نتایج جستجو برای: mog

تعداد نتایج: 1228  

Journal: :European journal of immunology 2004
Emily Mathey Constanze Breithaupt Anna S Schubart Christopher Linington

Myelin oligodendrocyte glycoprotein (MOG) is the only myelin protein known to initiate a demyelinating autoantibody response in EAE, an animal model for multiple sclerosis (MS). The pathophysiological significance of MOG-specific autoantibodies in MS is, however, controversial, as high titer antibody responses to MOG are also found in many patients with non-demyelinating neurological diseases. ...

Journal: :Journal of immunology 2008
Yolanda S Kap Paul Smith S Anwar Jagessar Ed Remarque Erwin Blezer Gustav J Strijkers Jon D Laman Rogier Q Hintzen Jan Bauer Herbert P M Brok Bert A 't Hart

The recombinant human (rh) myelin/oligodendrocyte glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE) model in the common marmoset is characterized by 100% disease incidence, a chronic disease course, and a variable time interval between immunization and neurological impairment. We investigated whether monkeys with fast and slow disease progression display different anti-...

Journal: :The Journal of biological chemistry 2012
Katrien L de Graaf Monika Albert Robert Weissert

It has become increasingly clear that only antibodies recognizing conformation-dependent epitopes of myelin oligodendrocyte glycoprotein (MOG) have a demyelinating potential in the animal model of multiple sclerosis, experimental autoimmune encephalomyelitis (EAE). Nevertheless, for the induction of EAE, most studies to date have used MOG peptides or bacterially expressed MOG, neither of which ...

Journal: :Neurology 2011
A K Pröbstel K Dornmair R Bittner P Sperl D Jenne S Magalhaes A Villalobos C Breithaupt R Weissert U Jacob M Krumbholz T Kuempfel A Blaschek W Stark J Gärtner D Pohl K Rostasy F Weber I Forne M Khademi T Olsson F Brilot E Tantsis R C Dale H Wekerle R Hohlfeld B Banwell A Bar-Or E Meinl T Derfuss

OBJECTIVE To study the longitudinal dynamics of anti-myelin oligodendrocyte glycoprotein (MOG) autoantibodies in childhood demyelinating diseases. METHODS We addressed the kinetics of anti-MOG immunoglobulins in a prospective study comprising 77 pediatric patients. This was supplemented by a cross-sectional study analyzing 126 pediatric patients with acute demyelination and 62 adult patients ...

Journal: :Journal of neurology, neurosurgery, and psychiatry 2015
M Baumann K Sahin C Lechner E M Hennes K Schanda S Mader M Karenfort C Selch M Häusler A Eisenkölbl M Salandin U Gruber-Sedlmayr A Blaschek V Kraus S Leiz J Finsterwalder T Gotwald G Kuchukhidze T Berger M Reindl K Rostásy

BACKGROUND Myelin oligodendrocyte glycoprotein (MOG) antibodies have been recently described in children with acute disseminating encephalomyelitis (ADEM), but the clinical and neuroradiological characterisation of this subgroup is lacking. OBJECTIVE To compare the clinical and neuroradiological features of paediatric ADEM with and without MOG antibodies. METHODS Clinical course, cerebrospi...

2015
H.-Christian von Büdingen Feng Mei Ariele Greenfield Sarah Jahn Yun-An A. Shen Hugh H. Reid David D. McKemy Jonah R. Chan

Myelin oligodendrocyte glycoprotein (MOG) is a central nervous system myelin-specific molecule expressed on the outer lamellae of myelin. To date, the exact function of MOG has remained unknown, with MOG knockout mice displaying normal myelin ultrastructure and no apparent specific phenotype. In this paper, we identify nerve growth factor (NGF) as a binding partner for MOG and demonstrate that ...

Journal: :Journal of neuroimmunology 2002
Andreas Lutterotti Markus Reindl Christoph Gassner Katharina Poustka Kathrin Schanda Florian Deisenhammer Thomas Berger

We investigated the association of the antibody response to myelin oligodendrocyte glycoprotein (MOG) and myelin basic protein (MBP) with human leukocyte antigen (HLA) class II alleles in 41 patients with sporadic multiple sclerosis (MS) and 12 multiplex MS families. We found significantly increased antibody response to MOG and MBP in MS patients without any difference to asymptomatic relatives...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2006
Dun Zhou Rajneesh Srivastava Stefan Nessler Verena Grummel Norbert Sommer Wolfgang Brück Hans-Peter Hartung Christine Stadelmann Bernhard Hemmer

Multiple sclerosis (MS) is a chronic inflammatory disease of the central nervous system. Although the cause of MS is still uncertain, many findings point toward an ongoing autoimmune response to myelin antigens. Because of its location on the outer surface of the myelin sheath and its pathogenicity in the experimental autoimmune encephalomyelitis model, myelin oligodendrocyte glycoprotein (MOG)...

Journal: :Journal of clinical oncology : official journal of the American Society of Clinical Oncology 2016
Shigeo Fuji Yoshitaka Inoue Atae Utsunomiya Yukiyoshi Moriuchi Kaoru Uchimaru Ilseung Choi Eiichi Otsuka Hideho Henzan Koji Kato Takeaki Tomoyose Hisashi Yamamoto Saiko Kurosawa Ken-Ichi Matsuoka Takuhiro Yamaguchi Takahiro Fukuda

PURPOSE Allogeneic hematopoietic stem-cell transplantation (allo-HSCT) is one important treatment option for patients with aggressive adult T-cell leukemia/lymphoma (ATLL). Mogamulizumab (anti-CCR4 monoclonal antibody; Mog) was recently approved as a treatment for ATLL in Japan. Major concerns exist about the possible adverse effects of pretransplantation Mog because Mog depletes regulatory T c...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2003
C B Marta C M Taylor T Coetzee T Kim S Winkler R Bansal S E Pfeiffer

Myelin oligodendrocyte glycoprotein (MOG) is, quantitatively, a relatively minor component of the myelin membrane. Nevertheless, peritoneal administration of MOG evokes potent cellular and humoral immunoreactivity, resulting in an experimental allergic encephalitis with immunopathology similar to multiple sclerosis. Moreover, antibodies against MOG cause myelin destruction in situ. Therefore, i...

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