نتایج جستجو برای: acid maltase deficiency

تعداد نتایج: 868406  

Journal: :Muscle & nerve. Supplement 1995
A J Reuser M A Kroos M M Hermans A G Bijvoet M P Verbeet O P Van Diggelen W J Kleijer A T Van der Ploeg

Glycogen storage disease type II (GSD II/glycogenosis type II/Pompe's disease/acid maltase deficiency) is caused by the deficiency of lysosomal alpha-glucosidase resulting in lysosomal accumulation of glycogen. The disease is inherited as an autosomal recessive trait and is clinically heterogeneous. Early and late onset phenotypes are distinguished. Insight in the molecular nature of the lysoso...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1986
H Isaacs N Savage M Badenhorst T Whistler

An adult patient with lysosomal acid alpha-glucosidase deficiency was fully investigated, and then placed on various forms of therapy with favourable response to a high protein, low carbohydrate diet. The rationale for the employment of this therapy, the problem of acid maltase deficiency and the relationship to weakness and glycogenosome formation with accumulation or otherwise of glycogen wit...

Journal: :Journal of Neurology, Neurosurgery & Psychiatry 1984

Journal: :AJNR. American journal of neuroradiology 1991
J Cinnamon A E Slonim K S Black M T Gorey D M Scuderi R A Hyman

CT studies of the lumbar spine were performed in 19 patients with glycogen storage disease. Nine of 10 patients with McArdle's disease and seven of nine patients with acid maltase deficiency demonstrated posterior paraspinal muscle atrophy out of proportion to their ages. In addition, the psoas muscles were spared in all 10 patients with McArdle's disease and were involved with atrophy in seven...

Journal: :Medical archives 2013
Myftar Barbullushi Alma Idrizi Eriola Bolleku Anila Laku Arben Pilaca

Pompe disease is an acid maltase deficiency being part of glycogen storage diseases that affects all age groups. In both childhood and adult forms, the classic clinical picture is that of a progressive myopathy. Respiratory muscle involvement is common, may occur early in the course of the disease, and is the most frequent cause of mortality from acid maltase deficiency. Its association with rh...

Journal: :Annals of neurology 1995
J H Wokke M G Ausems M J van den Boogaard E F Ippel O van Diggelene M A Kroos M Boer F G Jennekens A J Reuser H K Ploos van Amstel

We performed a clinical, biochemical, and genetic study in 16 patients from 11 families with adult-onset acid maltase deficiency. All patients were compound heterozygotes and carried the IVS1(-13T --> G) transversion on one allele; the second allele harbored either a deletion of a T at position 525 in exon 2 (7 probands, 64%) or a deletion of exon 18 (1 proband, 9%). Deterioration of handicap w...

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