نتایج جستجو برای: dr1 transgenic mice

تعداد نتایج: 423333  

2017
Laurie Rangan Jeanne Galaine Romain Boidot Mohamad Hamieh Magalie Dosset Julie Francoual Laurent Beziaud Jean-René Pallandre Elodie Lauret Marie Joseph Afag Asgarova Christophe Borg Talal Al Saati Yann Godet Jean Baptiste Latouche Séverine Valmary-Degano Olivier Adotévi

HLA-A*0201/DRB1*0101 transgenic mice (A2/DR1 mice) have been developed to study the immunogenicity of tumor antigen-derived T cell epitopes. To extend the use and application of this mouse model in the field of antitumor immunotherapy, we described a tumor cell line generated from a naturally occurring tumor in A2/DR1 mouse named SARC-L1. Histological and genes signature analysis supported the ...

2014
Bo Tang Seunghyun Kim Sarah Hammond David L Cullins David D Brand Edward F Rosloniec John M Stuart Arnold E Postlethwaite Andrew H Kang Linda K Myers

INTRODUCTION T cells orchestrate joint inflammation in rheumatoid arthritis (RA), yet they are difficult to study due to the small numbers of antigen-specific cells. The goal of this study was to characterize a new humanized model of autoimmune arthritis and to describe the phenotypic and functional changes that occur in autoimmune T cells following the induction of pathological events. METHO...

2012
Masaru Kimata David L Cullins Monica L Brown David D Brand Edward F Rosloniec Linda K Myers John M Stuart Andrew H Kang

INTRODUCTION We used DR1 transgenic mice and covalently linked DR1 multimers to characterize analog-specific inhibitory T cells in collagen-induced arthritis (CIA). Because of the low numbers of antigen-specific T cells in wild-type mice, functional T-cell studies in autoimmune arthritis have been challenging. The use of T-cell receptor (TCR) transgenic mice has provided useful information, but...

Journal: :Journal of immunology 2002
Edward F Rosloniec Karen B Whittington Dennis M Zaller Andrew H Kang

Rheumatoid arthritis is an autoimmune disease in which susceptibility is strongly associated with the expression of specific HLA-DR haplotypes, including DR1 (DRB1*0101) and DR4 (DRB1*0401). As transgenes, both of these class II molecules mediate susceptibility to an autoimmune arthritis induced by immunization with human type II collagen (hCII). The dominant T cell response of both the DR1 and...

Journal: :JCI insight 2016
Cristina C Clement Halima Moncrieffe Aditi Lele Ginger Janow Aniuska Becerra Francesco Bauli Fawzy A Saad Giorgio Perino Cristina Montagna Neil Cobelli John Hardin Lawrence J Stern Norman Ilowite Steven A Porcelli Laura Santambrogio

Juvenile idiopathic arthritis (JIA) is the most common pediatric rheumatological condition. Although it has been proposed that JIA has an autoimmune component, the autoantigens are still unknown. Using biochemical and proteomic approaches, we identified the molecular chaperone transthyretin (TTR) as an antigenic target for B and T cell immune responses. TTR was eluted from IgG complexes and aff...

2017
Laurent Gilardin Sandrine Delignat Ivan Peyron Mathieu Ing Yu-Chun Lone Bagirath Gangadharan Baptiste Michard Yousra Kherabi Meenu Sharma Anastas Pashov Jean-Baptiste Latouche Mohamad Hamieh Olivier Toutirais Pascale Loiseau Lionel Galicier Agnès Veyradier Srini Kaveri Bernard Maillère Paul Coppo Sébastien Lacroix-Desmazes

Acquired thrombotic thrombocytopenic purpura is a rare and severe disease characterized by auto-antibodies directed against "A Disintegrin And Metalloproteinase with Thrombospondin type 1 repeats, 13th member" (ADAMTS13), a plasma protein involved in hemostasis. Involvement of CD4+ T cells in the pathogenesis of the disease is suggested by the IgG isotype of the antibodies. However, the nature ...

Journal: :Journal of immunology 2002
Meera Unnikrishnan Daniel M Altmann Thomas Proft Faisal Wahid Jonathan Cohen John D Fraser Shiranee Sriskandan

The gene encoding streptococcal mitogenic exotoxin Z (SMEZ) was disrupted in Streptococcus pyogenes. Despite the presence of other superantigen genes, mitogenic responses in human and murine HLA-DQ transgenic cells were abrogated when cells were stimulated with supernatant from the smez(-) mutant compared with the parent strain. Remarkably, disruption of smez led to a complete inability to elic...

Journal: :Blood 2006
John Stagg Sandra Pommey Nicoletta Eliopoulos Jacques Galipeau

Several studies have demonstrated that marrow stromal cells (MSCs) can suppress allogeneic T-cell responses. However, the effect of MSCs on syngeneic immune responses has been largely overlooked. We describe here that primary MSCs derived from C57BL/6 mice behave as conditional antigen-presenting cells (APCs) and can induce antigen-specific protective immunity. Interferon gamma (IFNgamma)-treat...

Journal: :Journal of immunology 2005
Lee Faulkner Anneli Cooper Cristina Fantino Daniel M Altmann Shiranee Sriskandan

The profound clinical consequences of Gram-positive toxic shock are hypothesized to stem from excessive Th1 responses to superantigens. We used a new superantigen-sensitive transgenic model to explore the role of TCRalphabeta T cells in responses to staphylococcal enterotoxin B (SEB) in vitro and in two different in vivo models. The proliferative and cytokine responses of HLA-DR1 spleen cells w...

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