نتایج جستجو برای: mitohormesis

تعداد نتایج: 66  

2012
Ning Li Suzana Stojanovski Pierre Maechler

In pancreatic β cells, mitochondrial metabolism translates glucose sensing into signals regulating insulin secretion. Chronic exposure of β cells to excessive nutrients, namely, glucolipotoxicity, impairs β-cell function. This is associated with elevated ROS production from overstimulated mitochondria. Mitochondria are not only the major source of cellular ROS, they are also the primary target ...

Journal: :Experimental gerontology 2010
Michael Ristow Kim Zarse

Recent evidence suggests that calorie restriction and specifically reduced glucose metabolism induces mitochondrial metabolism to extend life span in various model organisms, including Saccharomyces cerevisiae, Drosophila melanogaster, Caenorhabditis elegans and possibly mice. In conflict with Harman's free radical theory of aging (FRTA), these effects may be due to increased formation of react...

2017
Hemantkumar Chavan Pamela Christudoss Kristen Mickey Robert Tessman Hong-Min Ni Russell Swerdlow Partha Krishnamurthy

Arsenite is a known carcinogen and its exposure has been implicated in a variety of noncarcinogenic health concerns. Increased oxidative stress is thought to be the primary cause of arsenite toxicity and the toxic effect is thought to be linear with detrimental effects reported at all concentrations of arsenite. But the paradigm of linear dose response in arsenite toxicity is shifting. In the p...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Wouter De Haes Lotte Frooninckx Roel Van Assche Arne Smolders Geert Depuydt Johan Billen Bart P Braeckman Liliane Schoofs Liesbet Temmerman

The antiglycemic drug metformin, widely prescribed as first-line treatment of type II diabetes mellitus, has lifespan-extending properties. Precisely how this is achieved remains unclear. Via a quantitative proteomics approach using the model organism Caenorhabditis elegans, we gained molecular understanding of the physiological changes elicited by metformin exposure, including changes in branc...

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