نتایج جستجو برای: lysosomal myopathy

تعداد نتایج: 27249  

2013
Jonathan Zirin Joppe Nieuwenhuis Norbert Perrimon

Several myopathies are associated with defects in autophagic and lysosomal degradation of glycogen, but it remains unclear how glycogen is targeted to the lysosome and what significance this process has for muscle cells. We have established a Drosophila melanogaster model to study glycogen autophagy in skeletal muscles, using chloroquine (CQ) to simulate a vacuolar myopathy that is completely d...

2015
Mehmet Tecellioglu Ozden Kamisli

INTRODUCTION Pompe disorder is a rare glycogen storage disorder that is due to a deficiency of the lysosomal alpha glycosidase enzyme. The heart, skeletal muscle, liver and nervous system can be affected from the lysosomal glycogen accumulation. Symptoms such as muscle weakness, hypotony, myopathy and respiratory failure develop. The onset may be at the infantile, adolescent or adult period dep...

Journal: :Cell 2009
Nivetha Ramachandran Iulia Munteanu Peixiang Wang Pauline Aubourg Jennifer J. Rilstone Nyrie Israelian Taline Naranian Paul Paroutis Ray Guo Zhi-Ping Ren Ichizo Nishino Brigitte Chabrol Jean-Francois Pellissier Carlo Minetti Bjarne Udd Michel Fardeau Chetankumar S. Tailor Don J. Mahuran John T. Kissel Hannu Kalimo Nicolas Levy Morris F. Manolson Cameron A. Ackerley Berge A. Minassian

X-linked myopathy with excessive autophagy (XMEA) is a childhood-onset disease characterized by progressive vacuolation and atrophy of skeletal muscle. We show that XMEA is caused by hypomorphic alleles of the VMA21 gene, that VMA21 is the diverged human ortholog of the yeast Vma21p protein, and that like Vma21p it is an essential assembly chaperone of the V-ATPase, the principal mammalian prot...

Journal: :Autophagy 2013
James K Ching Jeong Sun Ju Sara K Pittman Marta Margeta Conrad C Weihl

Colchicine treatment is associated with an autophagic vacuolar myopathy in human patients. The presumed mechanism of colchicine-induced myotoxicity is the destabilization of the microtubule system that leads to impaired autophagosome-lysosome fusion and the accumulation of autophagic vacuoles. Using the MTOR inhibitor rapamycin we augmented colchicine’s myotoxic effect by increasing the autopha...

Journal: :Pathobiology : journal of immunopathology, molecular and cellular biology 2007
Noriyuki Kimura Toshihide Kumamoto Yosuke Kawamura Takahiro Himeno Ken-Ichro Nakamura Hidetsugu Ueyama Ryuki Arakawa

OBJECTIVE Chloroquine modulates autophagocytic protein degradation in the lysosome system, thereby inducing the formation of rimmed vacuoles consisting of autophagosomes and autolysosomes in skeletal muscle. The goal of this study was to investigate the contribution of the lysosomal system, particularly autophagosome formation (an autophagic process) at the molecular level, to the abnormal accu...

Journal: :Physiological research 2014
Z Papáčková M Cahová

Autophagy is the basic catabolic mechanism that involves degradation of dysfunctional cellular components through the action of lysosome as well as supplying energy and compounds for the synthesis of essential biomacromolecules. This process enables cells to survive stress from the external environment like nutrient deprivation. Autophagy is important in the breakdown of proteins, carbohydrates...

بینش, فریبا, فلاح, راضیه, مرتضوی زاده, محمد رضا,

Mitochondrial myopathy, lactic acidosis, and siderobastic anemia (MLA SA) syndrome is one of the newly reported mitochondrial diseases, seven cases of which have been reported. We report a child with inflammatory myopathy, sideroblastic anemia and lactic acidosis .The patient is a 8.5 year old boy with normal cognitive function suffering from chronic progressive weakness in lower extremities, ...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1999
A Amalfitano A J McVie-Wylie H Hu T L Dawson N Raben P Plotz Y T Chen

This report demonstrates that a single intravenous administration of a gene therapy vector can potentially result in the correction of all affected muscles in a mouse model of a human genetic muscle disease. These results were achieved by capitalizing both on the positive attributes of modified adenovirus-based vectoring systems and receptor-mediated lysosomal targeting of enzymes. The muscle d...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید