic variants in each of ...

Journal: :Journal of Alzheimer's disease : JAD 2010
Jean-Charles Lambert Kristel Sleegers Antonio González-Pérez Martin Ingelsson Gary W Beecham Mikko Hiltunen Onofre Combarros Maria J Bullido Nathalie Brouwers Karolien Bettens Claudine Berr Florence Pasquier Florence Richard Steven T Dekosky Didier Hannequin Jonathan L Haines Gloria Tognoni Nathalie Fiévet Jean-François Dartigues Christophe Tzourio Sebastiaan Engelborghs Beatrice Arosio Elicer Coto Peter De Deyn Maria Del Zompo Ignacio Mateo Merce Boada Carmen Antunez Jesus Lopez-Arrieta Jacques Epelbaum Brit-Maren Michaud Schjeide Ana Frank-Garcia Vilmentas Giedraitis Seppo Helisalmi Elisa Porcellini Alberto Pilotto Paola Forti Raffaele Ferri Marc Delepine Diana Zelenika Mark Lathrop Elio Scarpini Gabriele Siciliano Vincenzo Solfrizzi Sandro Sorbi Gianfranco Spalletta Giovanni Ravaglia Fernando Valdivieso Saila Vepsäläinen Victoria Alvarez Paolo Bosco Michelangelo Mancuso Francesco Panza Benedetta Nacmias Paola Bossù Olivier Hanon Paola Piccardi Giorgio Annoni David Mann Philippe Marambaud Davide Seripa Daniela Galimberti Rudolph E Tanzi Lars Bertram Corinne Lendon Lars Lannfelt Federico Licastro Dominique Campion Margaret A Pericak-Vance Hilkka Soininen Christine Van Broeckhoven Annick Alpérovitch Agustin Ruiz M Ilyas Kamboh Philippe Amouyel

The only established genetic determinant of non-Mendelian forms of Alzheimer's disease (AD) is the ε4 allele of the apolipoprotein E gene (APOE). Recently, it has been reported that the P86L polymorphism of the calcium homeostasis modulator 1 gene (CALHM1) is associated with the risk of developing AD. In order to independently assess this association, we performed a meta-analysis of 7,873 AD ca...

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