نتایج جستجو برای: hereditary spastic paraplegia

تعداد نتایج: 94252  

Journal: :Archives of neurology 2004
Peter Hedera Gerald M Fenichel Marcia Blair Jonathan L Haines

BACKGROUND Mutations in a novel GTPase gene SPG3A cause an autosomal dominant hereditary spastic paraplegia linked to chromosome 14q (SPG3), which accounts for approximately 10% to 15% of all autosomal dominant hereditary spastic paraplegia cases. The mutational spectrum of the SPG3A gene and the phenotype/genotype correlations have not yet been established. OBJECTIVE To describe a kindred wi...

Journal: :Archives of Neurology 2004

2017
Ricardo H. Roda Alice B. Schindler Craig Blackstone

Alterations in proteins that regulate endoplasmic reticulum morphology are common causes of hereditary spastic paraplegia (SPG1-78, plus others). Mutations in the REEP1 gene that encodes an endoplasmic reticulum-shaping protein are well-known causes of SPG31, a common autosomal dominant spastic paraplegia. A closely-related gene, REEP2, is mutated in SPG72, with both autosomal and recessive inh...

2017
Rehana Basri Ichiro Yabe Hiroyuki Soma Asako Takei Hiroyuki Nishimura Yuka Machino Yasumasa Kokubo Masafumi Kosugi Ryuichirou Okada Motohiro Yukitake Hisao Tachibana Yasuo Kuroda Shigeki Kuzuhara Hidenao Sasaki

Hereditary spastic paraplegia (HSP) is a group of genetically heterogeneous neurodegenerative disorders characterized by slowly progressive spasticity and weakness of the lower limbs. HSP is caused by failure of development or selective degeneration of the corticospinal tracts, which contain the longest axons in humans. The most common form of HSP is caused by mutations of the spastin gene (SPA...

Journal: :Journal of Medical Genetics 1997

2014
Filipa Flor-de-Lima Mafalda Sampaio Nahid Nahavandi Susana Fernandes Miguel Leão

Mutations in the ALS2 gene cause three distinct disorders: infantile ascending hereditary spastic paraplegia, juvenile primary lateral sclerosis, and autosomal recessive juvenile amyotrophic lateral sclerosis. We present a review of the literature and the case of a 16-year-old boy who is, to the best of our knowledge, the first Portuguese case with infantile ascending hereditary spastic paraple...

Journal: :Archives of neurology 2006
Shirley Rainier Carron Sher Orit Reish Donald Thomas John K Fink

BACKGROUND Mutations in the SPG3A gene (atlastin protein) cause approximately 10% of autosomal-dominant hereditary spastic paraplegia. For many subjects with an SPG3A mutation, spastic gait begins in early childhood and does not significantly worsen even over many years. Such subjects resemble those with spastic diplegic cerebral palsy. To date, only 9 SPG3A mutations have been reported. OBJE...

Journal: :IP Indian Journal of Neurosciences 2023

The hereditary spastic paraplegias (HSP) are a large group of inherited neurologic disorders that share the primary symptom difficulty in walking due to weakness and spasticity lower limbs. Spastic paraplegia-48 (SPG48) is an autosomal recessive disorder characterized by limbs resulting gait difficulties. Biallelic mutations AP5Z1 known cause this complex form paraplegia referred as SPG48 (MIM#...

2017
Jennifer Gass Patrick R Blackburn Jessica Jackson Sarah Macklin Jay van Gerpen Paldeep S Atwal

Hereditary spastic paraplegia is a group of clinically and genetically heterogeneous neurodegenerative disorders, often characterized by weakness and spasticity in the lower limbs. In our study, we describe a spastic paraplegia type 7 patient with an expanded phenotype who was diagnosed after the discovery of pathogenic variants in SPG7.

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