نتایج جستجو برای: consanguineous pakistani family

تعداد نتایج: 425994  

2009
Maleeha Azam Muhammad Imran Khan Andreas Gal Alamdar Hussain Syed Tahir Abbas Shah Muhammad Shakil Khan Ahmed Sadeque Habib Bokhari Rob W.J. Collin Ulrike Orth Maria M. van Genderen A.I. den Hollander Frans P. M. Cremers Raheel Qamar

PURPOSE To identify the gene mutations responsible for autosomal recessive retinitis pigmentosa (arRP) in Pakistani families. METHODS A cohort of consanguineous families with typical RP phenotype in patients was screened by homozygosity mapping using microsatellite markers that mapped close to 21 known arRP genes and five arRP loci. Mutation analysis was performed by direct sequencing of the ...

Journal: :Journal of medical genetics 1998
N J Lench A F Markham R F Mueller D P Kelsell R J Smith P J Willems I Schatteman H Capon P J Van De Heyning G Van Camp

We report a mutation in the connexin 26 gene (Cx26) in a consanguineous Moroccan family linked to the DFNA3/DFNB1 locus on human chromosome 13q11-q12. Affected subjects display congenital, bilateral, sensorineural hearing loss. We have previously identified Cx26 mutations in consanguineous Pakistani families. This current finding indicates that Cx26 mutations are not restricted to ethnically an...

Journal: :Investigative ophthalmology & visual science 2002
Shagufta Khaliq Abdul Hameed Muhammad Ismail Khalid Anwar S Qasim Mehdi

PURPOSE To map the disease locus in a four-generation, consanguineous Pakistani family affected by autosomal dominant congenital nuclear cataract (adNCat). All affected individuals had early onset of bilateral nuclear cataract. METHODS Genomic DNA from family members was typed for alleles at more than 300 known polymorphic genetic markers by polymerase chain reaction. The lod scores were calc...

Journal: :Journal of the College of Physicians and Surgeons--Pakistan : JCPSP 2011
Nadia Aquil Irfan Ahmed Khan Bashir Soomro

Although generally a sporadic disease, a few cases of Guillain Barre syndrome clustered in families have been reported. We describe four siblings of a Pakistani family from a consanguineous marriage out of whom two developed definite GBS and two had probable GBS at different times. This suggests that there may be a genetic element in the pathogenesis of at least some forms of Guillain Barre syn...

Journal: :Archives of ophthalmology 2010
Haiba Kaul S Amer Riazuddin Zaheeruddin A Qazi Idrees A Nasir Ahmad U Zafar Shaheen N Khan Tayyab Husnain Javed Akram J Fielding Hejtmancik Sheikh Riazuddin

OBJECTIVE To investigate the genetic basis and molecular characteristics of the isolated form of ectopia lentis. METHODS We ascertained a consanguineous Pakistani family with multiple individuals with ectopia lentis. All affected as well as unaffected members with isolated ectopia lentis underwent detailed ophthalmologic and medical examination. Blood samples were collected and DNA was extrac...

2010
Afshan Yasmeen S. Amer Riazuddin Haiba Kaul Sadia Mohsin Mohsin Khan Zaheeruddin A. Qazi Idrees A. Nasir Ahmad U. Zafar Shaheen N. Khan Tayyab Husnain Javed Akram J. Fielding Hejtmancik Sheikh Riazuddin

PURPOSE To identify the pathogenic mutations responsible for autosomal recessive congenital cataracts in consanguineous Pakistani families. METHODS All affected individuals underwent detailed ophthalmologic and medical examination. Blood samples were collected and genomic DNA was extracted. A genome-wide scan was performed with polymorphic microsatellite markers on genomic DNA from affected a...

2011
Adeel Ahmad Shakeela Daud Naseebullah Kakar Gudrun Nürnberg Peter Nürnberg Masroor Ellahi Babar Michaela Thoenes Christian Kubisch Jamil Ahmad Hanno Jörn Bolz

PURPOSE To determine the cause of Leber congenital amaurosis (LCA) and developmental cataracts in a consanguineous Pakistani family. METHODS The diagnosis was established in all affected individuals of a Pakistani LCA family by medical history, funduscopy, and standard ERG. We performed genome-wide linkage analysis for mapping the disease locus in this family. RESULTS Congenitally severely ...

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