نتایج جستجو برای: congenital myasthenic syndrome

تعداد نتایج: 719275  

2013
Keivan Basiri Katsiaryna Belaya Wei Wei Liu Susan Maxwell Maryam Sedghi David Beeson

Mutations in DPAGT1 are a newly recognised cause of congenital myasthenic syndrome. DPAGT1 encodes an early component of the N-linked glycosylation pathway. Initially mutations in DPAGT1 have been associated with the onset of the severe multisystem disorder - congenital disorder of glycosylation type 1J. However, recently it was established that certain mutations in this gene can cause symptoms...

Journal: :Brain : a journal of neurology 2014
Sophie Nicole Amina Chaouch Torberg Torbergsen Stéphanie Bauché Elodie de Bruyckere Marie-Joséphine Fontenille Morten A Horn Marijke van Ghelue Sissel Løseth Yasmin Issop Daniel Cox Juliane S Müller Teresinha Evangelista Erik Stålberg Christine Ioos Annie Barois Guy Brochier Damien Sternberg Emmanuel Fournier Daniel Hantaï Angela Abicht Marina Dusl Steven H Laval Helen Griffin Bruno Eymard Hanns Lochmüller

Congenital myasthenic syndromes are a clinically and genetically heterogeneous group of rare diseases resulting from impaired neuromuscular transmission. Their clinical hallmark is fatigable muscle weakness associated with a decremental muscle response to repetitive nerve stimulation and frequently related to postsynaptic defects. Distal myopathies form another clinically and genetically hetero...

Journal: :Annals of neurology 2002
Christopher M Gomez Ricardo A Maselli Bhupinder P S Vohra Manuel Navedo Joel R Stiles Pierre Charnet Kelly Schott Legier Rojas John Keesey Anthony Verity Robert W Wollmann Jose Lasalde-Dominicci

We investigated the basis for a novel form of the slow-channel congenital myasthenic syndrome presenting in infancy in a single individual as progressive weakness and impaired neuromuscular transmission without overt degeneration of the motor endplate. Prolonged low-amplitude synaptic currents in biopsied anconeus muscle at 9 years of age suggested a kinetic disorder of the muscle acetylcholine...

2013
Judith Cossins Katsiaryna Belaya Debbie Hicks Mustafa A. Salih Sarah Finlayson Nicola Carboni Wei Wei Liu Susan Maxwell Katarzyna Zoltowska Golara Torabi Farsani Steven Laval Mohammed Zain Seidhamed Peter Donnelly David Bentley Simon J. McGowan Juliane Müller Jacqueline Palace Hanns Lochmüller David Beeson

Congenital myasthenic syndromes are a heterogeneous group of inherited disorders that arise from impaired signal transmission at the neuromuscular synapse. They are characterized by fatigable muscle weakness. We performed linkage analysis, whole-exome and whole-genome sequencing to determine the underlying defect in patients with an inherited limb-girdle pattern of myasthenic weakness. We ident...

Journal: :Arquivos de neuro-psiquiatria 2016
Paulo Victor Sgobbi de Souza Gabriel Novaes de Rezende Batistella Valéria Cavalcante Lino Wladimir Bocca Vieira de Rezende Pinto Marcelo Annes Acary Souza Bulle Oliveira

Neuromuscular junction disorders represent a wide group of neurological diseases characterized by weakness, fatigability and variable degrees of appendicular, ocular and bulbar musculature involvement. Its main group of disorders includes autoimmune conditions, such as autoimmune acquired myasthenia gravis and Lambert-Eaton syndrome. However, an important group of diseases include congenital my...

Journal: :Journal of neurology, neurosurgery, and psychiatry 2003
Marguerite Hill

Neuromuscular junction (NMJ) disorders result from destruction, malfunction or absence of one or more key proteins involved in neuromuscular transmission, illustrated diagrammatically in fig 1. The most common pathology is antibody mediated damage or down regulation of ion channels or receptors, resulting in myasthenia gravis (MG), Lambert-Eaton myasthenic syndrome (LEMS), and acquired neuromyo...

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